Type 2 Diabetes Type 1

Type II Diabetes, also known as diabetes mellitus, is also called non-insulin-dependent diabetes or aadult onset diabetes. It is a medical disorder that, due to a number of factors codependent with the modern world, is characterized by higher than normal blood glucose levels that play havoc with insulin deficiency and resistance. Insulin resistance means that cells do not respond appropriately when there is free insulin in the blood system. Essentially, they body is reacting to an improper balance of sugars and insulin. Because obesity is often present, research suggests that even thought the mechanisms controling glucose and insulin are unclear, the adopose tissue likely

Type 2 Diabetes effects about 90% of people who have diabetes. Ways of controlling this type is by losing weight, having a healthy diet, plenty of exercise, and monitoring their blood-glucose levels. Although people people do these things to help, Type 2 is usually a progresses disease where is gets worse, and causes the person to have to take insulin usually in the form of a tablet. People who are overweight and obese have a significantly higher risk of developing Type 2 Diabetes as apposed to those who are at a healthy body weight. Those that are especially at risk are those who have visceral fat, which is also known as central obesity, belly fat, or abdominal obesity. Being overweight of physically inactive along side with eating the wrong foods all increase the risk of developing Type 2 Diabetes. The risk also rises with our age, because as we age we tend to become less active as well as gain weight. When men have low levels of testosterone levels they also get placed at a higher

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The increase in the prevalence of type 2 diabetes is causing huge health problem through out the world including developed countries. Mostly people with low income groups are affected in developed countries (Zimmet 2001).The magnitude of the healthcare problem of type 2 diabetes results mainly from its association with obesity and cardiovascular risk factors. Indeed, type 2 diabetes has now been identified as one manifestation of the “metabolic syndrome”, a condition characterised by insulin resistance and associated with a range of cardiovascular factors (Jonathan 2003)

Diabetes occur when there is a combination of inadequate secretion of insulin by the pancreatic beta cells and the peripheral insulin resistance. Insulin resistance leads to a reduced glucose transport into the muscle cells, increases both hepatic glucose production and breaking down of fats because it has been attributed to the elevated level of free fatty acids and proinflamatory cytokines in the plasma.1

Type two diabetes is the most common disease today, and it has a great impact on the lives of many people in the United States. Type two diabetes results when the body is resistant to insulin or impaired secretion of insulin by the pancreatic beta cells. The pancreas produces too much insulin, but over time it is not able to make an adequate amount of insulin needed in order to move the glucose into the cells. However, there are many factors that lead to type two diabetes. They are known to be genetic, high body weight, previously identified impaired fasting glucose levels or impaired glucose tolerance, family history of diabetes, and history of gestational diabetes or delivery of a baby over 9 pounds. Type two diabetes effects about 90% to 95% of the cases of disease in the US (Hinkle & Cheever, 2013). It's

Type 2 diabetes is a self-caused disease that prevents your body from using and producing insulin correctly which leads to high levels of sugar in the blood. When people consume too much or too little sugar, it affects their insulin resistance, which causes the pancreas to not maintain the right amount of insulin to keep the body's

Type 2 diabetes is a form of diabetes that is insulin resistant and it often results from excess body weight and physical inactivity.

if people are overweight and obese, there are greater risk of developing type 2 diabetes by in this case, type 2 diabetes can effectively leads diabetic people into different types of diseases such as eye damage, kidney failure, heart damaged and etc…into to death. my research question may have relates to this issue because obesity causes type 2 diabetes when the body doesn't produce enough insulin and due to people's poor diet and lack of exercise daily because the body needs to move around or do activity so that the glucose would fit to our cells and produce enough insulin.

writing this research paper will help me to understand the effect of type 2 diabetes have on the body and the complication of type 2 diabetes. The research was conducted in order to figure out whether the risk of developing type 2 diabetes can be reduced through lifestyle changes. Such changes include proper dieting and rigorous physical activities. While conducting the research it was apparent that some of the cause of type 2 diabetes, are sedentary lifestyle and bad dieting. Articles from the wed of science from USF and science direct elaborate on how that aspect may maximize the possibility of developing type 2 diabetes . People with type2 diabetes usually are overweight because of unhealthy lifestyle. Healthy habits may leads to weight loss in obese patients that suffer from type2 diabetes (Westman, E. C. 2014). This is also true for any person that want to prevent type 2 diabetes or suffer from related complications of that disease.

