Therapeutic Disorders: A Case Study

The first type of anemia is macrocytic-normochromic. Macrocytic-Normochromic anemia occurs when large stem cells derive into erythrocytes. The erythrocytes are problematic because they also end up being large in thickness and volume. This type of anemia is considered normochromic because the hemoglobin remains its normal size. Macrocytic anemia will happen if DNA is synthesized incorrectly. The DNA synthesizes incorrectly due to a deficiency in either vitamin B12 of folic acid. If there is a deficiency in vitamin B12 then it is considered pernicious anemia, and if there is a deficiency in folic acid then it is considered folate deficiency anemia. The enlarged erythrocytes die off

Anemia is a disorder of the blood. It occurs when your body does not produce enough erythrocytes or red blood cells (RBCs). Without the erythrocytes oxygen can not be adequately delivered to the tissues and organs throughout the body. This will cause you to become weak and tired. A person may also experience headaches, skin pallor, and faintness. Your body may attempt to compensate for these symptoms by speeding up the heart rate and respiratory rate. This is the body’s attempt to return oxygen levels to normal(Thibodeau and Patton, 2005).

According to the information of Nikoleta’s case, Cooley’s anemia has features that the beta proteins of hemoglobin do not function properly. This is caused my mutations in the beta chain of the hemoglobin molecule. The symptoms of Cooley’s anemia matches the symptoms of Nikoleta. The main reason why giving a high iron formula did the opposite is that the constant buildup of iron causes the symptoms to grow worse, and can even result in heart failure in the future. Cooley’s anemia decrease the red blood cell’s ability to deliver oxygen to certain tissues. The number of red blood cells are high, however, the number of hemoglobin is very

As a provider, one will care for many patients that have different types of anemia. Anemia is not so much as a disease as a symptom of an underlying issue. Although there can be particular signs and symptoms associated with anemia, the basis of a diagnosis is from laboratory data. For the purpose of this discussion, I will evaluate a case study and give a differential diagnosis. I will also assess how patient history, physical exam, and lab reports support my diagnosis. I will explain the pathophysiology of the type of anemia and give causes and treatment options available.

Based on Ms A’s preliminary worked she mostly likely has microcytic anemia or iron deficient anemia. The symptoms of microcytic anemia and iron deficient anemia are synonymous, making them difficult to differentiate

The type of anemia this patient has is Iron Deficiency Anemia. Iron Deficiency Anemia (IDA) has been revealed to be the greatest mutual foundation of anemia globally, and it is acknowledged that people with chronic kidney disease (CKD) progress to anemia as their kidney function deteriorates (Wittwer, 2013).

Dalia and I reviewed her labs from 06/11 and advised the patient they were normal and iron was not needed. The patient advise us she had a new set of labs done on Wednesday. We advise that those labs were not in the system and once they appear in the system, we will give them to you ,so you can determine if she needs iron.

also had a complete blood count, a blood test, as a baseline to assess his overall health. The complete blood count is “To evaluate numerous conditions involving red blood cells, white blood cells, and platelets. This test is also used to indicate inflammation, infection, and response to chemotherapy (Bladh et al., 2013).” T.A.’s abnormal blood results were as follows: RBC 3.3 10^6 cells/microL (low), hemoglobin 11.0 g/dL (low), hematocrit 33.2% (low), and mean corpuscular volume 99.7 fL (high). The normal range for RBC should be 3.8-5.81 10^6 cells/microL. Hemoglobin and hematocrit levels should be 12.6-17.4 g/dL and 36-52%. Normal mean corpuscular volume should be 79-103 fL. T.A.’s abnormal lab values indicate anemia. A decrease in red blood cell is related to nutrient deficiency such as a deficiency in iron or vitamins that are needed for red blood cell production and maturation. The decrease production or maturation of red blood cells lead to anemia in the patient. A low hemoglobin is associated with an overall decrease of red blood cell count and anemia caused by nutritional deficit of iron, vitamins, and folate. It can also be low due to a decrease level of erythropoietin caused by the AKI. Like hemoglobin, a low hematocrit is associated with an overall decrease of red blood cell count due to anemia, nutrition deficiency, and AKI. T.A.’s increased mean corpuscular volume is related to anemia caused by vitamin B12 or folate deficiency (Bladh et al.,

Pernicious anemia due to decreased B12 affects the hematopoietic,gastrointestinal and nervous systems of the body. The hematopoietic system is affected in that, B12 is essential for the formation of red blood cells, therefore without adequate B12, there is a reduction in the production of red blood cells causing abnormally large shaped red blood cells to be produced. Red blood cells carry oxygen , therefore the NP will anticipate patients with anemia to have symptoms like dizziness, fatigue, pallor, tinnitus, cardiomegaly with murmur, tachycardia, and chest pain. Additionally, symptoms of the affected gastrointestinal system include anorexia, weight loss, abdominal cramping, weight loss, and glossitis. Moreover, symptoms of the affected

MDD is a disorder that affects a significant number of people, and is difficult to treat effectively

The patient is an 84-year-old female who did stop her iron supplement, which she was taking for a history of iron deficiency anemia thought related to a Mallory-Weiss tear, which was seen on an EGD done in February 2015. Her hemoglobin and hematocrit had been stable. Because of her symptoms, I did ask her to stop the iron supplement. She does tell me her abdominal pain, increased stooling did go away, since that time, although, she does state today that several weeks ago she did have one further episode of it, but none since that time.

This 72-year-old white male with progressive microscopic iron deficiency anemia on whom I asked to consult on. The patient is asymptomatic from the GI perspective. He has a history of reflux but this is controlled with omeprazole. There is no abdominal pain, change in bowel habits, weight loss or visible blood per rectum. He has mental forgetfulness and is not a good historian. He also has an active right inguinal hernia that is undergoing evaluation for possible hernia repair.

Anemia is a condition that is characterized by a lack of red blood cells or hemoglobin (a polypeptide). The focus of this report will be on the lack of hemoglobin as it is the most prevalent case in most anaemic patients. The protein molecule is found in red blood cells and carries oxygen from the lungs to the body's tissues and returns carbon dioxide from the tissues back to the lungs. It is composed of four protein chains, two alpha chains and two beta chains, each with a ring-like heme group containing an iron atom. The aspects of anemia and hemoglobin that will be examined in this report are; the synthesis of hemoglobin, the structure of hemoglobin (secondary, tertiary, quaternary), the effect of anemia caused by Crohns on absorption,

Aplastic Anemia is a rare disease in which a person’s bone marrow fails to produce new blood cells. Bone marrow is the spongy tissue found in some of our bones and is responsible for blood cell formation, hematopoiesis. Our bone marrow is given the task to generate red blood cells, which carry oxygen, white blood cells, which fight foreign entities, and platelets, which are responsible for blood clotting. In children, bone marrow is found in the long bones such as the femur, and in adults it is mostly found in flat bones such as the sternum and pelvic girdle. Patients who are diagnosed with Aplastic Anemia are deficient in all three blood cells, due to damage to

My topic for this paper is aplastic anemia. I intend to discuss the definition of aplastic anemia, pathophysiology of the disease, statistics, signs and symptoms, diagnoses, treatment, and prognosis.