What Is The Hypothesized Role Of Liver X Receptor Active?

Skin cancer is a disease that can affect any one. Skin cancer includes many types that can be classified as severe or superficial. Consequently, treatement of skin cancer depends on the degree of severity a person has. In the following paper, we will shed lights on the types of skin cancer and its causes. Finally, we will investigate how to treat the different types of cancer and how to avoid it.

Generally this means a pleasing appearance is not a guide to character. In this case, we

The skin is divided into three different parts including the epidermis, dermis, and hypodermis. The epidermis is the outermost layer of the skin. This region of the skin has no blood cells or blood vessels running through it. All of the nutrients that the epidermis needs are received through diffusion from the dermis. The epidermis is made up of stratified squamous epithelial cells. The epidermis is made of five separate layers: the stratum basale, stratum spinosum, stratum granulosum, stratum lucidum, and the stratum corneum. Starting at the innermost layer, the stratum basale is where mitosis of keratinocytes takes place. This layer of the epidermis also contains melanocytes which give the skin it’s pigment. As mitosis takes place in the stratum

In Chao Yuanfang’s Zhu Bing Yuan Hou Lun (Treatise on Causes and Symptoms of Diseases), there had been more than 130 kinds of skin diseases and some rarely seen diseases recorded. Records of these diseases and their treatments

Xeroderma pigmentosum is an inherited condition characterized by an extreme sensitivity to ultraviolet rays from

Until the early 1980s, psoriasis was believed to be a disease primarily of epidermal keratinocyte proliferation and the cutaneous inflammatory infiltrate to be a secondary event. However, subsequent studies presented evidences that innate as well as adaptive immunity are crucial in the initiation and maintenance of psoriatic plaques. Type 1 and type 17 T lymphocytes secrete respectively (IFN-γ), (IL-2), IL-17, IL-22, in addition to TNF-α and IL6 (Gisondi and Girolomoni 2009).

The skin disorders that might results from hereditary, congenital, or acquired pathological processes are ichthyosis, bullous diseases, chronic infections of the skin, dermatitis, hiradenitis suppurativa, genetic photosensitivity disorders, and burns.

According to the article that talks about basal cell carcinoma, it says that this skin cancer originates from basal keratinocytes in the top layer of the skin, better known as the epidermis. Sometimes these tumors

This condition develops when the body's defense system (immune system) attacks the layer of tissue beneath the skin. It is not known why this happens.

Scientists do not fully understand Vitiligo yet; they are still trying to find out why it occurs. Evidence points towards the conclusion that it is an autoimmune disorder, in which the body’s immune system mistakenly targets the melanocytes in your body. The changes in the skin can vary from patient to patient, being that

Discoid lupus erythematosus (DLE) is a chronic, scarring, atrophic, photosensitive dermatosis. DLE may occur in patients with systemic lupus erythematosus (SLE), and <5% patients with DLE progress to SLE. The primary lesion is an erythematous papule or plaque with slight-to-moderate scaling. As the lesion progresses, the scale may thicken and become adherent, and pigmentary changes may develop. Patients with DLE rarely fulfill four or more of the criteria used to classify SLE. Serologic abnormalities are uncommon. Therapy with sunscreens, topical corticosteroids, and antimalarial agents is usually

Epidermolysis bullosa first presents itself between birth and early childhood and affects both genders and every race and ethnicity (Mayo Clinic Staff, 2014). When important structural proteins are faulty or not present within the skin of a patient, the skin loses its strength and stability. More specifically, EB Simplex, one of the major subtypes of EB, lacks proper functioning of Keratins 5, 14 and/or a structural protein called plectin in the epidermis (Debra). Since keratin proteins provide strength and resiliency to the outer layer of skin, and plectin helps make up the cell’s cytoskeleton, not having properly functioning proteins created an unstable epidermal layer. As a result, fluid-like blisters form in the top most layer of skin

The skin is often involved in vasculitic syndromes, because it is so large and sois richly supplied with blood vessels. Furthermore, iIt is exposed to low temperatures and is subject to the stagnation of blood by pressure and by gravity-dependent stasis. Thus it is peculiarly susceptible to vasculitis, whether primary or secondary as in hepatitis C and other infections, serum sickness, autoimmune disorders or malignant neoplasms.

According to Brown & Phillips (2010) several biological process in the skin require nutrients for proliferation. The

that are important for maintaining the barrier function of the skin (2). The pathogenesis of