Sclerosis Case Study Essay

Mr. GL’s sedation was paused thirty minutes prior to performing assessment. The patient was unresponsive throughout the entirety of the head-to-toe assessment with a GCS of 3. The patient’s pupillary response was sluggish. He was on mechanical ventilation set to assist control with a respiratory rate of 26, FiO2 of 80%, PEEP of 5, and tidal volume of 500. His endotracheal tube remained at 23 inches. His chest rise was equal. Wheezing was heard in all lung fields. Heart sounds were muffled, but S1 and S2 were present. EKG revealed sinus tachycardia with a heart rate of 106. The patient’s abdomen was soft and non-distended with hypoactive bowel sounds in all four quadrants. The patient’s foley catheter was intact and patent with dark yellow, clear urine output. A

Multiple sclerosis is a chronic, progressive neurological disease affecting all aspects of life: physical, cognitive, emotional, and social (Abma). It is known as an autoimmune disease, Where the body’s immune system turns against the body and destroys the protective covering that surrounds nerve cells. This damage to the nerve cells causes many problems for the patient including weakness, muscle stiffness, poor coordination and balance, tingling, numbness, tremors, blurred vision, slurred speech, and memory and concentration problems (Bren)

Multiple sclerosis, or MS, is a disease of the central nervous system. The central nervous system is made up of the brain and spinal cord. Both have nerve fibers that are wrapped in a myelin sheath. In MS, the myelin sheath becomes inflamed and gradually is destroyed. With the destruction of the myelin sheath comes an array of symptoms that may include numbness or tingling, balance problems, weakness, muscle spasms, and blurred vision.

Multiple Sclerosis is an autoimmune disorder where the myelin sheath within the Central Nervous System is attacked (National Multiple Sclerosis Society, 2017). The myelin sheath protects the axon of the nerve cell. When the myelin sheath is intact, the axon is able to carry impulses away from the neuron’s cell body, and the message carried is clear. With Multiple Sclerosis, the myelin sheath becomes scarred, hence the word “sclerosis”, and distorts the nerve impulses traveling over the CNS (National Multiple Sclerosis Society, 2017). This may cause the message to be changed or stopped altogether.

It was a raw, blustery March day and I was leading four classmates to my house to hash out the remaining details of our current English presentation. When I opened the door, however, I received a surprise. I had not anticipated my mother still being home and neither had my group members. Their faces turned slightly blank, as if they were trying to hide their confusion and surprise. The previously relaxed atmosphere had become very formal and quiet. I had seen this before.

Multiple sclerosis is a complex, autoimmune disease caused by damage of the fatty myelin sheaths around axons of the brain and spinal cord which leads to demyelination, lesions (scaring) and inflammation1, 2, 3. This damage leads to disruption of nerve impulse along axons resulting in the many symptoms seen in multiple sclerosis patients. Although its exact etiology is unknown, current research indicates that multiple sclerosis is a complex combination of genetic and environmental factors. It has been established that multiple sclerosis is not inherited but research is being focused on genetic influence and suceptibility3. Like many other diseases with genetic components to their etiology, disease risk for multiple sclerosis can be

Multiple Sclerosis (MS) is a debilitating chronic disease where the immune system attacks the myelin sheaths of nerve cells in the central nervous system, affecting more than 400,000 people in the US with about 200 new cases being diagnosed each week (Pietrangelo and Higuera 2014). We still do know what causes MS and although there is no cure, there are several treatments for MS focused on slowing down the progression of the disease and managing symptoms. Although the causes of MS have not been identified, there has been much research into genetic susceptibility and exposure to environmental risk factors as well as research into how different populations manage their MS.

