Symptoms And Treatment Of Eisenmenger Syndrome

Atrioventricular Canal Defect is an abnormality that causes the mixing of blood. There is a hole in center of heart where the wall between the upper and lower chambers meet. The tricuspid and mitral valves aren’t formed properly and one large valve crosses the defect. The defect lets oxygen rich blood pass to the heart’s right side and mix with deoxygenated blood, then go back to the lungs. Another abnormality is Atrial Septal Defect (ASD), where the walls of the upper chambers of the heart don’t close completely, causing a left to right movement of blood due to the higher pressure. The mixing of oxygenated and deoxygenated blood may cause the right atrium and ventricle to enlarge due to the higher volume of blood.

Apical hypertrophic cardiomyopathy is a disease that mainly affects the apex of the heart and does not cause any obstruction. [1] These abnormalities in the heart muscle can cause a wide variety of symptoms. As the heart becomes stiff it increases the pressure in the left ventricle which can push blood back into the lungs, causing shortness of breath in exercise. Chest pain can occur as there is not enough oxygen available to the cardiac muscle due to insufficient blood supply. Palpitations and lightheadedness, along with other conditions can occur as a result of HCM. In addition to these discomforting symptoms, the patient may develop an arrhythmias that often goes unnoticed. An arrhythmia takes place as the electrical conduction of the heart is disturbed by the abnormal scattering of myocytes. The two most common arrhythmias are atrial fibrillation causing palpitations, and ventricular tachycardia that can be life threatening causing sudden death. Both conditions can be controlled with medication. [4]

Congenital heart disease is a cardiovascular condition resulting from an abnormality in the structure of the heart. The exact time this defect forms is unknown but it is during the fetal development in the womb. Researchers think the defect could be case partially by genetics and medical conditions of the fetus and the parents. If the baby’s mother had rubella, while pregnant or other conditions such as diabetes could have caused the defect. Some medications that were taken by a pregnant mother could also have caused birth defects. An error on chromosome 22 could also have caused the heart defect. Some symptoms that a person may have a congenital heart may not show up until later in life but many children also have the symptoms. Symptoms of a congenital heart defect are abnormal heartbeat, bluish tint to skin, shortness of breath, dizziness or fainting, and swelling of body tissues and organs.

Pulmonary hypertension is a lung disorder. The arteries that carry blood from the heart to the lungs become narrowed, making it very hard for the blood to get through the vessels, this then causes the pressure in the arteries to increase more than usual (high blood pressure). Scientists think that the procedure starts with injury to the layer of cells that line the small blood vessels of the lungs.

2. The defect in Caleb’s heart allows blood to mix between the two ventricular chambers. Due to this defect would you expect the blood to move from left-to-right ventricle or right-to-left ventricle during systole? Explain your answer based on blood pressure and resistance in the heart and great vessels. It goes left to right during systole. The difference is normally, oxygen-poor (blue) blood returns to the right atrium from the body, travels to the right ventricle, and then is pumped into the lungs where it receives oxygen. Oxygen-rich (red) blood returns to the left atrium from the lungs, passes into the left ventricle, and then is pumped out to the body through the aorta. But when an infant has ventricular septal defect it still allows oxygen-rich (red) blood to pass from the left ventricle, through the opening in the septum, and then mix with oxygen-poor (blue) blood in the right ventricle. (ROCHESTER.EDU) but instead when systole occurs the blood gets mixed because of the septum therefore heart needs to pump harder to ensure that enough blood with oxygen reaches the body.

