Symptoms And Treatment Of Multiple Sclerosis

Multiple sclerosis (MS) involves an autoimmune process that develops when a previous viral insult to the nervous system has occurred in a genetically susceptible individual. B lymphocytes, plasma cells, and activated T cells, along with proinflammatory cytokines, cause inflammation, oligodendrocyte injury and demyelination. Early inflammation and demyelination lead to irreversible axonal

Multiple Sclerosis (MS) is a neurologic disease that affects the Central Nervous System (CNS) through cellular immune response and the demyelination of CNS white matter (McCance et al., 2014, pp. 630–633). The initial causes of MS are unknown however, it is believed that it could possibly be due to an immune response to an initiating infection or an autoimmune response to CNS antigens on the myelin itself (Brück, 2005) (Miljković and Spasojević, 2013). MS is a result of the degradation of the myelin sheath surrounding neurons and therefore disrupts the transmission of action potentials along these cells. MS can display itself in the form of symptoms ranging from muscle weakness to trouble with sensation and coordination (NHS, 2016). The degradation of myelin leads the body to attempt to remyelinate the neurons, a process that in turn leads to the thickening of the cell by glial cells and this causes lesions to form (Chari, 2007). It is this thickening (sclerae) from which the disease gets its name. Sufferers of MS can either have a relapsing type of MS, in which there are episodes that lead to the worsening of symptoms for a period of time, or a progressive type of MS where symptoms gradually progress and worsen (McCance et al., 2014, pp. 630–633).

The central nervous system (CNS) comprises grey matter, which contains neuron cell bodies and white matter, which contains the nerve axons. Most of the nerve axons are concentrically wrapped around by lipid-rich biological membrane, known as the myelin sheath. In the CNS, myelin is produced by oligodendrocyte. a type of glial cell. (Pfeiffer et al., 1993). These electrical insulating, multilamellar membranes significantly increase the electrical resistance, in which to prevent leakage of electrical currents from the axons, as well as decrease electrical capacitance to reduce the ability of the axons to store electrical energy (Shivane &

Multiple sclerosis is a chronic, progressive neurological disease affecting all aspects of life: physical, cognitive, emotional, and social (Abma). It is known as an autoimmune disease, Where the body’s immune system turns against the body and destroys the protective covering that surrounds nerve cells. This damage to the nerve cells causes many problems for the patient including weakness, muscle stiffness, poor coordination and balance, tingling, numbness, tremors, blurred vision, slurred speech, and memory and concentration problems (Bren)

Multiple Sclerosis (MS) is a chronic inflammatory disease of the central nervous system. MS affects approximately 3.0 million people globally, with about 300,000 cases in the United States. Twice as many women as men have MS. The average ages for onset of MS is 20-40 years. In MS, cells in the immune system attack and destroy myelin, the fatty tissue surrounding nerve cells (http://www.phylomed.com/MS.html). Scar tissue replaces the myelin, interfering with the transmission of nerve signals and leading to numbness, fatigue, spasticity, loss of muscle control, and various other debilitating symptoms. There are four broad theories

Multiple Sclerosis (MS) is an autoimmune disease that attacks the central nervous system. The myelinated nerve cells get attacked which results in scars and lesions that destroy the myelin sheath. The reason for the body attacking its own nerve cells is unknown. Many believe that the cause of MS is from a virus, a deficiency in Vitamin D, or genetics. Studies are being done to find out what is the real cause of MS.

Multiple Sclerosis (MS) a disease which the immune system attacks the protective sheath also known as the myelin that covers the nerves. Damages myelin disrupts the communication between the brain and the rest of the body. The nerves itself may weaken, process that is currently irreversible.

Multiple Sclerosis, commonly known as MS is an autoimmune disease of the central nervous system. Scientists have been studying MS since the 19th century. In MS, the body’s immune system produces cells and antibodies that attack myelin in your brain which is essential for the nerves in your brain and spinal cord to conduct electricity to perform its function. The attack on myelin results in vison loss, paralysis, numbness, muscle weakness, difficulty walking, stiffness, spasms, and bladder and bowel problems. MS has varying degrees of severity and affects people between the ages of 20-50, mostly women. Although there are treatments, there is no cause and cure yet.

