What Is Obstructive Sleep Apnea?

Sleep apnea is a common sleeping disorder where a person has experiences of not breathing during sleep. Over 20 million Americans, mostly overweight men, suffer from sleep apnea. Despite these numbers, sleep apnea is often not treated directly because its symptoms are thought to be those of depression, stress, or just loud snoring. There may be a genetic component to this disorder as it often occurs within families.People with sleep apnea stop breathing for at least 10 seconds at a time; these short stops in breathing can happen up to 400 times every night.

Obstructive sleep apnea (OSA) is an ever-increasing problem affecting millions of people in the United States. The prevalence of OSA has risen drastically over the past few decades concurrently with the increasing prevalence of obesity. Subsequently, there has been an ever-increasing rise in the use of CPAP. While there are many adverse effects to the use of CPAP, the majority are described as being relatively benign. Here we discuss a case of significant sudden sensorineural hearing loss (SSNHL) in relation to a suspected perilymphatic from traumatic barotrauma resulting from excessive self-titration of CPAP in an in-home setting.

There is a strong association between obstructive sleep apnea and hypertension, among cardiac conditions such as heart failure, cardiac arrhythmias and coronary heart disease. OSA and hypertension share similar risk factors such as age and obesity, and are commonly co-morbid. During an obstructive sleep apnea episode the patient experiences reduced airflow due to obstruction in the upper airway, leading to hypoxia and hypercapnia, the patient is typically awakened by these episodes. Due to hypoxia, OSA causes an increase in sympathetic activity, endothelial dysfunction and increased oxidative stress. Increase systemic activity leads to an increase in both heart rate and blood pressure. Endothelial dysfunction is an imbalance of vasorelaxation

Sleep Apnea, by Davis, Andrew S, explains the breathing cessations during sleep. Sleep Apnea reduces the amount of oxygen flowing to the brain which causes brain issues. People with sleep apnea usually wake up at night because of the shortness of breathing, then resume back to sleep after adjusting their breathing. Davis states that due to sleep apnea, a few individual has sleeping issues such as snoring, headaches or even find it hard to fall asleep. Sleep apnea is also existed in children (from 0.5% to 3% of the population) which cause them often be crying at night and tired in the next morning. In his writing, Davis states that there are six characteristics of sleep apnea. The most noticeable characteristic is the common nighttime symptoms

Obstructive sleep apnea is up to four times as common in men as in women, but women are more likely to develop sleep apnea during pregnancy and after menopause. In older adults, the gender gap narrows after women reach menopause. More than half of people with obstructive sleep apnea are either overweight or obese, which is defined as a body mass index (BMI) of 25-29.9 or 30.0 or above, respectively. In adults, excess weight is the strongest risk factor associated with obstructive sleep

* Obstructive Sleep Apnea is when the pharyngeal airway collapses or narrows respectively during sleep. It is accompanied by excessive daytime sleepiness and risk for cardiovascular disease. Risk factors for OSA include obesity, large soft palate/tongue, the male sex, and aging.

Apnea is frightening for parents as breathing stops suddenly and a baby may become limp and blue. Breathing usually restarts after a few moments and gentle stimulation of a baby, stroking their face or moving them slightly, can encourage a quick recovery unless the child is otherwise ill with pneumonia or heart failure. Before a

Obstructive rest apnea is a typical unending illness in Western culture whose commonness is evaluated at 2% of ladies and 4% of men in the overall public. It is portrayed basically by repetitive impediment of the upper aviation route that outcomes in oxyhemoglobin desaturation and occasional feelings of excitement from rest. It now creates the impression that even gentle to direct rest apnea is connected with the improvement of hypertension, diabetes mellitus, and cardiovascular danger. With expanding corpulence, rest apnea can add to the advancement of daytime alveolar hypoventilation (weight hypoventilation disorder), cor pulmonale, and candid respiratory disappointment. Subsequently, given its high pervasiveness and grimness, rest apnea represents a huge clinical weight to Western culture.

