1. Voldemort was diagnosed with dysarthria. How is dysarthria different from apraxia of speech? Include definitions of each. (3 points)
Dysarthrias are a group of motor speech disorders characterized by various forms of articulatory mistakes, and poor intelligibility. Dysarthrias typically include lots of speech errors. Apraxia of speech is the result of a brain injury (stroke, degenerative disease, TBI, anoxia and tumors). In apraxia of speech, the individual has a breakdown in motor planning and programming. The individual would have difficulty with voluntary movement for speech tasks. In apraxia of speech there is a deficit in prosody and articulation. The major difference between dysarthria and apraxia of speech is that dysarthria is a
…show more content…
4. Explain the neurological bases for Voldemort’s problems (i.e, which brain structures are affected)? (1 point)
In ataxic dysarthria, the cerebellum and its connections are affected. Following damage to the cerebellum, complex movements seem to be broken down or decomposed into individual sequential components.
5. In your opinion, how severe is the speech problem and what (from the case study) indicates the level of severity? (2 points)
I would say that the level of severity is an eight on a ten-point scale. This dysarthria is affecting Voldemort’s day to day life and it is difficult for him to have a simple conversation without him or others getting frustrated. When the case study says, “He complains that his people frequently ask him to repeat what he says and that he finds communication frustrating” this is a sign that the disorder is taking over too much of his life. Another reason this speech problem is very severe is because he is not able complete daily tasks because there is a slowness in walking and at some points he cannot even
…show more content…
Read the journal article on D2L based on LSVT and answer the following questions:
i. Cite the article in APA format (0.5 point)
Ramig, L. O., Sapir, S., Fox, C., & Countryman, S. (2001). Changes in Vocal Loudness following Intensive Voice Treatment (LSVT) in Individuals with Parkinson's Disease: A Comparison with Untreated Patients and Normal Age-Matched Controls. Movement Disorders, 16 (1), 79-83. ii. State the aim of the study in one sentence (1 point)
This study assesses the impact of LVST on vocal loudness in individuals with dysarthria, who have been diagnosed with Parkinson’s Disease. iii. State the number and type of groups and number of participants in the study (1.5 points)
Three groups were included. The first group was individuals with Parkinson’s disease receiving LVST. The second group was individuals with Parkinson’s disease receiving no treatment. The third group was individuals who are neurologically normal. Forty-three individuals participated in this study. iv. Summarize the LSVT technique as described in the article in your own words (2
7. If a study had a result of F(2, 147) = 4.56, p = 0.003, how many groups were in the study, and what was the sample size?
In 1985 Lorraine Olson Raming, a speech language pathologist, discovered the Lee Silverman Voice Treatment (LSVT) LOUD while working on it for a family to communication with an individual with Parkinson’s Disease (PD). Recently components of LSVT-LOUD have been applied to limb movement by Dr. Becky Farley to provide intensive amplitude specific therapy for those with PD (Millage, Vesey, Finkelstein, & Anheluk, 2017). This is known as LSVT-BIG, a protocol designed to address the movement impairments associated with people who have PD. LVST-BIG therapy improves proprioception by increasing amplitude with maintaining attention and cognitive involvement on individual movements (Dashtipour et al., 2015). LVST-BIG has a standard protocol: 4 consecutive days a week for 4 weeks (16 sessions in one month) for one hour
As a Speech Language Pathologist it is my responsibility to provide a means of communication to any individual who has an impairment as it relates to communication. No matter how severe the deficit may be as it relates to cognitive functioning, motor skills,etc.. and any other disability that may impact the traditional means of communication. A Speech language Pathologist who works with individuals who require the use of an Augmentative and alternative communication device, must keep in mind that it is their responsibility to make sure that more than one mode of alternative and or augmentative can be used, monitor the progression of intervention as well as evaluate the individual and most importantly keep up to date with current practice through
SPLP 635 - Voice Disorders. Voice Disorders is part of the core curriculum in the master’s degree program in speech-language pathology, approved by the American Speech-Language and Hearing Association (ASHA), and the accrediting body for professional training programs in speech-language pathology, CAA-CSD. This graduate level course is designed to provide students with knowledge of normal voice including the vocal parameters of pitch, intensity, quality, resonance and flexibility. Various idiopathic, structural, resonance, neuropathologic, and congenital disorders of voice are addressed in terms of symptomatology, perceptual features, diagnosis, prevention, and intervention. The course provides a survey of voice disorders that students
Spastic dysarthria is caused by damage to one or more CN nuclei, mysthania gravis, stroke trauma, bell’s palsy and infection. Unlike spastic dysarthria flaccid is caused by primary lateral sclerosis, multiple sclerosis and others. They also differ in the physiology
“‘Atticus, he was real nice….’… ‘Most people are, Scout, when you finally see them.’” is a dialogue written by Harper Lee that stuck with me after reading To Kill A Mockingbird in high school. Despite physical appearance or intellect, everyone deserves to be valued at their core; what better way to affirm someone than by conversing with them as Scout did with Boo Radley. Thus, my aspiration is to enable a voice to those with communication disorders. This desire to be a speech language pathologist has been influenced by personal and professional exposures to the field.
