All the highlighted answers are wrong please explain why it is wrong and give me the correct answer. Thanks, in advanced 1) The insulin receptor catalyzes the phosphorylation of several substrates and is therefore cd assified as a Transferase Phosphate Kinase Lyase Oxidoreductase
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- Discuss the aetiology and pathogenesis of type 2 diabetes mellitus. In your answer, make clear the term ‘insulin resistance’ by providing an example to illustrate your understanding of this occurrence. Determine whether the C-peptide test would be useful in diagnosing type 2 diabetesMention the chemical change that proinsulin undergoes, to be able to act as mature insulin.Hexokinase deficiency (HKD) is a congenital disease that can disrupt intracellular glucose levels. Please provide an explanation of the diseases impact on intracellular glucose levels and the mechanism by which how GLUT1 regulates this disrupted glucose level.
- Which type of insulin can never be combined with any other insulin:All of the following molecules are involved in insulin signal transduction except a. IRS b. RAS c. ANF d. PIP3Which of the following medications does not have hypoglycemic effects (does not augment insulin activity)?A. Glicised (Glicinum)B. SalicylatesC. SulfanilamideD. EthanolE. Fluoxetine
- What adipokine has anti-inflammatory and anti-thrombotic effects and also acts to reduce insulin resistance? Question 15 options: Leptin TNF-alpha Adiponectin ResistinInsulin binding to its cellular receptor causes dimerization and phosphorylation of ( ) and assembly of a ( ) membrane transporter.TRUE or FALSE: Type 2 diabetes is a ketosis-resistant, adult-onset diabetes that always require insulin therapy
- Which of the following medications does not have hyperglycemic effects (does not decrease insulin actions)?A. Calcium channel blockersB. Beta 2 agonistsC. PentoxifyllineD. H2 histamine receptor blockersE. IsoniazidWhy can't insulin be given per os? Please explain Please cite/reference the book in which you found the solution.Hi, can you please help me with the following multiple choice question - Thanks!! Correct cause-and-effect relationships following insulin withdrawal in a person with diabetes mellitus include: the ratio of K+ concentration inside the cell to K+ concentration outside the cell decreases in untreated diabetes for multiple reasons, including intracellular H+ buffering, which results in a shift of K+ to the extracellular compartment. ketonemia does not increase urine flow because it is entirely reabsorbed in the renal tubule urinary phosphate decreases because renal excretion of H+ results in increased phosphate reabsorption. serum sodium rises because of fluid shift from the extracellular compartment to intracellular compartment. glomerular filtration rate increases as a result of increased serum glucose concentration. The most effective direct stimulus for the release of glucagon is: an increase in serum glucose an decrease in serum glucose somatostatin insulin (direct action on the…