Temporal Lobe Amnesia Case Study

Retrograde Amnesia has pros and cons. Pros would be forgetting about bad ex’s and tragic events like rape or witnessing a murder. Not remembering were keys are or where you live would be a con. Retrograde Amnesia is recollection failure Derivative of neurological or psychological nature. Retro means recent past or before so the name really speaks for itself. Early signs for retrograde amnesia would be consistency of forgetting things that not so long ago happened or getting lost in places where you should have known were to be. Forgetting names like I myself sometimes forget might seem like a symptom but it’s not. People generally differ in their knowledge in degrees when it comes to factual information. However, if forgetting names of relatives

In Chapter 12 of The Man Who Mistook His Wife for a Hat, Mr. William Thompson suffered from an extreme case of Korsakov’s, also known as Wernicke-Korsakoff syndrome (Sacks, 1985, p. 109). According to Anderson (2010), Korsakoff syndrome can cause serious damage to one’s hippocampus and temporal lobe due to habitual alcoholism, resulting in amnesia (p. 201). Similar to HM (Anderson, 2010, p. 200), Mr. Thompson suffered from anterograde amnesia and could remember nothing for more than a few brief moments (Sacks, p. 109). Anderson (2010) states that anterograde amnesia results in the incapability of creating new memories, leading to fractional or absolute inability to remember something that just happened (p. 201). Yet, one’s long-term memories before the incident remain intact; such as when Mr. Thompson recognized his younger brother, Bob, as he walked by the window (Sacks, 1985, p. 112-113). Mr. Thompson and HM both suffered from anterograde amnesia and could remember some long-term memories but not remember new ones, signifying that the neural makeup involved in making fresh memories are diverse from maintaining past memories (Anderson, 2010, p. 202).

Anterograde Amnesia is the inability to store new information after the brain damage has occurred. (Luke Mastin 2010). Anterograde Amnesia is very rare, in fact there have a few cases where the amnesia was "pure". The symptoms and hardship of the person depend on the cause for the memory loss. Some symptoms of Anterograde Amnesia are partial memory loss, having a hard time recognizing relatives or family, feeling of confusion, difficulty taking in new information, inability to remember familiar places, and difficulty in learning and remembering new things.(PHC Editorial Team 2013) Characteristics of Anterograde Amnesia are abnormally small hippocampi bilaterally and elevated hippocampal water.(Mayo Clinic 2014) There are many ways to recognize

There are two main dimensions of amnesias. The first cause is physical or psychological; the second proceeds or follows a traumatic event. Most amnesia is a result either from organic or neurological causes (damage to the brain through physical injury, neurological disease or the use of certain drugs), The seriousness of the amnesia depends on the areas in which the brain is damaged. The second dimension is the dichotomy between forgetting the past versus an inability to form new memories. Terry, W. Scott. Learning and memory: basic principles, processes, and procedures. Boston, MA: Pearson, 2009. Print. Retrograde amnesia is not being able to remember events before the disorder. The amount of time that can't remember depends on the severity. Anterograde amnesia is the inability to form new memories, it is usually caused by severe brain trauma.

Recollecting some of life’s experiences can be done very accurately at times where we can depict details even after a long period of time. For patient H.M. this is the exact case. Patient H.M. suffered from severe anterograde amnesia. Anterograde amnesia patients quite often show normal memory for events that have occurred prior to the incident. Therefore, making it extremely difficult to recall or store information after the incident has occurred. H.M. had been knocked down by a bicycle at the age of 7, began to have minor seizures at the age of 10, and had major seizures after the age of 16. Patient H.M. had brain surgery in the early 1950’s around the time he had been 27 years old. The surgery had been done to alleviate severe symptoms

Amnesic patients tend to suffer from permanent or temporal, normally partial or total loss of episodic memory and an impaired capacity to form and store new memories. On the other hand, they seem to demonstrate unimpaired performance of behaviours involving prior experiences on tests which do not require any intentional or conscious recollection of those experiences (Schacter, 1992). Hence, most amnesic patients demonstrate the ability to retain and learn new behaviours through priming and conditioning, and acquire new perceptual, cognitive, motor and emotional skills which cannot be traced back to an explicit memory but can be demonstrated implicitly.

If the episode is associated with a traumatic event, the amnesia may clear when the individual is removed from the traumatic situation.

Amnesia is characterized as loss of memory, or the failure to recollect certainties or occasions. We have two sorts of recollections: the short-term (late, new) and long haul (remote, old) recollections. Fleeting memory is modified in a piece of the cerebrum called the worldly flap, while long haul memory is put away all through broad nerve cell systems in the transient and parietal projections. In Alzheimer's malady, fleeting memory stockpiling is harmed first.

