Types of Shock Essay

How is Toxic Shock caused? Well its is caused by many ways. The first way could be cause is by a bacterial infection. The other way it could be caused would be your body might produce a little much of Staphylococcus Aureus bacterium. Another way could be the use of Tampons for women. The Tampon can scratch the vagina creating an opening for bacteria to get into your bloodstream. The last thing could be women who are menstruating or just had a baby.

According to information published by the Mayo Clinic, sepsis has three stages: sepsis, severe sepsis and septic shock. The mortality rate for septic shock is nearly 50 per cent, and an episode of severe sepsis increases the risk of future infections. Severe sepsis causes blood flow to the vital organs, such as the brain, heart and kidneys, to become impaired. Sepsis can also cause blood clots to form in organs and extremities such as arms, legs, fingers and toes, which can lead to organ failure and tissue death (gangrene).

blood is accelerated by the heart toward the head along the ascending aorta. In response, the patient's body recoils

This scenario helped me understand the pathophysiologic process of pneumonia and decompensated shock and how they could possibly manifest in children. Since in our first simulation of the semester we learned different methods of assisting a patient in improving his/her breathing status I was better able to intervene and know what to do to improve our patient’s breathing status. However, I have never been exposed to a patient undergoing decompensated shock. Therefore, this time I was able to learn what to do in case a situation as such arises in the future on a real patient. Shock can be due to several reasons such as bleeding or severe dehydration. However, it was apparent in this case that the patient was not externally bleeding, but she was

The unresolved infection has landed the patient on septic shock. Sepsis is the body’s response to inflammation of a particular or unknown infection. The presence of hypotension despite adequate fluid infusion and inadequate tissue perfusion is the result of septic shock. The treatment of shock is centered on the restoration of blood pressure to normal, the presence of adequate tissue perfusion, making sure organs return to functioning well, and avoiding further complications (Lewis, Dirksen, Heitkemper, Bucher, & Harding,

Septic shock is a subset of severe sepsis and was defined as “sepsis-induced hypotension persisting despite adequate fluid resusci- tation” (see Fig. 12.1). While the quantity of fluid that qualifies as “adequate fluid resuscitation” is controversial, we believe septic shock is best defined as a “mean arterial pressure (MAP) less than 65 mmHg after a fluid challenge of 20 mL/Kg body weight (given 30–60 minutes) in patients with sepsis and in the absence of other causes for hypotension” (also see Chap. 14). According to the ACCP/SCCM defini- tions, three stages in the hierarchy of the host’s response to infection were recognized, namely, sepsis, severe sepsis and septic shock, with sepsis having the best prognosis and septic shock the worst. While the use of the SIRS criteria to define sepsis is some- what controversial [3–6], many consider sepsis to be best defined as the “systemic response to infection with the presence of some degree of organ dysfunction”

Sepsis is a phenomenon in which an infective agent results in a physiological response in excess of the pathogenic insult. Efforts to define and investigate sepsis have been somewhat fruitful in elucidating the nuances of this phenomenon. Yet, gaps exist in identifying and investigating sepsis. Recent efforts have streamlined the definition and identification criteria of sepsis and septic shock. The treatment of sepsis is based on the elements of identification, hemodynamic interventions (fluid and vasopressor resuscitation), and antimicrobial therapy/source control. A two-tiered approach employing the qSOFA (Quick Sepsis-Related Organ Failure Assessment) and SOFA (Sepsis-Related Organ Failure Assessment) tools now provide a succinct criteria for sepsis and septic shock. Various approaches may be taken to assess fluid responsiveness, provide fluid resuscitation, and determine the need for vasopressor support. Early, aggressive antimicrobial therapy is essential to the treatment of sepsis. Obtaining source control and considering early surgical

Next we have Cardiac tamponade [which is caused by blunt or penetrating trauma or a few medical conditions such as cancer or autoimmune dieses] which is a serious condition where the blood accumulates in the pericardial sac [the sack around the heart]. While there is no definitive treatment on a basic level and ALS provider can relieve fluid with a needle in a risky but necessary procedure the last form of cardiogenic shock is a pulmonary embolism. A pulmonary embolism is when there is a blood clot blocking the flow of the pulmonary veins leading to obstructive shock and causing heart failure. Interesting enough to mention is the fact that one of the more common causes to cardiogenic shock is damage to the heart muscle however just because you have a heart attack does not necessarily mean you have had caridogenic

Septic shock is the stage of sepsis when multiple organ failure is evident and uncontrolled bleeding of the body occurs. Septic shock results from the complication of an infection where toxins initiate a full body inflammatory response. Viral, fungal, and bacterial infections can all lead to septic shock, but the most common causes of sepsis are gram- negative bacteria and gram-positive bacteria. Sepsis causes severe hypovolemic shock and hypodynamic cardiac function to affect the body, platelets and clotting factors are consumed and thus the body cannot clot blood. Capillary leak occurs and cardiac contractility is poor from cellular ischemia (Ignatavicius & Workman, 2011). For a person to be diagnosed with septic shock, they must have symptoms

Losing just one pint of blood is all it takes to send the body into shock. The heart pumps

Sepsis can develop into septic shock which is the point where the patient’s blood pressure drops to a dangerous level due to the presence of bacteria in the body.

Hypovolemic shock “is an emergency condition in which severe blood and fluid loss make the heart unable to pump enough blood to the body” ( Heller). Hypovolemic shock can be a result of numerous things such as dehydration, excessive diarrhea or bleeding (Nall). Excessive bleeding, both externally and internally, due to injuries is known as Hemorrhagic shock. Hemorrhagic shock is a type of hypovolemic shock that is “associated with the sudden and rapid loss of significant amounts of blood” (Dictionary).

According to the National Trauma Institute, hypovolemic shock is the second leading cause of death in people with traumatic injuries. [1] This makes the initial identification and management of Hypovolemia detrimental to the outcome and survival of many patients involved in traumatic events. Hypovolemia refers to a medical condition in which rapid fluid loss results in multiple organ failure due to inadequate circulating volume and consecutive inadequate perfusion. [1,2,3]

Shock is a common condition in critical care, affecting about one third of patients in the intensive care unit(ICU).

Possible signs and symptoms of toxic shock syndrome include a sudden high fever, low blood pressure (hypotension), vomiting or diarrhea, a rash resembling a sunburn located particularly on the palms and soles of the feet, confusion, muscle aches, redness of the eyes, mouth and throat, seizures, and headaches. If you have signs or symptoms of toxic shock syndrome call your doctor immediately. This is especially important if you’ve use of tampons or if you have a skin or wound infection. Toxic shock has been associated with using contraceptive sponges, diaphragms or superabsorbent tampons, and having a viral infection such as the flu or chickenpox. Toxic shock syndrome can progress rapidly into shock, renal failure or even