Type 1 Diabetes Pathophysiology

Type 1 diabetes is well known disease, some of us or someone we know are the victim of this chronic illness. There are controversial explanation such as, genetic susceptibility and in contrary, environmental factors that are viral infection, prenatal and neonatal influence, nitrate in drinking water, (Norris et al, 2003) early exposure to cow’s milk towards why the immune system destroy the insulin producing beta cell. In this essay, I will be discussing about the pathophysiology of type 1 diabetes and how it affect the homeostasis of our normal functioning body. In subject to Carol, I will be explaining the signs and symptoms of the illness and also the possible effects to her developing foetus and herself.

Type 1 Diabetes effects millions of people around the world (Michel & Montada-Atin, 2014). Most often, a person is diagnosed before their 30th birthday, with diagnosis commonly being between 11 and 13 years of age (Michel & Montada-Atin, 2014). Diabetes is a disease of the pancreas. More specifically, the breakdown of β cells in the pancreas causes the production of insulin to cease (Michel & Montada-Atin, 2014). Once the body is unable to

Type 1 is characterized by the body’s inability to produce insulin. It is caused by autoimmune-mediated destruction of pancreatic beta cells, which are responsible for producing insulin. There appears to be a hereditary link in people with Type 1 diabetes. Other factors have been known to cause Type 1 diabetes such as viral infections, toxins, and other environmental factors. Type 1 diabetes is the rare form, affecting about 10% of the diabetes population. Its onset usually occurs in people less than the age of 20.

Type one diabetes is an autoimmune disease. There are cells in the pancreas that produce insulin, the hormone that regulates the amount of insulin in your blood and

With over 30 million people having diabetes, the prevalence is continuing to rise. An estimated 200,000 American youth are effected by type 1 diabetes and 40,000 new cases are diagnosed each year¹. The rate for type 1 diabetes in youth has increased by 1.8% each year. In comparison, the prevalence for type 2 diabetes in youth has increased at a rate of 4.8% annually². No matter whether diabetes is type 1 or type 2 in children, family involvement is essential for optimal outcomes³.

Type 2 diabetes is essentially Type 1 but in reverse; the body becomes desensitized to insulin being produced, which causes the beta cells to overwork and become weakened and eventually die.

Diabetes type 1 develops when the immune system attacks the only cells that create insulin, the pancreatic beta cells. Due to the cells being destroyed, the person with diabetes type 1 has to be

Type 1 and type 2 diabetes are different disorders with different causes. Type 1 diabetes is an autoimmune disease. The immune system destroys the insulin producing cells, also known as beta cells, in the pancreas. Type 1 is also known to appear shortly after a viral infection. The

Firstly, those who have Type 1 diabetes; do not produce any insulin. Type 1 diabetics experience broken down fat and have high ketone levels, which destroys the insulin produced by beta cells located in the pancreas.Symptom development depends on the number of beta cells that are destroyed. Type 1 can also be caused by genetic factors, also it is believed that diabetes can be caused from environmental factors. However, type 2 diabetes occurs when insufficient amounts of insulin are produced. Alos, it supplies some insulin, but not enough to maintain healthy blood glucose levels. Typically, this particular type of diabetes occurs in the mid-forties, and there is a genetic factor, over three-quarters are overweight. Diabetes can be regulated by exercise, diet, weight loss, and oral medications to lower blood glucose levels. Millions of people have diabetes and are not aware of it, that is why it is important, to become familiar with symptoms of this disease and its risk

Type 1 diabetes is an autoimmune disorder, in which the pancreas does not produce insulin. It usually begins in childhood or adolescence. In Type 1 diabetes, the body’s immune system destroys beta cells in the pancreas that produce insulin. Insulin is a hormone that converts sugar, starches, and food into energy. Without insulin, blood glucose levels become too high, which is known as hyperglycemia. To prevent hyperglycemia, people with Type 1 diabetes must take insulin daily to survive. Genetic and environmental factors affect the onset of Type 1 diabetes. [1] According to the American Diabetes Association, a predisposition to Type 1 diabetes is

Type 1 diabetes is an autoimmune condition where the immune system wrongly identifies and subsequently attacks pancreatic cells that produce insulin, leading to little or no insulin production. This life-long disease requires daily monitoring and self-treatment. The two main methods are insulin injections and portable insulin pumps.

Type 1 diabetes, also referred to as Insulin Dependent Diabetes Mellitus (IDDM) or Juvenile Diabetes, can be caused by a genetic disorder. It can occur at any age, but it is most often diagnosed in children, adolescents, or young adults around 20 years old or before a person is 30 years of age. Insulin is a hormone produced by special cells, called the beta cells, in the pancreas, an organ located in the area behind the stomach. Insulin is needed to move blood sugar (glucose) into cells, where it is stored and later used for energy. In type 1 diabetes, these cells produce little or no insulin. Without enough insulin, glucose builds up in the bloodstream instead of going into the cells. The body is unable to

Type 1: In type 1 diabetes the cells that produce insulin in the pancreas are destroyed and cannot be made, this type of diabetes usually occurs in childhood due to the autoimmune system attacking and destroying its own cells that produce the insulin, the beta cells in the islet of Langerhans are destroyed and thus unable to produce insulin. This type of diabetes in incurable and is treated with insulin injections.

As previously stated, there is Type 1 and Type 2 diabetes. Due to the differentiation, the etiology of diabetes is divergent. Being insulin-dependent, Type 1 diabetes is brought about by the immune system eradicating beta cells, leading to the deficiency of insulin. In contrast, Type 2 diabetes has a basis that can often times be prevented to an extent (“Causes of Diabetes”, 2014).

Type 1 diabetes is an autoimmune disease that begins when the pancreas quits making insulin. Insulin plays a key role by letting glucose enter the body’s cells, and then uses it for energy. When the body doesn’t get the