Anterograde amnesia is a common and distinguished neurological disorder in the psychological and neurobiological field. Media depictions of this neuropsychological disorder are not as common as other forms of amnesia, but it is still incorrectly portrayed. Anterograde amnesia refers to the inability to retain new information, while still having intact memories that occurred before brain damage (Carlson, 2014). There have been several studies that focus on anterograde amnesia and its diagnostic criteria, course, associated features, assessment, etiology, underling neurobiological mechanisms, treatments, and future research.

About three months ago, during the summer, I was living a very unhealthy lifetsyle. For about a week I felt like I wasn't remembering things that should have been easily known. During normal daily activites such as work, I would tend to forget things that would have been easy to remember any other day. I've been working as a deilvry driver at a pizzeria now for close to 2 years, and I know the neighborhood extremly well. However, during this time, I was starting to forget items at the pizzeria such as sodas or where streets were, that I knew extremly well. It is for these reasons that I feel like I was experiecing symptoms of disaciative amnesia

The first point to be discussed is posthypnotic amnesia. The article “Hypnosis, Memory and Amnesia” by John Kihlstrom states that “posthypnotic amnesia is a functional amnesia, an abnormal amount of forgetting which is attributable to psychological factors rather than to brain insult, injury or disease.” Posthypnotic amnesia does not occur unless it has been specifically suggested to the subject. It can be seen as more of a temporary state of amnesia, for the amnesia can be reversed and the subject is able to remember events with out any trouble. In the movie Get Out, there is a scene where the main character, Chris Washington, gets out of bed in the middle of the night to get some fresh air. When he is on his way back up to the bedroom he

Hippocampus is a small, curved region, which exists in both hemispheres of the brain and plays a vital role in emotions, learning and acquisition of new information. It also contributes majorly to long term memory, which is permanent information stored in the brain. Although long term memory is the last information that can be forgotten, its impairment has become very common nowadays. The dysfunction is exemplified by many neurological disorders such as amnesia. There are two types of amnesia, anterograde and retrograde. Anterograde amnesia is inability in forming new information, while retrograde refers to the loss of the past memory. As suggested by Cipolotti and Bird (2006), hippocampus’s lesions are

Losing one’s memory can be a mysterious affliction, and the causes can be quite complex. Severe memory loss is introduced in author Oliver Sacks’ collection of stories The Man Who Mistook His Wife for a Hat, and lectures given by professor Jim Davies can help with understanding of some of the concepts introduced in the book. In chapter two, The Lost Mariner, the patient Jimmie is suffering from aspects of both retrograde and anterograde amnesia, which Davies explained as loss of memory of events or facts learned before an event (the event that caused the amnesia), and loss of ability to create new memories after the event, respectively. In more detail, and in relation to our book (here, the target example), retrograde amnesia would consist of any loss of memory that happened prior to an event, such as an injury or onset of disease in Jimmie’s case. Dr. Davies’ explanation of retrograde amnesia helps to understand Jimmie’s case, where in the year 1975 he is unable to recall any events after 1945. As well, the explanation of anterograde amnesia as including symptoms such as inability to form new memories, learn information or tasks, or to recall the recent past is useful when applied to Jimmie’s experience of not being able to recall events that happened even a few minutes prior. Jimmie’s suffering from both retro and anterograde amnesia, as explained by Sacks, results from Korsakov’s syndrome – a destruction of memory caused by alcoholic

The brain is dividing into several sections, including the cerebellum, the frontal lobe, and the temporal lobe, among others. The temporal lobe exists in two parts, one on each side of the brain close to the ears. It is largely responsible for the memory system (2). On the medial surface of the temporal lobe there are three important structure that are essential for human functioning. These structures are named, in order from rostral to caudal, the olfactory cortex, the amygdala, and the hippocampus. Together these three structures are referred to as the "limbic system" (1). Their functions became understood after studying how the brain functions upon loss of each structure. For example, in 1953, a patient suffering from epilepsy underwent surgery which removed most of his medial temporal lobe (1). After the surgery, the patient was able to remember who he was and was able to carry out coherent, intelligent conversations. However, if the person with whom he was talking left the room, he would have no

Case 1 tells the story of Henry Molaison (HM), a man with no memory. He lost his memory due to the operation of suctioned out the hippocampus to treat his epileptic seizures. At that time, it was not known yet that the hippocampus was essential for making memories. After operation, his seizures were significantly reduced, but Henry suffered a global amnesia. Owen et al. (2007) describes patients with global amnesia as perceptive and attentive but with a total loss of short-term memory and some trouble accessing memories of the recent past. Henry could not learn new things as he quickly forgot everything that he had learned. He could learn at a subconscious level only.