The Effects Of Deep Breathing On Heart Rate And Blood Pressure Essay

Before performing this experiment we hypothesised that breathing exercises would cause the heart to relax, causing a decrease in heart rate and blood pressure. In addition, we predicted that the systolic pressure would decrease and the diastolic pressure would stay similar to the control test. Due to all of the three t-tests exceeding .05 (0.64659, 0.380067, 0.184003) the null hypothesis was accepted. The data didn’t show a significant change from the basal readings to the treatment readings so we failed to reject the null hypothesis.

All tests were conducted in the same posture; leaning over the lab table with elbows resting on the table and the head down. Students worked in groups of three to four, and each one took turn being the experimental subject, taking the pulse and handling the stopwatch. To measure the radial pulse manually, the subject’s palm was facing upward. The index and middle fingers were used to locate the pulse between the radial bone, which is on the same side as the thumb, and with a slight pressure, the pulse could be found.

The purpose of arterial pressure and the pulse lab is to determine the effect of posture and exercise on systolic and diastolic pressure and the heart rate. And also in order to find the differences in the reading taken under these condition compares to the baseline reading. The Sphygmomanometer and stethoscope are used to measure the systolic and diastolic blood pressure, counting the beat on the radial artery will give the reading for pulse rate and by using the lab scribe software and IWX214, the blood pressure will be measured. In the heart, the aorta and the carotid arteries have baroreceptors and the chemoreceptors that identify the changes in arterial pressure and the changes in

The heart rates of participants was tested before the step test, one minute, two minutes, and three minutes after the step test was performed in this experiment. Since heart rate increases while someone is performing physical activity, it was expected that heart rates of the students would be higher than before the step

In healthy individuals, heart rate can be lowered 2 to 5 beats per minute during short-term relaxation interventions (Paul 1969; Melville, Chang, Colagiuri, Marshall, and Cheema 2011; Wallace, Benson, and Wilson 1971; Schandler and Grings 1976). Relaxation interventions can lower blood pressure 6 mmHG to 14 mmHG in systolic blood pressure (Melville et al 2011; Schandler and Grings 1976) and respiration rate can be lowered by 2 to 4 breaths per minute in healthy patients (Benson 1993; Paul 1969; Melville et al 2011; Wallace et al 1971).

Comparing my Rebreathing Carbon Dioxide graph to my peers, I noticed that we all share an increase in both the breathing rate and depth. Talking to my peers, I noticed that we all mutually had the pressure of the pressure cuff go down once we finished this experiment. This release of pressure most likely occurred because during the different intervals – breathing normally, holding our breaths, slowly breathing, and breathing through the Ziploc bag – the kPa, or unit of pressure, gradually decreased. The relationship between breathing rate and breathing depth is that they strongly influence HRV, or heart rate variability. This is most efficient because the tidal volume decreases as heart rate does too, although the changes are insignificant.

The baseline value of this experiment was the PO2, PCO2 and Hb-O2 saturation during normal breathing. These baseline value (table 1) allow the comparison between different breathing patterns to normal breathing. The results (Figure 1, 2, 3 & table 1) of this experiment shows that breath-hold compare to normally breathing, mean PO2 decrease, mean PCO2 increase (figure 1) and mean Hb-O2 saturation slightly decrease (Figure 2). Moreover, comparing p values of breath-hold to breath-hold after hyperventilation, which is less than 0.05, meaning there is significant changes in mean PO2 and PCO2 (table 2), mean PO2 decrease, mean PCO2 increase (figure 1), and mean oxygen haemoglobin saturation increase (figure 2), the mean duration of breath hold decrease

Determining the Effect of Arousal on the Heart Rate and Blood Pressure of Adolescent Males

Figure 1. Compared effects of breathing in air (control treatment) and a simulated dive (apnea in 10C water) on human heart rate (n=6). Bars represent the combined mean heart rate of the subjects for both treatments over each measurement period. Error bars represent 95% CI.

The effects of exercise on blood pressure, heart rate, respiration rate and electrical activity of the heart were assessed. The measurements of respiration rate, pulse rate and blood pressures were noted as described in Harris-Haller (2016). Data was first taken from subjects in a relaxed position and then followed by sets of reading after exercising based on one minute intervals. The data also noted sitting ECG traces from Harris-Haller (2016). The respiratory rate, pulse, blood pressure, P wave, QRS complex and T wave were defined for each subject. The class average was calculated for males and females and graphed to illustrate the results by gender for each cardiopulmonary factor.

This experiment was conducted to see how exercise affects breathing and heart rates. My hypothesis was that if exercise puts strain on the body, then the heart and breathing rates will increase and I was correct. This is because as the exercise increased the heart and breathing rates also increased. For example in trial #1 the resting heart rate was 96 beats per minute then after 1 minute of exercise it increased to 144 beats per minute and then increased again to 160 beats per minute after 4 minutes of exercise. After the exercise was completed the heart rate went back down to 132 beats showing that the heart rate is based off of the amount of exercise rather than it just going increasing or decreasing randomly. The breathing rate also increased

As the intensity of exercise increased, so did the rates of the heart and breathing. After a small period of rest, the heart rate and breathing rate both decreased to a point close to their resting rate. This proved the stated hypothesis. First, the hearts average resting rate was recorded to be 76 bpm. The heart is therefore transporting oxygen and removing carbon dioxide at a reasonably steady rate via the blood. During the low intensity exercise (Slow 20) the heart rate increases to 107 bpm, which further increases to 130bpm at a higher intensity level (Fast 20). The heart therefore needs to beat faster to increase the speed at which oxygen is carried to the cells and the rate at which carbon dioxide is taken away by the blood.

The results obtained for this experiment were somewhat expected as the results had reflected to the theory. While the composition of oxygen in air remains constant only the atmospheric pressure to decrease with altitude. The respiration pattern should have shown a more frequent incidence of trough and crest, but the amplitude decreased with the increasing elevation, as the oxygen molecules contained in each breathe are low. Since less oxygen are presented the oxygen and carbon dioxide should decrease with increasing altitude. Equally since more breathe are taken to allow the body to be oxygenated, the heart would need to work faster and to allow such occurrence. These ideas correlates to the results obtained, as with increasing elevation the heart rate increased from 88 beat/min to 104 beat/min at rest and the 19.8% to 18.0%, 147711.6ppm to 130739.6ppm, oxygen and carbon dioxide respectively, when condition had remained the same (pre exercise). As one exercised, regardless of the altitude, component such as heart rate carbon dioxide should have increased and that oxygen and breathing pattern to decrease. This is because the heart will need to pump more blood and use more oxygen thus increasing the production of carbon dioxide. Additionally the body would act to the oxygen deficiency, hence resulting in a decrease in breathing pressure. These ideas can be correlate to the results as on table, as an exercise was performed the heart rate had increased to 109 beats/min, the

The main purpose of this experiment, was to demonstrate and understand the effect of breath-holding on the heart rate (apnoea).

We decided to measure the subject’s pulse and respiratory rates whilst sitting down because there would be no additional stress on their heart, which would increase their heart rate. Their heart rate should also return to its resting heart rate due to the decrease of muscle use.