Sleep apnea is a chronic disorder where breathing repeatedly stops and starts during sleep. It leads to snoring, fatigue and interruptions of oxygen flowing through the body. The need for life insurance varies with age, responsibilities and your health. Life insurance policies become valuable financial resources for maintaining a standard of life. Policies are designed to provide a fixed amount of money according the policy terms and conditions to helping families avoid hardships. Life insurance policies can include the insurer’s ability to draw against the face value of the policy in the event of an illness. In the case of having sleep apnea, the insurer becomes the beneficiary of the policy value. There are also instances, where the policy also functions as a secondary health coverage, should the primarily carrier deny the medical claim.

Obesity is established as one of the leading risk factors for development of both type 2 diabetes and obstructive sleep Apnea(OSA). Obesity is medically defined as a complex disorder involving an excessive amount of body fat. Clearly, this isn’t just a cosmetic concern. The Body Mass Index (BMI) numerical range is 30.0-34.9 is consider obese (1) 35.0-39.9 obese (2), finally 40.0 above is extreme obesity (3). When it comes to Obesity it normally does not have just one underlining issue. Other factors are cardiovascular disease, and hypertension. Most likely a patient will develop a serious sleeping disorder, one called obstructive sleep apnea(OSA). This causes breathing to repeatedly stop and start during sleep. The throat muscles intermittently relax and block the airway during sleeping. Several signs of sleep apnea exist for example; loud snoring, shorter sleeping duration, poor quality of sleep, high blood pressure, morning headaches, decrease libido, weight gain, and even death due to lack of air. Polysomnography is medically performed to evaluate the presence of OSA and its severity.

Certain conditions can also promote sleep apnea causes such as bulky throat tissue, large tonsils, a recessed jaw, adenoids, uvulas, tumors or cysts in the airway, and nasal deformity. All these can cause the airway to collapse when you breathe.

Obstructive sleep apnea (OSA) is highly prevalent but very frequently undiagnosed. OSA is an independent risk factor for depression and cognitive impairment/dementia. Herein we reviewed studies in the literature pertinent to the effects of OSA on the cerebral microvascular and neurovascular systems and present a model to describe the key pathophysiological mechanisms that may underlie the associations, including hypoperfusion, endothelial dysfunction, and neuroinflammation. Intermittent hypoxia plays a critical role in initiating and amplifying these pathological processes. Hypoperfusion and impaired cerebral vasomotor reactivity lead to the development or progression of cerebral small vessel disease (C-SVD). Hypoxemia exacerbates these processes,

Obstructive sleep apnea is associated with cognitive decline and depression. This is the first study explicitly evaluating the impact of obstructive sleep apnea (OSA) on neurocognitive functions and changes in brain morphology in older adults with both depression and cognitive impairment. Under the mentorship of Dr. D.P Devanand, I prospectively screened OSA with the STOP-Bang questionnaire in patients enrolled into the Donepezil Treatment of Cognitive Impairment and Depression (DOTCODE) trial. High and low probability of OSA were defined as a STOP-Bang score of ≥5 (h-OSA) and of <5 (l-OSA), respectively. Baseline magnetic resonance imaging (MRI) was used to evaluate brain morphology. The initial 16 weeks of antidepressant treatment were part

Obesity is a leading risk factor for OSA, as weight increases the risk of OSA increases as well. The prevalence of OSA in obese patients is almost twice compared to normal weight patients. As a patient gains 10% of their weight their risk of developing OSA increase by 6. It is estimated that almost 25% of adults with a BMI between 25-28kg.m2 have some form of OSA. Being overweight can worsen OSA due to fat deposition in specific spots that may induce apnea. Patients who are overweight have additional fat tissue by the upper airway causing an increase in collapsibility leading to apnea events. In addition, the fat deposits around the trunk reduce the chests functioning leading to an increase in oxygen demand.