The Stages Of Diabetes Mellitus And Hypothyroidism

The Consultation Report dated 12/07/2016, indicated that the claimant presented with uncontrolled type 2 diabetes mellitus. He also had a chronic back pain, arthritis, and a history of diabetic neuropathy. His blood sugar was 800 (critical stage). His blood pressure was 129/62 mmHg. The physical examination revealed mild thyromegaly, a decreased air entry at the base of the lungs, and a decreased peripheral palpable pulse. The exam also revealed loss of hair and chronic ischemic changes. Toe deformity and peripheral deformity were noted. He was on Levemir and Novolog. According to the provider, the claimant was on steroids, which has caused an increased blood sugar level. HBA1c

The proband’s family history consist of both type 1 and type 2 diabetes; especially the females within her maternal linage. Family histories where type 1 and type 2 diabetes co-occur happens often (Nogueira, 2013). Both types of diabetes are caused by a loss of physical or functional beta cell mass (Nogueira, 2013). Type 1 diabetes (T1D) is due to an autoimmune process and type 2 diabetes (T2D)

The wife reported Mr. P was diagnosed with type 1 diabetes mellitus 6 months ago. Additionally, he had the flu for one week with increased vomiting and anorexia and stopped taking insulin three days ago because he was unable to eat.

History of present illness: 50 year old African American female presents to the clinic today to follow up on her Diabetes. Patient diagnosed with Diabetes in 2000. Last Diabetes checkup three months ago. Patient reports that she takes all of her medications as prescribed. Patient is currently on metformin and Lipitor. Patient denies any episodes of hypoglycemia. Patient denies experiencing symptoms of polyuria, polydipsia, and polyphagia. Patient reports that she has been checking her

Diabetes Mellitus (DM) or Type 2 Diabetes is seen as a metabolic disease that is categorized by abnormally high blood glucose or hyperglycemia. Diabetes Mellitus is also formerly known as noninsulin-dependent diabetes mellitus and is the most common form of diabetes that is seen. Insulin is a hormone that is supplied to the body that allows us to efficiently use glucose as fuel. When carbohydrates are broken down into sugars in the stomach glucose enters the blood circulation simulating the pancreas to release insulin in an appropriate amount to become used for energy. With diabetes mellitus the body does not properly make use of the insulin supplied for the body. This causes the pancreas to produced an extra amount if insulin which the body cannot keep up with, causing an imbalance to the blood glucose levels (American Diabetes Association, 2015). In the united states diabetes affects almost 29.1 million people, while the another 86 million people have pre-diabetes but do not know. It is also known as the 7th leading cause of death in the country in the recent years (MedicineNet.com, 2016). For a patient suffering from a chronic form of diabetes mellitus understanding how these mechanisms lead to the condition can be used as preventative measures. Potential consequences as well as the causes and clinical manifestations will ensure a better knowledge on the issue to monitor the condition.

Diabetes mellitus type 2 is an endocrine disorder that causes impaired use of carbohydrates while enhancing the use of proteins and lipids. This is called insulin resistance, in which the pancreas cannot make enough insulin to keep blood glucose levels normal, or the body is unable to use what is produced. The impairment causes blood glucose level to rise higher than normal. There is no cure for type 2 diabetes mellitus and it is life threatening when left untreated. Signs and symptoms of this disorder include vision changes, increased thirst, increased hunger, increased frequency of urination, stomach pain, nausea and vomiting, erectile dysfunction, and absences of mentruation. These can occur abruptly, or over a long period of time.

Type 1 diabetes is a chronic illness characterized by the body’s inability to produce insulin. Onset most often occurs in childhood, but the disease can also develop in adults in their late 30s and early 40s. The classic symptom of type 1 diabetes is: The need to urinate frequently, excessive drinking as a result of thirst, excessive hunger and abnormally large intake of solids by mouth. Unexplained weight loss, Other symptoms may include fatigue, nausea, and blurred vision. The onset of symptomatic disease may be sudden and is a classic sign of diabetes mellitus that is under poor control or not under treatment

Objective She is currently on sliding scale insulin, 1200 Kcal diet, and Captopril 50 mg orally twice daily. She has high random blood glucose of 325 mg/dL. Her vital signs are generally normal except for BP (150/97mmhg). Physical exam revealed dry nasal and throat mucus membranes and mild cool leg edema. She is overweight with a BMI of 31kg/m2.

