Periodontal Disease Essay

Any of these indications may flag a significant issue, which ought to be checked by a dental practitioner. At your dental visit: The dental specialist will get some information about your medicinal history to recognize fundamental conditions or hazard elements, (for example, smoking) that may add to periodontal illness. The dental practitioner or hygienist will inspect your gums and note any indications of irritation. The dental specialist or hygienist will utilize a little ruler called a "test" to check for periodontal pockets and to gauge any pockets. In a sound mouth, the profundity of these pockets is more often than not somewhere around 1 and 3 millimeters. The dental practitioner or hygienist may take a X-beam to see whether there is any bone misfortune, and

     Periodontal disease is more commonly known as gum disease or gingivitis. This infection is serious enough, that it can lead to tooth loss if left untreated. This chronic infection starts around the tooth and it affects the supporting bone and gums. Periodontal disease can affect anywhere from one tooth to all thirty-two teeth. The disease pathology starts with the plaque that builds up on your teeth everyday.

My job shadowing experience with Dr. D. Lance Taylor has helped me in learning about the quality of education and intensive clinical training conducted at the SIU School of Dental Medicine. In addition, the clinical certification course will aid in enhancing my knowledge and developing skills required to make a smooth transition into the class. More importantly, opportunities to serve at the Give Kids a Smile and Mission of Mercy clinics will help in continuing my passion for community service. Also, it will be a great opportunity to learn from the ongoing clinical research at the school and improve my critical thinking. I am particularly interested in the research of Dr. Nathalia Garcia about the role of biomarkers in the progression of periodontal

The most important cariogenic bacteria are Streptococcus mutans. Other cariogenic bacteria include S. salivarius and S. sanguis as well as various species of Eikenellan and Lactobacillus. Dental caries is a disease of the tooth enamel and the tooth itself, while periodontal disease is a combination of gum swelling and erosion of the ligaments and bone that supports the teeth. Mildest form of periodontal disease is gingivitis, while the most severe form is called acute necrotizing ulcerative gingivitis also known as trench mouth. Poor oral hygiene, unhealthy diet, alcohol and tobacco use may constitute in the development of dental caries and periodontal

From the Gothenburg group, Lindhe et al., carried out a 14 year follow up of 61 out of initially 75 patients after active treatment of advanced periodontal disease, being defined as

As stated by the Center for Disease Control and Prevention, “Each year, more than 30,000 new cases of cancer of the oral cavity and pharynx are diagnosed and over 8,000 deaths due to oral cancer occur.” (CDC). Most adults are unaware that they have periodontal disease or gum disease, this occurs because they do not maintain routine checkups with their dentist. When we insert food and other bacteria into our mouths, it affects our teeth and gums. We need routine dental checkups and cleanings throughout our lives. Dental checkups usually involve x-rays, cleanings, and the examination of the mouth.

For example, a patient that does not live in a fluoridated community, has previous caries, and signs of xerostomia would be diagnosed as a high risk for caries. Another example, a patient that uses tobacco would be diagnosed as a high risk for oral cancer. The periodontal risk assessment is used to determine the risk factor for periodontal disease based on the findings during the assessment. For example, if a patient has irregular visits to a dentist, lack of brushing and flossing, and has signs of xerostomia the patient would be diagnosed as a high risk for periodontal

Patients were selected according to the following inclusion criteria: 1) no periodontal treatment within the previous 12 months; 2) no use of antibiotics within the previous 6 months; 3) no systemic disease that could influence the outcome or progression of periodontal therapy; 4) no use of anti-inflammatory drugs within the past 3 months; 5) no pregnancy; 6) no use of hormonal contraceptives; and 7) no smokers, alcohol drinkers, or drug users. Patients with <16 teeth, partial dentures, or fixed prosthodontic appliances were excluded from the study. Teeth with mobility grade III or periodontal pockets deeper than 10 mm in the studied areas were not evaluated. All selected patients signed an informed consent form before undergoing the research

Some studies have been conducted but the World Workshop on Periodontics stated that controlled clinical trials that evaluated the role that occlusion had on the progression of periodontal disease in humans, was unethical. To avoid unethical situations, patient records from a private practice facility were collected and studied to see if there was a connection between occlusal discrepancies and the progression of periodontal disease. The records that were studied were from patients that had periodontal evaluations as well as occlusal assessments. All of the patients studied had periodontal disease but only some of them had occlusal decencies. After a twelve month period some patients returned and had another periodontal evaluation and occlusal assessment and the data was compared to the data that was collected twelve months prior. The data collected was compared. Patients without occlusal discrepancies and patients with occlusal discrepancies both had worsening periodontal disease after twelve months of no treatment but, the progression of periodontal disease and increased pocket probing depths

Herpesvirus infections induce a proinflammatory response including expression of cytokines and chemokines. In periodontitis, herpesvirus-induced expression of cytokines is particularly intriguing. HCMV infection can up-regulate interleukin-1β and tumor necrosis factor alpha gene expression of monocytes and macrophage. In turn, interleukin-1β and tumor necrosis factor alpha may up-regulate matrix metalloproteinase, down-regulate tissue inhibitors of metalloproteinase, and mediate periodontal bone destruction. Increased production of these proinflammatory cytokines by macrophages and monocytes has been associated with enhanced susceptibility to destructive periodontal disease. EBV may act as a potent polyclonal B-lymphocyte activator, capable of inducing proliferation and differentiation of immunoglobulin-secreting cells, features associated with periodontal disease progression. Active EBV infection can also generate antineutrophil antibodies and neutropenia that may lead to increased bacterial pathogenicity and

Gingivitis is a dental disorder characterized by the inflammation of the gums. It occurs after films of bacteria accumulate around the teeth (plaque induced gingivitis). Gingivitis is one of the non-destructive types of periodontal diseases. Gingivitis could worsen and progress to periodontitis if left un-attended. Periodontitis is worse and could lead to the loss of your tooth. Individuals with gingivitis have puffy and red gums and bleed once they brush their teeth. Gingivitis can be solved with proper dental hygiene- flossing, as well as more frequent and longer brushing. In mild cases of the diseases, individuals may not realize that they have it since the symptoms

Bacterial plaque that forms on your teeth and around your gums is the culprit that causes periodontal disease. When plaque accumulates on your teeth and around your gums, they soon become inflamed, swollen, and bleed

1) Researchers have compared gingival cervicular fluid (GCF) LL-37 levels between smokers and non-smokers (16 smokers, 15 non-smokers) and came up with that smokers might have a highly possible surgical treatment because , by the time , the levels of the (GCF) LL-37 will be a highly effective factor in chronic periodontitis, whereas non-smokers might have a non-surgical treatment.( Does smoking affect gingival crevicular fluid LL-37 levels following non-surgical periodontal treatment in chronic periodontitis?. Türkoğlu O1, Eren G2. , 2016 Jan.NCBI.)

In the research article “Effect of nonsurgical periodontal treatment in conjunction with either systemic administration of amoxicillin and metronidazole or additional photodynamic therapy on the concentration of matrix metalloprotrinases 8 and 9 in gingival crevicular fluid in patient 's with aggressive periodontitis,” is used to

METHODS: Ten cases with clinical and radiological features suggestive of Aggressive Periodontitis are discussed in this paper. In two cases, anaerobic microbial analysis was carried out and a PCR was done.