Tendonitis Case Studies

The records have been reviewed. The member is an adult male with a birth date of 05/16/1973. He has a diagnosis of bilateral degenerative myopia. His treating provider, Amy Camarota, OD, recommended treatment options on 03/18/2016, one of which was scleral lenses, which the member opted and paid for on 04/01/2016.

“Patient is currently having blurred vision, facial pain, numbness in both upper and lower extremities,

Visual field defects, defects pertaining to everything a person sees from peripheral vision to central vision, and optic disc damage is the basis of diagnosis in glaucoma (Hoskins & Kass, 1989). If the eye pressure is not within the normal range or if the optic nerve looks unusual, doctors may perform a visual field test that maps out a person's field vision, or perform a test to check whether the angle where the iris meets the cornea is opened or closed. The optic nerve and visual field changes of glaucoma

During the assessment of sensory perception, it was noted that after having Lasik surgery approximately fifteen years ago, she has had no problems with vision and has her vision checked

Vision with correction 20/30 in the right, hand motion in the left. Applanation pressure are 11 in the right and 15-16 in the left. Anterior segments are clear and quiet. ____ visual field shows a dense superior hemifield in the right and generalized constriction in the left.

Patient denies any signs or symptoms of GI bleeding, N/V/D, bloody stool, chest pain, SOB, or blurred vision. Patient was in a fight over 20 years ago and has facial bone damage, now that is affecting his vision. Requesting community resources Patient was seen at Calhoun Eye Center several years ago.

Glaucoma can occur without much warning, whether its acute from a accident in which a trauma is issued on the eye, or if it is due to age and heredity. Glaucoma doesn 't discriminate, it is seen in all races and genders. It happens so slowly with age and heredity that you don 't even notice the loss of vision until it is too late, and with acute glaucoma it can be painful with pain that radiates over the face, a headache, nausea, vomiting and seeing colored halos around lights and even blurred vision can be a few symptoms (Ignatavicius & Workman, 2013). Once you start seeing halos, and lose peripheral vision, it may be an indicator that irreversible damage to the optic nerve has happened. In this paper we will discuss the pathophysiology of glaucoma, the types of glaucoma as well as the causes for them. the issues that glaucoma can cause someone, and the treatments and interventions. We will also have a education handout to help better understand how to administer the medication, and the effects it has on the eye.

Eyes: Pupillary light reflex: Pupils are equal, round, reactive to light. Accommodation is present. Eyebrows, eyelids, and lashes are present, symmetric, and eyelids are a proximal completely. Conjunctive is pink, clear, with no swelling or lesions. Sclera is china white. Patient wears glasses and the glasses are in good repair. The last vision test was last spring and the patient gets an eye exam done every two years. The client states her left eye did not fully develop so she has always had bad vision in that one eye. However, when you look at the eye, you would never notice. Cataracts were removed in both eyes, but it was 3 years apart.

A 67 year old female Caucasian patient comes in to the clinic complaining of decreased vision, affecting the left eye more than the right. She is being treated for hypertension, cholesterol, and diabetes. Her distance vision is 20/25 OD and 20/40 OS and her near vision is 20/25 OD and 20/50 OS. Her prescription was found to be -11.00-1.00X180 OD and -13.50-1.50X015 OS, which is classified as high myopia. A 1+ nuclear sclerosis was found in the right eye. A 2+ nuclear sclerosis and a paracentral subcapsular cataract were found in the left eye. Due to these findings, it was determined that glasses could not fully fix the patients vision. This lead to a referral to a retinal specialist and a cataract surgeon. No other abnormalities were noted on the initial examination. The patient then came back when the cataract surgery was performed on the left eye and the visual acuity improved to 20/25 at distance and 20/25- at near OS. All things concerning the surgery seemed normal for a day after the IOL placement. Six months later, the

Vision without correction is 20/20 hand motion. Applanation 16 and 14. The anterior segment is clear and quiet in the right eye and there is evidence of iris neovascularization. He has a well placed PC IOL in the right eye. The left eye shows 2+ nuclear sclerosis with 2+ cortical spoking. Posterior segment of the left eye shows a dense vitreous hemorrhage with a positive red reflex. No evidence of retinal detail.

Right pupil reactive to light. Mucus membranes moist. Neck supple. No JVD, LAD. Questionable right bruits. Extraocular muscles intact.

A 42 years’ male attend to the anterior segment clinic in at prince sultan military medical city for a routine eye examination. He was complained from blurry distance vision and difficulty during driving at night due to glare from oncoming headlights. Decreased vision was more noticeable in his right eye than in his left eye.

A 6-year-old male underwent an initial eye examination for evaluation of hyperpigmentation of both eyes at 1 year of age. He was diagnosed with benign conjunctival melanosis. He was also diagnosed with an increased cup-to-disc ratio and mildly elevated intraocular pressures in each eye. Intraocular pressure was maintained in the mid-teens bilaterally on topical glaucoma monotherapy. At 4 years of age, he relocated. When he was 5-years-old, he underwent bilateral glaucoma shunt placement elsewhere The operative reports and glaucoma drainage implant (GDI) specifics were not obtainable. No prior or subsequent ocular surgeries were performed. He returned to our care after 2 years. His eye pressures were controlled without glaucoma medications. There were GDIs in the superotemporal quadrant of each eye. In the right eye, there was corneal edema in the superotemporal quadrant extending to the pupillary margin and a 3 millimeter central subcapsular cataract. The shunt tube extended into the anterior chamber approximately 2 millimeters (Figure 1A). The anterior portion of the tube retracted posterior and was barely visible in the anterior chamber with adduction of the eye (Figure 1B). Upon abduction, the tip of the tube travelled up to 6 mm towards the 4:00 position (Figure 2A) (Video). Intermittently, the tip of the tube would dive posterior to the iris at the 4:00 position to touch the lens (Figure 2B).

The prevalence has been calculated to be between 0.06% and 0.1%. A nanophthalmic eye is normal in shape but unusually small, with a shortened axial length (<20 mm), a small corneal diameter and a relatively large lens for the volume of eye. Other findings in nanophthalmic eyes include a shallow anterior chamber both centrally and peripherally, an anteriorly displaced iris, high hyperopia, thick sclera, spontaneous and postoperative uveal effusions and a wide pulse pressure. A subset of patients with familial, recessive nanophthalmos also have a pigmented retinal degeneration. Spontaneous choroidal effusions can occur that may induce angle closure glaucoma. Pupillary block related to age and to lens swelling is a factor in some. The effusion rotates the ciliary body anteriorly, displaces the peripheral iris, and loosens the zonules, allowing the lens to move

Results: The uncorrected inferior RNFL thickness showed the highest correlation with AL (r = - 0.81, r2 = 0.66, p 21 mm Hg in either eye; those with a history of ocular trauma, intraocular or refractive surgery, or any ocular or neurologic disease that could have affected the optic nerve head or RNFL or could cause a visual disturbance; those who showed a closed or occludable angle by gonioscopy; and those whose RNFL or optic discs indicated an RNFL defect or a disc anomaly by fundus examination.