Self Destructive Behavior and Role of the I function
The I function describes all behavior associated with the notion of self. Is there really a specific I function and what is its role exactly? Because the I function is linked to the self, one would think that it would prevent harmful behaviors. However, there are cases where the I function does not intervene to terminate detrimental actions for example addiction and a mental disorder called Lesch-Nyhan syndrome. In both cases, a person is harming themselves and can not seem to stop. Does the I function play a role in self-destructive behavior? By researching addiction, more specifically alcoholism, and Lesch-Nyhan syndrome, it is clear that the I function can be overruled by other
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The three examples above increase the release of dopamine at synapses. This increase corresponds with satisfaction or reward (3). There is evidence of a mutation of the dopamine receptor gene (D2A1) in alcoholics. This mutation was present in 69% of severe alcoholics and 20% of non-alcoholics (4). This mutation does not mean that alcoholism is always genetic. The study was also done on Vietnam Veterans who had post-traumatic stress disorder due to severe combat conditions (4). This mutation was found in 40%-55% of these patients (4). Veterans were not born to go to war so the mutation could be caused by a chemical change due to the bodyis response to high stress. One hypothesis is that the D2A1 is affecting the degree of expression of the genes that are involved with addictive behaviors. The genes have not yet been identified (4).
Dopamine is associated with seratonin. Seratonin is affiliated with the part of the brain responsible for controlling emotions, paying attention, and thinking before acting (4). Research was done to determine whether or not alcoholics had seratonin abnormalities. A mutation of an enzyme, tryptophan oxygenase is hypothesized to be the cause of abnormal seratonin amounts in the blood stream of alcoholics and patients suffering from other mental disorders all of which exhibit addictive behaviors (4). Tryptophan oxygenase breaks down tryptophan. If
210). This creates difficulty with understanding the possible consequences or rewards of an action. According to Dolan, (2010), certain cues can be missed due to the deficits within the brain, creating a difficulty for that person to empathize. It has also been found that there are possible genetic links involving dopamine and serotonin, although these genes may be affected by the environment as well, which complicates the research (Rosenberg & Kosslyn, 2011).
Addiction is thought of a biological nature that is past through genetics according to the biological theory as explained in P2. A contrasting theory of the biological theory would be the classical and operant conditioning theory, this was made by Pavlov. The basis of conditioning theories is that addiction is the end result of the reinforcement of drug use. The drug acts as a reinforcer and gains control over the user’s behavior. In contrast to the biological models of the exposure theories, these conditioning models suggest that anyone can show addictive behavior if they are given reinforcements, regardless of their genetics. The advantage of this
The neurotransmitter dopamine is usually affected by addictive substances as well as implicated in disorders such as depression, schizophrenic psychosis, and bipolar. Because of the overlapping in substance abuse disorders and mental health illnesses, this suggests that brain changes in one will create changes in the other. Stress has also been identified as a risk factor for addiction and mental illnesses and is the biological link between these two disorders.
To begin with, studies have found that inherited genes are responsible for a considerably large part of the probability that someone will become addicted to any kind of substance or behavior. Addiction is strongly influenced by genetic factors in the later stages of addiction, such as problem use and dependence of an addictive substance. Some genetic factors can make an individual more likely to become addicted to only one specific drug. On the other hand, some genes can make an individual more likely to become addicted in general. Some individuals might be genetically inclined to risk-taking behavior such as receiving large jolts of dopamine through what they consider to be the “excitement” of drug use. These individuals could be subject to keep returning to the illicit and harmful drugs that they find enjoyable despite what they know of the harmful effects. These genetic influences could make them more likely to become addicted to alcohol, cocaine, tobacco, gambling, or any number of substances or activities. Addiction has an inherited component and it
This research paper will evaluate the biological aspects of addictive substance or behavior and how it affects the brain and organs. Biological aspects include dopamine levels that are replaced in the brain due to the reward system being overtaken and the absorption rate of the drug once it is ingested will be discussed. The biological aspects are extensive and permanent if the individual does not get the help they need. Furthermore the clinical issues of addictive substance or behavior will be discussed along with medical treatments and ethical issues. This includes treatments such as counseling
79). Overall, this is a problem, because there continues to be a lack of the public endorsement and support of those seeking treatment regardless of the addiction. Individuals suffer with addictive behaviors for many reasons that include genetic factors. These things are uncontrollable and are harder to be prevented. Inaba and Cohen (2014) stated, “One of the genes that signals a susceptibility to compulsive overreaction” (p. 7.47). The new definition of addiction non/substance and behavior is now based on the psychoactive drugs and certain behaviors that produce the surge of dopamine in the midbrain are biological substrate for addictive behavior (Smith, 2012). Overall, it is important to include all addictive behaviors and substance abuse when diagnosing individuals. It is more important to treat all conditions or addictions than it is to assume that it is not as an important or one is more problematic than one or the