Speech pathology focuses on aiding individuals who struggle with or cannot use their voice. Without a voice, individuals would not be able to communicate with one another face to face, or even talk on the phone. Within every field, controversy exists. In speech pathology, one such controversy is nonspeech oral motor exercises (NSOME), where a patient does different tasks involving their mouth or fingers. These exercises are believed to have no connection with actually assisting the speech pathological part of the brain, since they do not involve any sort of speech activity. Nonspeech oral motor exercises do not assist in the healing process of patients with actual disabilities, nor are they an efficient practice at a professional treatment center.
There are many different language delays and disorders found in the pediatric population. Childhood apraxia of speech (CAS) is one of the most common of these disorders. Dr. Libby Kumin defines CAS as “a motor speech disorder where children have difficulty planning, coordinating, producing and sequencing speech sounds” (Kumin, n.d.). Apraxia does not occur because of weakness or paralysis of facial and oral muscles. It occurs when a child’s brain cannot properly plan the movement of body parts necessary for normal speech production (“Childhood Apraxia,” 2011). Though CAS is the most common name for this specific disorder, it is also referred to as a variety of other names. Some of these names include: dyspraxia, developmental
Many may not know Parkinson’s disease is the second most common neurodegenerative disorder in the world. This disease is most seen in the elderly starting at 62 years of age although, younger individuals can still have the disease it isn’t common. Parkinson’s make it difficult for its victims to carry out everyday activities that might have once been easy for them. As the disease progresses it makes it hard for the patient to do things like walk, stand, swallow and speak. A great deal of people don’t realize how helpful therapy can be when dealing with such disease!
Spastic dysarthria; results from a lesion in the upper motor neurons and causes spasticity of the muscles. A lesion in on the lower motor neurons resulting
Parkinson’s disease is caused by the destruction of dopamine-producing nerve cells in the midbrain (Substantia nigra). These nerve cells are used to coordinate smooth and regular body movement. In the absence of these cells, people often experience tremors (involuntary shaking or
Her parent reported speech and voice concerns saying she would take a full tidal breath not always having enough breath. She would stop in the middle of sentences to get a breath. Her observed speech and voice signs were variable loudness and variable pitch. She has a inconsistent strained voice quality, especially at end of sentences and a mild imprecise articulation. She had moderate spastic Dysarthria. She was assigned to study condition D. Her gender matched control was a seven year old female. There was a gender matched control group that was recruited to participate in the study. This allowed for a direct comparison between the performance of participants with Cerebral Palsy and their matched peers, because data were collected using the same tasks and methodology. (Fox,Boliek
Parkinson disease (PD) is a progressive neurodegenerative disorder characterized mainly by physical and psychological disabilities. This disorder was named after James Parkinson, an English physician who first described it as shaking palsy in 1817 (Goetz, Factr, and Weiner, 2002). Jean- Martin Charcot, who was a French neurologist, then progressed and further refined the description of the disease and identified other clinical features of PD (Goetz, Factr, and Weiner, 2002). PD involves the loss of cells that produce the neurotransmitter dopamine in a part of the brain stem called the substansia nigra, which results in several signs and symptoms (Byrd, Marks, and Starr, 2000). It is manifested clinically by tremor,
“Dysphonetic means having difficulty connecting sounds to symbols; it also means they might have a hard time sounding out words. Dyseidetic means to have a good grasp of phonetic concepts, but great difficulty with whole word recognition and spelling still exists” (Wilkins URL). Typical mistakes would be reversals such as the word ‘at’ spelled ‘ta’ and mistakes in phonetic spelling of a word like ‘phone’ spelled ‘fon’.
Many studies show that singing and listening to music can stimulate various areas of the brain and encourage cognitive function(Miendlarzewska & Trost, 2013; Wan, Rüber, Hohmann, & Schlaug, 2010). Similarly, Lee Silverman’s Voice therapy also improves vocal function and respiratory drive in people with Parkinson’s disease. Both techniques help people with speech impairments cope with their symptoms. LSVT consists of a high intensity training program that has four 1 hour training sessions four times a week for four weeks. However, music based therapy that singing may naturally promote and intensify aspects of voice and speech production. Singing can encourage louder voice production and can help patients learn to develop and train their respiratory capacities. Additionally, singing may activate the limbic system, whose connections to various subcortical networks involved in the regulation of vocal intensity control. Lee Silverman’s Voice Therapy aims at increased healthy vocal loudness, measured as the sound pressure level (SPL) and music based therapy may naturally promote and intensify aspects of voice and speech production. Music based therapy has more popularity than LSVT because you can sing anywhere and LSVT is more lab based therapy which requires you to participate and physically go somewhere four times a week. In a recent study, people were asked to participate in LSVT and musical based therapy both for 1 hour, four times a week for four weeks to compare outcomes of each technique. Each technique showed great improvement however, the people that participated in LSVT were more likely to have long-term benefits (2 years longer) than music based therapy (Ramig, 2001). However, there should have been a placebo group that thought they were receiving treatment but did not because people may have been subject to a placebo effect. Even though LSVT produces