Given what you have learned from the first experiment, you prepare the other leg for a fresh experiment. When you add acetylcholine to the preparation, the muscle contracts. You add curare, and again add acetylcholine to the preparation, and the muscle does not contract. Based on these data, and the data from the first experiment, this means curare is potentially acting on: O Both the alpha motor neurons' ability to conduct action potentials, and the muscle's ability to conduct action potentials, but not on the neuromuscular junction. O Both the alpha motor neurons' ability to conduct action potentials, and the neuromuscular junction, but not the muscle's ability to conduct action potentials. O Both the neuromuscular junction and the muscle's ability to conduct action potentials, but not on the alpha motor neurons' ability to conduct action potentials. It must only be affecting the muscle's ability to conduct action potentials. It must only be affecting the neuromuscular junction. O It must only be affecting the alpha motor neurons' ability to conduct action potentials. O It could potentially be affecting any of these components.

Given what you have learned from the first experiment, you prepare the other leg for a fresh experiment. When you add acetylcholine to the preparation, the muscle contracts. You add curare, and again add acetylcholine to the preparation, and the muscle does not contract. Based on these data, and the data from the first experiment, this means curare is potentially acting on: O Both the alpha motor neurons' ability to conduct action potentials, and the muscle's ability to conduct action potentials, but not on the neuromuscular junction. O Both the alpha motor neurons' ability to conduct action potentials, and the neuromuscular junction, but not the muscle's ability to conduct action potentials. O Both the neuromuscular junction and the muscle's ability to conduct action potentials, but not on the alpha motor neurons' ability to conduct action potentials. It must only be affecting the muscle's ability to conduct action potentials. It must only be affecting the neuromuscular junction. O It must only be affecting the alpha motor neurons' ability to conduct action potentials. O It could potentially be affecting any of these components.

Human Physiology: From Cells to Systems (MindTap Course List)

9th Edition

ISBN:9781285866932

Author:Lauralee Sherwood

Publisher:Lauralee Sherwood

Chapter8: Muscle Physiology

Section: Chapter Questions

Problem 4TAHL

See similar textbooks

Similar questions

What is a motor unit? Why does a rapid series of muscle twitches yield a stronger overall contraction than a single twitch?
Figure 38.37 Which of the following statements about muscle contraction is true? The power stroke occurs when ATP is hydrolyzed to ADP and phosphate. The power stroke occurs when ADP and phosphate dissociate from the myosin head. The power stroke occurs when ADP and phosphate dissociate from the actin active site. The power stroke occurs when Ca2+ binds the calcium head.
We move a lot and do not even realize how intricate these movements may be from gross motor to fine motor manipulations. Briefly explain the steps involved in a single muscle contraction beginning at the neuromuscular junction through one complete contraction and relaxation for the sliding filament theory. Don’t forget to include all the major proteins (actin, myosin, troponin and tropomyosin) involved as well as calcium and ATP.
Which of the following will produce the LARGEST force of twitch contraction? Explain your answer.a. Stimulation of all alpha motor neurons in a muscleb. Stimulation of all alpha motor neurons plus all gamma motor neurons in a musclec. Both stimulations will cause a similar force of twitch contraction
In the process called "excitation-contraction coupling" (the events that lead up to a muscle cell's contraction), what part of this process is disrupted by death? (Actually, all of it is disrupted by death, but we can pinpoint a linkage between death, and the onset of rigor mortis--as well as explaining why rigor mortis eventually ends.)
In theory, could a muscle cell get away with having only acetylcholine-gated cation channels, which would serve the dual purpose of receiving neurotransmitter signals and propagating action potentials? Why or why not?
In the process of muscle contraction, which of the following steps immediately comes after the propagation of an action potential down the t-tubules of a myofiber? sliding of tropomyosin causing exposure of actin binding of myosin head to actin's active site release of calcium from the terminal cisternae binding of acetylcholine to its receptor influx of sodium at the sarcolemma
Why is it important to remove acetylcholine from its binding to acetylcholine receptor once neural stimulus ends? What can be the possible consequence of non-removal of acetylcholine to the skeletal muscle fiber? please do not just copy from google
Which of the following statements accurately describes the pattern of recruitment in a muscle when performing a maximal force contraction? A. A few small and medium motor units will be recruited; if that's not enough, then all of them will be. B. Small motor units are recruited first, then medium, and then the largest to reach maximum force.
In light of the “all or none” law of muscle contraction, how can you explain twitch recruitment (also called the graded response) when stimulating the muscle versus stimulating the nerve? Explain the difference in stimulation voltage required to elicit a maximum response when stimulating the nerve versus the muscle directly?
When an action potential from a motor neuron arrives at the neuromuscular junction, a series of events occurs that leads to muscle contraction. Arrange following events in the correct order from first to last.
In the process of muscle contraction, which of the following steps immediately precedes the influx of sodium into a myofiber at the neuromuscular junction? release of calcium from the terminal cisternae sliding of tropomyosin causing exposure of actin propagation of an action potential down the t-tubules binding of myosin head to actin's active site binding of acetylcholine to its receptor