Is addiction a brain disease? The definition of disease is a disorder in an organism that produces specific signs/symptoms that affects a specific location and isn’t a direct result of any kind of physical injury. Addiction is a compulsive need for any substance or service that can cause a person or organism to form a habit, and is a chronic disease of brain reward and causes dysfunctions in the circuits. Addiction is also characterized by the inability to consistently abstain impairment in behavioral control. The idea that addiction is a brain disease is just about universally accepted among scientist that focus on addiction (Corrigan,2008). However few believe that addiction is not a brain disease because they claim that neural dysfunction is not sufficient for disease. (NCBI, 2012). Some agree that addiction occurs in the brain but should not be considered a brain disease or a disease at all(Lawrence 2013). Substance abuse is the most common addiction that people base their decision upon. These drugs affect the brain and cause different reactions to the rest of the of the body. The types of drugs include: hallucinogens, opioids, sedatives, stimulants, etc. cause different
I used to be a cutter… but shh, don’t tell. Society didn't allow me to express my pain that way…Today’s society refuses to acknowledge many problems that teens deal with. People would rather believe in ‘happily ever after’ than face the truth, children are suffering. While it may seem to others that the problems are made up, its very real to the one dealing with them everyday. This suffering can leave permanent scars, and damage (if not ruin) their future. By remaining ignorant, self harm becomes more and more common, but at what cost? If educators and medical personnel were to be more educated on self harm and how to deal with it, teens and young adults would be more comfortable asking for help. Getttig rid of the stereotypes that
The results of these earlier studies were expounded on by research that suggested increased striatal dopamine
Substance addiction can hugely impact on a person’s thinking, functioning and behaviour. Whilst depressant drugs such as alcohol and opioids slow down your central nervous system (CNS), stimulants such as cocaine or amphetamine increase the activity of the CNS, leading to higher blood pressure, heart rate and increased alertness. Repeated abuse of the substance leads to tolerance and withdrawal, in turn this leads to the user showing signs of irrational behaviour. Another form of substance abuse is hallucinogens; they can cause powerful changes in sensory perceptions. It works by binding to the serotonin receptors, these neurons control visual information and emotions, and this can lead to various effects on the user. Neurobiology can help us understand the reasons for addiction and the effects they have on us. Groman and Jentsch (2012) discuss key issues of differentiating the causes and consequences of addiction through neuroimaging and behavioural research on monkeys, they found that the dysfunction of the dopamine
An individual’s behavior and emotion becomes chemically altered often resulting in dependency, aggression, onset of diseases and poor judgement. This poses a dangerous threat to the neurotransmitters since they have multiple jobs in different parts of the brain. Drugs of abuse are able to exert influence over the brain reward pathway either by directly influencing the action of dopamine within the system, or by altering the activity of other neurotransmitters that exert a modulatory influence over this pathway. These drugs are often powerful and have been known to trigger schizophrenic behavior and can also cause a person to cease breathing, for example hallucinogens such as LSD, mescaline, and psilocybin are able to artificially stimulate the serotonin receptor (Sapolsky, 2005).
Deliberate self-harm is a term that covers a wide range of behaviours some of which are directly related to suicide and some are not. This is a relatively common behaviour that is little understood. This essay provides an overview of the nature and extent of those most at risk of self-harm, including causes and risk factors. Examining some of the stereotyping that surrounds self -harm, and looking at ways in which self-harm can be prevented.
Cocaine, on the other hand, has been found to have a profound effect on increased dopamine activity. Kennedy & Hanbauer (1983; cited in Karch, 2007) found a strong correlation between cocaine binding and the inhibition of dopamine uptake, and therefore proposed that dopamine transporters are binding sites for cocaine. Such research, which correlates strong reinforcing properties of cocaine with inhibition of dopamine transporters, suggests that cocaine has a more significant affect upon dopamine, than than it has upon serotonin and noradrenaline. The excessive role of dopamine can explain the high degree of pleasure that users of cocaine experience, as well as depression and cravings, which follow.
In the article “Dopamine and Teenage Logic” by Daniel Siegel he states that “Dopamine is a neurotransmitter central in creating our drive for reward” (Source B). This indicates that dopamine is one of the key causes of risk-taking. Teens often take risks because they are focused on the reward they get after they take it, rather than what would actually happen to them during and after they take the risk. There are two significant ways that dopamine affects our brains; increased impulsiveness, and the susceptibility to addiction by the release of dopamine.
The complexity of the human brain creates mystery when determining the influence of neurophysiological factors and their role in the process of addiction. There is a proposed relationship between drug addiction and the mesolimbic dopamine system, with the mesolimbic pathway from the ventral tegmental area to the nucleus accumbens considered the ‘reward centre’ of the human brain (Alcohol Rehab, 2011). A release of dopamine is necessary for ‘reward’ which is hypothesised to initiate the addiction cycle by providing positive reinforcement for drug self-administration (Feltenstein & See, 2009). Methamphetamine triggers the release of dopamine from synaptic vesicles which flood the synaptic cleft activating feelings of euphoria, well-being