Type 1 Diabetes

Type 1 is characterized by the body’s inability to produce insulin. It is caused by autoimmune-mediated destruction of pancreatic beta cells, which are responsible for producing insulin. There appears to be a hereditary link in people with Type 1 diabetes. Other factors have been known to cause Type 1 diabetes such as viral infections, toxins, and other environmental factors. Type 1 diabetes is the rare form, affecting about 10% of the diabetes population. Its onset usually occurs in people less than the age of 20.

Type one diabetes is an autoimmune disease. There are cells in the pancreas that produce insulin, the hormone that regulates the amount of insulin in your blood and

Type 1 diabetes forms when the insulin forming cells in the pancreas are destroyed. Normally, the glucose you obtain from your food stimulates the pancreas to release insulin into into the blood. This insulin is used to help secrete and transport the glucose molecules

The Risk factors for type 1 diabetes are not as clear as type 2 but having a parent, brother, or sister with type 1 diabetes you at risk of developing type 1 diabetes,

Type 1 diabetes is a serious chronic condition that tends to arise prior to adulthood. The disease requires substantial lifestyle changes in order to cope, and can lead to several debilitating outcomes if left unchecked. According to the American Diabetes Association ([ADA], 2017), Type 1 diabetes is defined as a chronic condition where the body no longer produces the insulin hormone, and is therefore unable to utilize and store glucose. As a result, individuals with Type 1 diabetes may experience excessively high or low blood glucose levels: hyperglycemia and hypoglycemia respectively (ADA, 2017). Both sides of the spectrum can have devastating effects on the body’s cardiovascular and renal activities, in addition to deteriorating the peripheral

As of yet, the origins of diabetes are still unknown, however, for type 1 diabetes,

The islets of Langerhans also produce glucagon, another hormone which affects blood glucose levels. Glucagon is produced by the alpha

Complication of Type 1 Diabetes Mellitus (T1DM) can develop gradually over the decades and may be disabling or even-life-threatening. T1DM can affect multi-organ system including the heart, blood vessels, nerves, eyes and kidneys. Due to poor blood sugar level control and multiple episodes of hospital admission due to hyperglycemia and DKA, KK might be at risk of developing these sorts of complications. Thus a good blood sugar management and early intervention is necessary in order to prevent or delayed this kind of complications from happening in the future.

Diabetes mellitus (DM) is a group of metabolic diseases characterized by hyperglycemia resulting from defects in insulin secretion, insulin action or both. The chronic hyperglycemia of diabetes is associated with long term damage, dysfunction, and failure of various organs especially the eyes, kidneys, nerves, heart, and blood vessels. Type 1 DM once known as juvenile diabetes or Insulin dependent diabetes, is a chronic condition in which the pancreas produces little or no insulin. Although type 1 diabetes can develop at any age, it typically appears during childhood or adolescence (1).

Patients with Diabetes have very comprehensive learning needs. The learning needs arefocused on managing their glucose levels and preventing complications of diabetes. Learningneeds for managing diabetes are complex and include: monitoring blood glucose levels,menu/food planning, exercise, medications, skin care, management of co-existing disease processes, knowledge of medications, knowledge of the disease process and how to managehypo or hyperglycemic episodes. Many patients are diagnosed with diabetes every year andmany are unaware that it requires lifestyle changes, especially in the areas of nutrition and

Type 1 diabetes is an autoimmune disease that begins when the pancreas quits making insulin. Insulin plays a key role by letting glucose enter the body’s cells, and then uses it for energy. When the body doesn’t get the

Special cells in the pancreas produce a hormone called insulin to regulate metabolism. When this hormone is not present glucose cannot enter body cells and blood glucose levels rise. Hyperglycemia may result from this situation. This process is the development of diabetes mellitus. Type 1 diabetes, formerly called juvenile diabetes or insulin dependent diabetes mellitus, has two forms; immune-mediated and idiopathic. Type 1 diabetes accounts for 5-10% of cases in the United States. Research has shown that there is an inherited tendency for developing the disease; it may be triggered by environmental factors (Rosdahl & Kowalski, 2008). It also has an autoimmune component since antibodies to insulin and islet cells are present at the time of

Occurring as a multisystem disease of the metabolism of proteins, fats and carbohydrates due to the lack of insulin, type 1 diabetes (type 1 DM) has both anatomic and biochemical consequences. The prevalence of type 1 DM is across all ages but usually seen more in children and young adults around puberty. Out of the total population of persons with diabetes, only five percent suffer this form. The body fails to produce insulin, which is

Insulin is a significant hormone which regulates glucose homeostasis by respond to target cells such as muscle cell, adipose tissue or liver. Insulin is entirely secreted by the β-cells of the islets of Langerhans (identified by Paul Langerhans in 1869) which are the areas in pancreas stored some endocrine cells. Most of the islet mass are β-cells, and there are the pancreatic islets contain α-cells that secret glucagon, δ-cells that secret somatostatin as well, and all of these located in the islet periphery cells where produce pancreatic polypeptide. The molecular mechanisms illuminated by which these cell types are created constitutes a topic of intense study and has important implications for our understanding of the etiopathological processes

These two hormones work hand in hand to balance the body’s glucose levels. In normal function, the pancreas secretes insulin in response to the introduction of glucose into the body and to meet basal metabolic needs. In type 1 diabetes, there is a deficiency of the hormone insulin, which can have many contributing factors. There is no one cause for this disease. However, one cause stems from the body’s immune response. The immune mediated response of type 1 diabetes is an, “autoimmunity directed against pancreatic islet cells [that] results in slowly progressing beta-cell destruction, culminating over years in clinically manifested insulin-dependent diabetes mellitus” (Krishna & Srikanta, 2015). In short, an individual’s pancreatic beta cells begin to slowly die off which obviously affects the feedback mechanism that is regulating blood glucose concentration. Thus, it makes an individual rely on mechanically introduced insulin.