Ventricular Serptal Syndrome

Atrioventricular Canal Defect is an abnormality that causes the mixing of blood. There is a hole in center of heart where the wall between the upper and lower chambers meet. The tricuspid and mitral valves aren’t formed properly and one large valve crosses the defect. The defect lets oxygen rich blood pass to the heart’s right side and mix with deoxygenated blood, then go back to the lungs. Another abnormality is Atrial Septal Defect (ASD), where the walls of the upper chambers of the heart don’t close completely, causing a left to right movement of blood due to the higher pressure. The mixing of oxygenated and deoxygenated blood may cause the right atrium and ventricle to enlarge due to the higher volume of blood.

Congenital heart disease is a cardiovascular condition resulting from an abnormality in the structure of the heart. The exact time this defect forms is unknown but it is during the fetal development in the womb. Researchers think the defect could be case partially by genetics and medical conditions of the fetus and the parents. If the baby’s mother had rubella, while pregnant or other conditions such as diabetes could have caused the defect. Some medications that were taken by a pregnant mother could also have caused birth defects. An error on chromosome 22 could also have caused the heart defect. Some symptoms that a person may have a congenital heart may not show up until later in life but many children also have the symptoms. Symptoms of a congenital heart defect are abnormal heartbeat, bluish tint to skin, shortness of breath, dizziness or fainting, and swelling of body tissues and organs.

A ventricular septal defect or VSD is a heart defect. It’s congenital, this means that a baby is born with it. It is an opening or hole in the dividing wall called the Septum, between the two lower chambers of the heart, right and left ventricles. Ventricular septal defects are the most common type of congenital heart defects. A VSD allows oxygen-rich blood to pass from the left ventricle through the opening in the septum. Then it mixes with oxygen-poor blood in the right ventricle. There are different types of ventricular septal defects, the type your child has depends on which part of the wall between the ventricles is involved. Also the size of the opening or hole varies. A ventricular septal defect may occur more often in some families, this is because of gene defects. But most of the time, the cause is unknown. Similarly to ventricular septal defect, an Atrial septal defect is a birth defect of the heart in which there is also a hole in the wall in the septum, but instead of dividing the lower chambers it divides the upper chambers of the heart (atria), here a hole can vary in size and may close on its own or may require surgery as well. An atrial septal defect is one type of congenital heart defect, in which also the baby is born with it.

Atrial Septal Defect affects the cardiovascular system of a person’s heart and affects a person’s life. The understanding of the heart and the process it completes in our bodies. As we look into the difference of what the heart does and the challenges you go through when your heart is not healthy. When we our born we our born with heart murmur’s in which they are supposed to close throughout your growing process. Atrial Septsl Defect is a hole in your heart that should be fixed in order for the person to live longer or a full life.

Additionally, using a stethoscope, a doctor will often hear a heart murmur caused by the blood blowing through the atrial septal defect. It is not uncommon for a murmur to not be detected right at birth. PAPVR isn 't hereditary but is a result of when the veins don 't come together and grow properly

Congenital heart disease in newborns is a very common birth defect and effects roughly 1 in 100 or 1% of the babies born in the United States. Some of the heart defects that are a part of congenital heart defects include, hypoplastic left heart syndrome (HLHS), pulmonary atresia (PA), Tetralogy of Fallot (TOF), Total anomalous pulmonary venous return (TAPV/TAPVR), tricuspid atresia and many more.

Congenital heart defects (CHD) are birth defects that are considered the common in the United States. These defects affect a newborn child’s blood flow to and from the heart and sometimes can leave the heart lacking parts that make it necessary for the heart to do its job. According to the Centers for Disease Control and Prevention (CDC) (2014), 1 in 4 children born with a CHD is considered critical and will result in that child needing heart surgery or other similar procedures within the first years of its life. These CHD’s can range from Atrial Septal Defects, Coarctation of the aorta, Ebstien anomaly, Pulmanary artesia, Tetralogy of Fallot, Trancus Arteriosus, Hypoplastic Left Heart Syndrome, as well as many others (Centers for Disease Control and Prevention, 2014). The specific CHD that will be covered in detail will be the Hypoplastic Left Heart Syndrome (HLHS). HLHS accounts for nearly 960 congenital birth defects per year and