Multiple Sclerosis is an inflammatory demyelinating neurodegenerative disorder of the central nervous system that has the potential to cause significant disability in those affected through the body's immune system attacking and destroying the myelin sheaths surrounding the axons. Myelin is rich in lipids and proteins that form layers around the nerve fibers and acts as insulation and protection. This damage to the myelin in the CNS, and to the actual nerve fibers, has the potential to block the transmission of nerve signals between the brain and spinal cord and also other parts of the body. This disruption of the nerve signals produces the primary symptoms of MS which then possibly lead to secondary and tertiary symptoms stemming from these

Multiple sclerosis, also known as MS, is an autoimmune and inflammatory disease that is very painful for both the patient and care giver. In the disease, there is inflammation and neurodegeneration acting at the same time. There is currently no known primary cause of multiple sclerosis. The disease is however characterized by damaged fatty myelin sheaths around the axons of the brain and the spinal cord. Myelin is a mixture of proteins and phospholipids that protects many nerve fibers enabling speed at which impulses are conducted. It is pathologically characterized as the presence of glial scars all over in the central nervous system. The disease was discovered by the French neurologist Jean-Martin Charcot in 1868 when he examined the brain

Multiple sclerosis (MS) is autoimmune, inflammatory disease involving the central nervous system (CNS). This disease is a result of the immune system attacking myelin proteins. Usually affecting younger people between the ages of 20 and 50 years, multiple sclerosis slowly destroys the myelin sheath that are located in the CNS causing them to eventually create scleroses, or hardened lesions. Symptoms of multiple sclerosis can be different, but visual, sensory, and motor signs are usually affected with this disease. Some early symptoms care problems like blurred or double vision, numbness, loss of balance, and weakness in arms or legs, as well as fatigue, muscle spasms, speech problems. Approximately 400,000 people in the United States and 2.5 million people worldwide are affected by multiple sclerosis (Hersh and Fox, 2014). Educating ones self about multiple sclerosis is important because it has many unanswered questions. There are many theories and treatments that may seem like they work, but there is no cure or definite reason why MS happens. Multiple sclerosis is also a disease that affects many people and is very hard to treat.

Multiple sclerosis (MS) is a chronic, often disabling disorder, affecting the central nervous system. The symptoms of MS may be mild, such as numbness in the limbs, or severe, such as paralysis or loss of vision. The progress of symptoms and their severity are currently unpredictable and vary from individual to individual. To date, the new treatments available and the progress of science are giving new hope to people affected by this First you need to specify the characteristics of this disease. In multiple sclerosis we are witnessing an immune system attack against the central nervous system.

Multiple sclerosis (MS) is an autoimmune disorder that causes the immune system to attack myelin sheath causing neurological damage to areas of the brain and causes neurological deterioration. In the brain the myelin sheath is involved with what allows for communication between brain and the body. As the disease progresses the sheath deteriorates the neurons where the myelin sheath is found is damaged, causing a break or loss of information when the brain is trying to communicate with the brain. As the central nervous system (CNS) is deteriorating it causes permanent damage that is incurable. The symptoms are not as

Multiple sclerosis is an autoimmune disease that majorly affects the brainand the spinal cord (A.D.A.M. Medical Encyclopedia, 1). The disease affects the central nervous system and thus causes limitations of individuals to carry out various activities. In multiple sclerosis, the myelin sheath that covers nerve cell axon is destroyed causing inflammation (MediResource Inc., 1). Destructionof the membrane leads to slowed conveyance of signals from the spinal cord to the brain, which as a result leads to reduced response to different stimuli. Inflammation of the nerve occurs mostly when the immune cells from the body attack the nervous system. The inflammation is not only limited to the spinal cord, but sometimes extends to

Multiple sclerosis is an autoimmune disease that damages the myelin sheaths and nervous cells in the central nervous system. An autoimmune disease is when the body produces antibodies to attack its own tissues. Basically, myelin is a mixture of proteins and phospholipids forming a whitish insulating sheath around many nerve fibers, increasing the speed that impulses are sent. It is made by schwann cells in the peripheral nervous system and oligodendrocytes in the central nervous system. Multiple sclerosis attacks the oligodendrocytes and the myelin sheaths causing some very serious problems for people diagnosed with it. The disease affects motor, sensory, and autonomic nerves and has been seen to affect cognitive functions. Females are affected