If the gene for B-catenin is knocked out, what will happen to synapse development? a) Adhesion molecules will be anchored too tightly b) The number of synaptic spines will decrease c) Synaptic spines will not be pruned d) Synaptic spines will be over-pruned
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- Consider the circuit from Q5 (above). We want to add another inhibitory neuron to this circuit to increase the likelihood that neuron D will have an action potential. With which cell (A, B, C or D) should our new inhibitory neuron make a synapse to make it more likely that the post - synaptic neuron will have anaction potential?The diagram below shows a simple neural circuit with three pre - synaptic cells labelled 'A', 'B' and 'C', and a post - synaptic cell labelled 'D ' (see image below). Assume neurons A and B each have outputs of +2 and neuron C has output of -1.Which two proteins are essential for vesicle fusion at the synapse? v-snare, t-snare Snap, GAP synaptobravin, syntaxin NSF, COPSHow do proteins and other chemicals synthesized in the soma get to the axon terminals? By what process can a virus that invades a peripheral nerve fiber get to the soma of that neuron?
- What happens to synaptic transmission during an experiment if the preparation (containing 1 αmotor neuron and 1 skeletal muscle) is bathed in a low calcium solution?How is the synaptic cleft cleaned up to end Signal Transmission?Which of the following does not contribute to propagation of action potentials? a. As the area outside the membrane becomes negative, itattracts ions from adjacent regions; as the inside of the membrane becomes positive, it attracts negative ions from nearby in the cytoplasm. These events depolarize nearby regions of the axon membrane. b. The refractory period allows the impulse to travel in only one direction. c. Each segment of the axon prevents the adjacent segments from firing. d. The magnitude of the action potential stays the same as it travels down the axon. e. Up to a limit, increasing the intensity of the stimulus increases the number of action potentials.
- . Is protein synthesis necessary for short-term synaptic plasticity, long-term synaptic plasticity, or both? Please explain.synaptobrevin A-is activated by Sodium cations B-is an extracellular signalling molecule C-binds and stimulates post synaptic receptors D-is a synaptic protein involved in vesicular fusion with cytoplasmic membraneDescribe the process of obtaining the code for a patient that fell from a ladder and fractured their radius? Is falling from the ladder enough information?
- What is abortifacients?1.1 What would be the effect of blocking neurotransmitter degradation on neurotransmitter concentration at the synapse? 1.2You are studying an excitatory synapse that is not strong enough to evoke a postsynaptic action potential. Would the probability of a postsynaptic potential increase or decrease after blocking neurotransmitter degradation? 1.3 ) You are studying an excitatory synapse that evokes a postsynaptic action potential. Would the probability of a postsynaptic potential increase or decrease after blocking presynaptic voltage-gated calcium channels?How does a signal cross the synaptic cleft?