AS can occur in the congenital period or later in life, but is more common in adults due to lack of exercise, and nutrition causing calcium buildup. If it is congenital it will occur within the first eight weeks of pregnancy, which the cause is unknown. In a child with AS, the pressure is much higher than normal in the left chamber pumping. Blood that has leaked back into the left ventricle through the valve in between heartbeats, causing the ventricle to be enlarged. There are three stages of AS. Stage A occurs in patients with bicuspid aortic valve or aortic sclerosis who are symptom free but are at risk for AS. In stage B people have progressive AS with mild or moderate calcify valve leaflets, leaflets mobility, or mild or

Individuals with this disorder further acquire congenital heart defects. It specifically blocks off the natural blood flow from the lungs and right chamber of the heart and/or causes an anomalous gap in the barrier that divides the heart chambers in two. Another symptom of this disorder are the problems involved in the digestive system. People who have this syndrome are

Is the condition where the heart is not pumping the right amount of blood to the rest of the body as it should be

She was born with Tricuspid Atresia, Vetricula Steptum Defect, along with a Mirtral valve defect and numerous other medical issues. Tricuspid Atresia is a complex heart defect that occurs when the right ventricle in the heart fails to develop correctly during development in the womb. In Smilie’s case, it caused her to have little to no function in the right ventricle of the heart. To save Smilie’s life, doctors at the Prince Charles Hospital preformed a 3-stage surgery over 2 years to redirect blood flow within her body. Smilie had the BT Shunt at a few days old, the Hemi Fontan at 6 months old and the Fenestrated Fontan completed at 2 years of age. Smilie now has Hyploplastic Right Heart Syndrome as a result, which is a generalised term used to encompass the many conditions that can cause a lack of development in the Right Ventricle Side of the heart.

Patent Ductus Arteriosus (PDA) is one of the common congenital left to right cardiac defects seen in children. The ductus arteriosus is a large communication pathway that is naturally patent in the fetus, connecting the trunk of the major pulmonary artery to the descending aorta (Shinde, Basantwani, & Tendolkar, 2016). During the fetal life, ductus arteriosus is a normal structure that allows the blood to pass from the right ventricle to the descending aorta by bypassing the pulmonary circulation. The ductus arteriosus is an important structure in the fetal development as it allow the blood flow to the rest of the fetal organ and structure. In fetus pulmonary

Cardiovascular function results from the interplay of the heart, systemic vasculature, blood volume, and tissues in their functions as pump, transporting pathways, and oxygen carrying and consuming end-organs, and problems with structure or function of the filling and ejection mechanisms of the heart will lead to failing oxygen delivery and compensatory attempts, a complex disease condition known as heart failure (Porth, 2015). Dysfunction may involve initially primarily one ventricle only, though over time both sides may become affected. In left ventricular failure decreased cardiac output and outflow into the periphery leads to pulmonary congestion, evidenced by pulmonary edema and impaired gas exchange (Porth, 2015). As a result,

The human body is a very complex organism composed of different types of systems and functions. All the functions that each system has, is what makes possible for the body to obtain life. One of the most important systems in one’s body is the circulatory system, where the heart, the lungs, and the blood vessels work together to form the circle part of the circulatory system. The pumping of the heart forces the blood on its journey. The body’s circulatory system really has three parts: pulmonary circulation, coronary circulation, and systemic circulation. Each part must be working independently in order for them to all work together. However, when one of the parts of the circulatory system does not

Congenital cyanotic heart disease (CCHD) is a cardiac defect which facilitates complete mixing of the deoxygenated systemic venous blood returning from the tissue and the fully oxygenated pulmonary venous blood from the lung in a common receiving chamber. (4)

Atrial Septal Defect(ASD) is a very large problem concerning the heart in its overall function. When the heart, being the core of the cardiovascular system, has issues; it effects the rest of the body as a result. The core of the problem resides in the atrial septum. Normally the heart is divided into four separate chambers. But a person with atrial septal defect has an atrial septum that allows the blood from the left side of the heart back into the right side. This results in increased pulmonary blood flow and diastolic overload of the right ventricle. By having this constant left-to-right shunt, it can alter the pulmonary vascular resistance leading to hypertension or even the reversal of the shunt itself.

Patent ductus arteriosus (PDA), one of the more common cardiac defects at birth, is the persistence of an opening between the pulmonary artery and the descending thoracic aorta. This opening is as a result of failure of the physiological fetus ductus arteriosus to close, which normally occurs soon after birth. This hole allows for oxygenated blood from the aorta to mix with oxygen-depleted blood from the pulmonary artery. As a consequence, significant strain is placed on the heart and pressure within the lungs’ arteries is dramatically increased.