Multiple sclerosis (MS) is an immune mediated disease of the central nervous system, this progressive neurological disability is commonly found in young adults (Compston & Cole, 2002). Due to the intrusiveness and unpredictability of MS, people with MS experience difficulties in managing their illness while employed (Devins, Seland, Klein, Edworthy, & Saary, 1993; Devins & Shnek, 2000). According to McKeown et al. (2003) and Rudick (1998) the disease occurs: “between the ages of 20 and 50 years in 90% of cases. The disease strikes people during the peak years of their education, career development and family life, significantly impacting their ability to stay in the workforce.” Fraser et al. (2002) research

Corey is a 35-year-old male who suffers from multiple sclerosis (G35). His symptoms include mild right leg paresis and deep tendon reflexes are brisk throughout, and difficulty with balance. Corey has been on Glatopa since July 2016, is stable and in need of this medication. The prior authorization was denied due to no recent documentation. There is documentation attached with this appeal. To deny him coverage of a therapy that is working for him is negligent and unethical. MS patients should have the right to treatment regardless of disease duration or level of disability, to help improve their quality of life. Without this treatment, I fear he will progress and relapse, experiencing debilitating pain as well as quality of life

The occurrence of physically inactive people diagnosed with multiple sclerosis (MS) is widespread. There is a strong desire to research ways to improve physical activity among people with MS and to improve their quality of life. People with MS deserve the many health benefits that physical activity brings. The purpose of this article was to identify an intervention, a behavioral intervention strategy to increase physical activity, particularly walking, among people with MS. The article introduces a behavioral intervention strategy involving MS patient’s self-reported data along with objectively measured physical activity. The interest in identifying new ways to get people with MS more physically active is on the rise. Using social and internet behavior interventions may be a significant approach (Motl & Dlugonski, 2011).

The patient is 69-year-old male physician who had a syncopal episode that was witnessed. The patient was sitting talking to colleagues at a meeting and he suddenly became fail, fainted was diaphoretic and became bradycardic, never lost his pulse and awakened and was appropriate within approximately a minute after the episode. He has not felt well for approximately 2 days prior to the event. He has felt malaise and cold the morning of the event. He did not eat or drink , nor did he eat or drink the previous evening,he took his antihypertensives and then went to the meeting. He is known to have hypertension. He had a recent stress test within the past year which was negative. He also had an appendectomy and left hemicolectomy in the past.

The only medications he takes are metformin and hydrochlorothiazide. The patient appears dyspneic and diaphoretic. An ECG and chest Xx-ray are performed and shown herebelow:

a). Based in my family history, one of my grandfather died at age of 65 of multiple sclerosis (my mother’s dad). My other grandfather died at 101 years old (my dad’s dad), and my two grandmothers are still alive, one has almost 100 years old (my dad’s mother), the other has 89 and she is very active and she still does not need glasses to read (my mom’s mother). My mom (62 years old) has diabetes and high blood pressure and my dad (68 years old) he has no issues at all with his health. I ’am 31 years old, my only issues are migraine and scoliosis. I try to do exercise, eat clean and take care of myself. Considering my dad’s side of the family and my grandmother’s genes and if I keep a good routine of healthy habits, I’ll say that I’ll have

Mr. Armstrong has a history of renal insufficiency and uncontrolled hypertension, along with symptoms of fatigue, pedal edema, and occasional shortness of breath. He does not have a history of trauma or obstruction to his kidneys, but his creatinine and BUN levels are currently at 3.5 mg/dl and 40 mg/dl. Normal creatinine concentration values are 0.7 to 1.2 mg/dl and normal BUN values are 10 to 20 mg/dl; this reveals that Mr. Armstrong’s kidneys are not removing wastes properly (McCance, Huether, Brashers, & Rote, 2014).  Mr. Armstrong’s history of renal insufficiency and uncontrolled hypertension is commonly found in patients diagnosed with intrarenal (intrinsic) acute renal failure. Intrarenal acute renal failure can be categorized as

Case Study: A thirty six year old male has developed severe muscle weakness throughout the body. The condition began fifteen months ago with a left foot drop and within a year, he described difficulty with speech and swallowing, muscle twitching and cramping, and muscular atrophy throughout the upper and lower limbs. Within the last two months, his breathing has become more difficult, and there has been a noticeable difference in his voice. Prior to the fifteen months, he presented little to no symptoms. The patient, a medical doctor, has a wife and two young children.