Diabetes has become a widespread epidemic, primarily because of the increasing prevalence and incidence of type 2 diabetes. Diabetes is an endocrine disease in which the body has either a shortage of insulin or a decrease ability to use insulin or both. Insulin is a hormone that allows glucose to enter the cells and be converted into energy. Diabetes can be characterized as a prevailing, incapacitating, and deadly disease. There are a number of risk factors that increase a person’s tendency toward developing type II diabetes. Modifiable risk factors include obesity, physical inactivity and poor dietary habits are just a few. The

The first step is physical activity that helps the patients of diabetes type 2 to control the level of glucose in the blood. Diabetes type 2 means that the glucose in the blood has a high range. Physical activity can affect the level of glucose, so it can make it low. Moreover, it is an essential component to manage or prevent type 2. Exercises have many benefits that help the patients with type 2. For example, it assists the patients to lose weight, so the high level of glucose inside the blood will be lower. When the patients do exercise, their muscles use the glucose to do the work, so the glucose will be low

Being overweight is a primary factor to diabetes due to the storage of fatty tissue. The more it is, the more resistant to diabetes and individual is. With fat distribution, the body stores more fat in the abdomen, the risk factor to Type 2 diabetes is greater. Inactivity due to no exercise or inability to move normally makes cells inactive to insulin. Family history increases the risk if a parent and/or sibling has type 2 diabetes. African Americans (race) are the most likely to develop diabetes than any other race. The older we get, the less we are to exercise, lose muscle mass and gain weight. Untreated pre-diabetes will eventually turn into type 2 diabetes as well as gestational diabetes when pregnant. (Centers for Disease Control, pg.2, 2012)

The organs involved include the liver, muscle, fat cells, liver, alpha and beta cells of the pancreas, GI tract, kidney, and brain. While the liver and muscle ideally increase glucose uptake in the fed state when insulin levels are high, with type 2 diabetes this is impaired. To further exacerbate the hyperglycemic condition, the liver not only fails to properly exhibit glucose uptake, but it actually over produces more glucose and thereby creates a cycle of glucose accumulation and further production. While this is occurring in the muscle and liver, fat cells have accelerated lipolysis. The lipolysis results in an increase in plasma free fatty acids (FFAs), which then in-turn impairs both first and second phase insulin secretion and can lead to excess fat deposition in the liver and muscle, contributing further to impaired insulin production. At the GI tract, gastric inhibitory polypeptide becomes resistant and loss of GLP-1 occurs. With impaired GLP-1, insulin resistance is again further enabled and hyperglycemia can become more profound. This occurs because glucagon suppression from the pancreatic alpha cells post meal does not occur as it would under normal circumstances, thereby enabling even more hepatic glucose production. While theories are not completely conclusive, insulin resistance in the hypothalamus may contribute to excess intake thereby contributing to additional glucose in the circulation. Finally, the kidney also plays a role in glucose dysregulation as it increases glucose reabsorption. All of these discussed mechanisms ultimately contribute to hyperglycemia and chronic hyperglycemia itself contributes to impaired beta cell function (DeFronzo,

“A major concern at this time is the rapid climb in incidence of Type 2 diabetes, with prevalence now estimated at about 9% (18 million) of the population greater than 20 years of age.” Obesity and Type 2 diabetes are commonly associated (Gould & Dyer, 2011). In addition to the two types of diabetes, there are signs and symptoms that are easily detected and some that develop after diagnosis.