The definition of TOF reads, a type of congenital heart defect. Congenital means that it is present at birth (UMMC, 2013). TOF occurs when there is 4 abnormalities all together. One of the 4 abnormalities is when a massive hole is in the wall of the muscle (septum) that separates the 2 bottom pumping cambers (ventricles) of the heart. The massive hole in the abnormality itself is a VSD. A VSD is known as a Ventricular Septal Defect. Due to the location of the VSD, two more abnormalities develop. One being that there is a vast blood vessel which take the blood to the body (aorta) is pulled toward, and “overrides” the ventricular septum so that it sits over both the left and right ventricles; this is called an overriding aorta (UMMC, 2013). The second abnormality that takes place is that the muscle produces obstruction to the blood flow going out the right across the pulmonary valve. The right ventricular outflow tract obstruction, can be due to obstruction below the valve, at the valve or in the pulmonary arteries as they deliver blood out to the lungs (UMMC, 2013). There is different severities of TOF. The most acute form is known as pulmonary atresia. In patients with pulmonary atresia there is no functional pulmonary valve. The right ventricular outflow obstruction leads to thickening of the

Congestive cardiac failure is by far the most frequent symptom. It may be associated with massive peripheral edema or massive pleural effusion leading to cardiac tamponade. It may rarely mimic other congenital cardiac malformations such as Ebstein’s anomaly or functional pulmonary atresia. Arrhythmias and conduction disturbances are not a predominant feature of Uhl’s anomaly, probably due to absence of residual foci to initiate or transmit anomalous electrical activity. (Gerlis 2003)

Atrial Septal defect is the most common form of atrial heart defect that occurs when the atrium septum fails to form properly. It is a congenital defect that creates an opening between the two chambers (right and left atrium).

An atrial septal defect (ASD) is a hole in the heart. This hole is located in the thin tissue (septum) that separates the two upper chambers of the heart, the right and left atrium. This hole is present at birth (congenital). A few minutes after birth, this hole normally closes so that blood is not able to go between the right and left atrium.

Congenital left ventricular outpouching (LVO) is a rare disorder in which a confined protrusion arises from the left ventricular (LV) free wall. The terms LV aneurysm, LV diverticulum, double-chambered LV and LV accessory chamber have been used interchangeably in the literature [1–9], however anatomical differences do exist between these terms [10]. LV aneurysm is an expansible thin-walled, usually fibrotic myocardium and pericardium, pouch contracting in asynchrony with the LV and has a wide communication with the LV cavity. Conversely, an LV diverticulum has three cardiac wall layers (endocardium, myocardium and pericardium), contracts in synchrony with the LV and has a narrow connection to LV. LV accessory chamber presents an entity that combines some criteria of both aneurysm and diverticulum. Its wall is similar to a diverticulum but it has a wide communication with the LV as an aneurysm. Lastly, double-chambered left ventricle displays a combination of the above-mentioned characteristics.

Echocardiography is the initial diagnostic modality for a patient with suspected congenital heart disease. In some patients, however, use of this modality is encumbered by its limited ability to delineate great arteries and intra cardiac anomalies, pulmonary veins, and coronary arteries. (7)

Atrial Septal Defect(ASD) is a very large problem concerning the heart in its overall function. When the heart, being the core of the cardiovascular system, has issues; it effects the rest of the body as a result. The core of the problem resides in the atrial septum. Normally the heart is divided into four separate chambers. But a person with atrial septal defect has an atrial septum that allows the blood from the left side of the heart back into the right side. This results in increased pulmonary blood flow and diastolic overload of the right ventricle. By having this constant left-to-right shunt, it can alter the pulmonary vascular resistance leading to hypertension or even the reversal of the shunt itself.

Atrial Septal Defect is a common and interesting disorder. Atrial Septal Defect is mostly know as “a hole in the heart.” This disorder is birth defect. It can happen to male, female, adult, and kids. This disorder mostly happens to kids and infant. Atrial Septal Defect is when there is a hole in your heart that doesn’t allow oxygen to flow through. This can causes difficulty breathing, heart murmur, and e.t.c... There three different types of level. These level are called ostium secundum, ostium primum, and sinus venosus. The most severe level is ostium secundum which is the true atrial septal. Then is ostium primum, and last sinus venosus. This genetic disorder is typically diagnosed by noticing .symptoms.