Starting At The Age Of Seven, I Can Distinctly Remember

Scientifically, Hashimoto’s Disease is, “a condition in which your immune system attacks your thyroid, a small gland at the base of your neck. The thyroid is a part of your endocrine system, which produces hormones that coordinate many of your body’s activities. The resulting inflammation from Hashimoto’s often leads to an under active thyroid” (Mayo Clinic.) Unfortunately, my thyroid didn’t work properly, and my body attacked it because of this.

Hashimoto’s Thyroiditis is an autoimmune disease in which the immune system attacks the thyroid. The thyroid is a small gland at the base of the neck that produces hormones and that help control most of the bodies activities. Inflammation to the thyroid, from Hashimoto’s, often leads to hypothyroidism, an underactive thyroid gland. Hashimoto’s disease is the number one cause of hypothyroidism in the United States. It mainly affects middle aged women but Hashimoto’s can affect men, women of any age, and even children (“Hashimoto’s Disease-National Library of Medicine-PubMed Health,” 2014).

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There are a few symptoms that are results of iodine deficiency. Iodine deficiency can cause hypothyroidism. Some symptoms of hypothyroidism include a decline in energy, can make weightloss challenging, and even hair loss. Glands need proper amounts of iodine in order to function properly, and create hormones. Lastly, iodine deficiency can lead to goiter, which is thyroid inflammation.

Hashimoto’s Thyroiditis, also called Hashimoto’s disease is an autoimmune disease in which the body’s immune system attacks the thyroid gland. It was named for the Japanese surgeon who discovered it in 1912 The thyroid is a small butterfly shaped gland in the front of the neck. The thyroid makes hormones called T3 and T4. These hormones regulate metabolism. The thyroid is controlled by hormones of the pituitary gland, which is also called the “master gland.” It is a pea-sized gland located in the base of the brain which, among others, makes thyroid stimulating hormone, or TSH. TSH stimulates the thyroid to make thyroid hormone. With Hashimoto’s disease, the thyroid cells are damaged resulting in the inability to make enough thyroid

pituitary is not creating as much TSH. This would cause the Thyroid to stop producing T4

Hashimoto thyroiditis is a common disease caused by hypothyroidism. The immune system cells attack the thyroid gland, causing inflammation and eventually destruction of the gland. This reduces the thyroids ability to make hormones. These hormones are known as thyroxine (T4) and tri-iodothyronine (T3) (Better health channel 2015).

Hypothyroidism, while a common disorder, is often misdiagnosed due to its ability to mimic other common health problems. A simple, yet specialized blood test, coupled with a complete medical history, will allow the experts at RCMC Medical Center to not only diagnose the problem in a timely manner, but also develop a treatment plan based on the results, and your personal lifestyle, with the end goal of returning you to the level of health and wellness you

Hashimoto’s Thyroiditis can also come as an attack on the thyroid via an autoimmune disorder. The immune system can attack the thyroid; however, no one knows exactly why this

When looking at my genogram, I see a strong genetic presence. Hypothyroidism has been diagnosed in my father, my grandmother on my mom’s side, my mother, all three of my mother’s sisters, one female cousin on my mom’s side, and me. This being said, all adult females on my mother’s side are diagnosed with hypothyroidism. Each of them were diagnosed in their adulthood, where as I was diagnosed at the age of 5. I believe that the early onset was in association with my Type 1 Diabetes. However, it seems to me that there is a clear genetic disposition running in my family. By analyzing my familial, I have concluded that the younger females in my family are also at high risk of developing hypothyroidism at some point in their

In Hashimoto’s disease, the immune system attacks the thyroid gland, causing inflammation and interfering with its ability to produce thyroid hormones.

Response: Hashimoto’s thyroiditis is characterized as a type of immune disorder and is the leading cause of hypothyroidism in America.1 The gradual failure of the thyroid is due to an autoimmune disorder that is causing glandular destruction.2 Thyroxine (T4) and triiodothyronine (T3) are two hormones

JG, a 34-year old Caucasian female presented to the primary care physician office with complaints of losing weight and feelings that "her heart is beating out of her chest". On assessment it was noted that JG had lost twenty pounds in 3 months. JG exhibited tremors in hands, bulging eyes, and pretibial edema. On physical examination there was a goiter, or enlarged thyroid gland noted.

The purpose of this abstract is to examine Hashimoto’s thyroiditis, also known as Hashimoto’s disease, and chronic lymphocytic thyroiditis, which is an autoimmune disease. By researching this condition through medical journals, related associations, and relevant reports, I was able to gather information on the effects to health associated with this disease. The causes of Hashimoto’s thyroiditis are not clearly understood, however certain people are more likely than others to have this condition. Also, those with Hashimoto’s thyroiditis are more likely to have other autoimmune diseases. Normally, the condition develops slowly, and its signs and symptoms are indistinct, and can be similar to other conditions. To diagnose Hashimoto’s disease,

Thyroid hormones have been known to be essential for normal neurodevelopment for over a century1. A deficiency in thyroid hormone (TH) during gestation, often due to inadequate levels of iodine have serious consequences for offspring including cretinism which is characterized as severe cognitive impairment, ataxia and sensory deficits. Cretinism is the most common preventable cause of brain damage worldwide2. Research has shown that even mildly decreased thyroid hormone during this critical period can effect cognitive function3.