Reason for Referral: Mrs. R was misdiagnosed with Type two diabetes a few years ago. She was re-tested and found to have Type one. The misdiagnosis by her general practitioner (GP) caused her to lose weight, become anemic, depressed, and unable to sleep as a result of taking the wrong medications. It has taken her two-and-a-half-months to have an appointment with an endocrinologist to find out she was misdiagnosed. She has been diabetic for at least 9 years.

Diabetes is a very common chronic medial disorder and expected to be a big medical challenge of the twenty first century (Clark, 2004). It is a condition, in which the glucose level in the blood becomes so high that the body is unable to utilize it properly. This long-term condition results when the pancreas of the victim is either not able to produce enough insulin (sometimes even cannot produce any insulin) or the insulin produced cannot work properly. Diabetes is divided into two main types: Type 1 and Type 2. Type 1, also known as insulin dependent diabetes milletus (Masharani, 2008) cannot be prevent while Type 2 can be prevented but if the patient is at pre-diabetes stage.

Tina does not regularly monitor her blood sugars, and has stopped taking Metformin the medication that stimulates the pancreas to make more insulin. Tina's present condition of uncontrolled Diabetes Mellitus, manifested by a blood glucose level of 238, combined with her poor dietary habits, and sedimentary lifestyle

Rowena’s has a history of DM type II, which is also known as Non-insulin dependent (NIDDM) and occurs as a result of the inability of the pancreas to produce enough insulin or the body cells not able to absorb and utilise the insulin produced (American Diabetes Association, 2009). It has a late onset, usually from the age of 45years. The main cause of this condition is unknown. However, two types of risk factors which are, the non-modifiable risk factors, for example, aging, genetic, and familial and the modifiable risk factors which include, sedentary lifestyle, alcoholism, smoking, poor diet, lack of exercise, and obesity) could be associated with the condition.

The disease I chose is one that is known for it's low production of necessary hormones because of an underactive thyroid gland. Hypothyroidism usually results from some other disease in the same area such as autoimmune disease, radiation therapy or some medications like lithium. It is also most often seen in women over the age of 60. Chemically what is happening is your thyroid stops producing the hormones that the body needs to work properly which causes your metabolism to slow down. The symptoms of hypothyroidism are very slow to produce but over a number of years would develop into impaired memory, depression, loss of hair, fatigue and a variety of other symptoms. The diagnosis of this disease has progressed since the invention of the TSH

When Y.L.’s laboratory test results were analyzed, it was shown that she negatively exceeded the optimal range of each diagnostic test that was performed. Y.L.’s fasting glucose showed as 184 mg/dL, which was the first indication that she was diabetic, based on scientific testing that has proven that any amount over 126 mg/dL points to diabetes. Her HbA1C was 8.8%, another marker for DM (diabetes mellitus). Studies have shown that any level over 6.5% is a sign of being diabetic. An increase in total cholesterol, triglycerides, and LDL, and a decrease in HDL, although not definitive markers, can be underlying signs of diabetes in a patient; this is also known as diabetic dyslipidemia. Our patient’s total cholesterol was 256 mg/dL; anything over 225 mg/dL is considered high. The test showed that her triglycerides were 346 mg/dL (anything over 200 mg/dL is thought to be high); her LDL was 155 mg/dL, with anything from 120-159 mg/dL being deemed to be

The tests came back and nothing seemed to stick out. Her glucose level was slightly elevated but not enough to cause the lethargia and inappetence she was exhibiting. Dr. Brian Stockinger told the keepers to keep an eye on her and watch for developing signs or signs of improvement. A few days later, the keepers had noted in the reports that she has