pe's dad had SCD and his mom.had normal hemoglobin. His partner Sue's brother also has the disease, but Sue has never had her arrier status tested. They have sent protein samples to Genetics R Us for analysis. • Choose all CORRECT statements from the choices below. • (more than 1, less than all - choosing all = zero points). Joe can produce two types of gametes: one with hemoglobin A gene (HA) and one with hemoglobin S gene (HS), Joe's genotype must be heterozygous. Joe has sickle cell disease; he got it from his dad. We can determine if Sue is a carrier by whether or not she has the slow-moving hemoglobin band. The mutant hemoglobin S moves more slowly because it has a much greater negative charge than normal hemoglobin A.
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- The father of five children begins to show symptoms of huntington disease. what is the probability that same, the man's second oldest son (11-2),, will suffer from the disease if he lives a normal life span? Sam's mother and her ancestors do not have the diseas. Sam's father was adopted, so we do not know his biological family history for hunutington disease. Explore all possible outcome for sam and explain your reasoning. A gene called Q has two alleles, QFand QG, that encode alternative forms of a red blood cell protein thatallows blood group typing. A different, independentlysegregating gene called R has two alleles, RC and RD,permitting a different kind of blood group typing. Arandom, representative population of football fanswas examined, and on the basis of their blood typing,the following distribution of genotypes was inferred(all genotypes were distributed equally betweenmales and females):QFQFRCRC 202QFQG RCRC 101QGQG RCRC 101QFQFRCRD 372QFQG RCRD 186QGQG RCRD 186QFQFRDRD 166QFQG RDRD 83QGQG RDRD 83This sample contains 1480 fans in total.a. Is the population at Hardy-Weinberg equilibriumwith respect to either or both of the Q andR genes?b. After one generation of random mating within thisgroup, what fraction of the next generation of football fans will be QFQF(independent of their Rgenotype)?c. After one generation of random mating, what fraction of the next generation of football fans will…Elise is a freshman college student taking Introduction to Biology. In lab, she is asked her parents’ blood types to complete a Punnett square. She knows that she has type A blood and that her mother has type B, but she does not know her father’s blood type. Elise’s professor assures her that she can figure out her father’s blood type just by knowing her own and her mother’s.How can this be determined, and what is her father’s blood type?
- Introduction: A person’s blood type is an inherited trait governing special marker proteins appear-ing on the surface of their red blood cells. Blood typing is important, because it governs who anindividual can give or receive blood donations from; blood with proteins markers not present in theirown blood will be rejected by an individual’s immune system.There are two separate genes that govern blood type; the ABO gene has two dominant options, Aand B, and a recessive option called O. For example, an individual with AO or AA would presentthe A protein, and an individual with AB will present both the A and B proteins on their blood;an individual with OO has no proteins from this gene. The Rh gene has two options; the dominantoption is called positive (or + for short), while the recessive option is called negative or null (or –for short). For example, an individual with ++ or +– would present the Rh protein on their blood,while an individual with – – would not present the Rh…Introduction: A person’s blood type is an inherited trait governing special marker proteins appear-ing on the surface of their red blood cells. Blood typing is important, because it governs who anindividual can give or receive blood donations from; blood with proteins markers not present in theirown blood will be rejected by an individual’s immune system.There are two separate genes that govern blood type; the ABO gene has two dominant options, Aand B, and a recessive option called O. For example, an individual with AO or AA would presentthe A protein, and an individual with AB will present both the A and B proteins on their blood;an individual with OO has no proteins from this gene. The Rh gene has two options; the dominantoption is called positive (or + for short), while the recessive option is called negative or null (or –for short). For example, an individual with ++ or +– would present the Rh protein on their blood,while an individual with – – would not present the Rh…Adenike and her partner, Debare, are expecting their first child. Adenike is healthy as are her two sisters but her brother has PKU, a recessive disorder of the PAH gene. Genotypically he must be pah-/pah-. Neither of Adenike's parents has PKU but both of her grandmothers did. Debare is a carrier of Sickle Cell trait, which means he has one allele for wildtype hemoglobin (HbA) and one allele for Sickle Cell (HbS), making him HgA/HbS. More importantly, he has two alleles for Marfan Syndrome, a pleiotropic dominant disorder that can affect up to 30 different traits (M+/M+). No one in Debare's family has ever had PKU and no one in Adenike’s family has ever had Sickle Cell or Marfan Syndrome. They consult a genetics counselor over concern of the health of their baby. The genetics counselor collects the medical histories of three generations; their grandparents, their parents, and them. In her calculations, she produces a phenotypic key that indicates M = Marfan, Hb = sickle cell, and pah-…
- Adenike and her partner, Debare, are expecting their first child. Adenike is healthy as are her two sisters but her brother has PKU, a recessive disorder of the PAH gene. Genotypically he must be pah-/pah-. Neither of Adenike's parents has PKU but both of her grandmothers did. Debare is a carrier of Sickle Cell trait, which means he has one allele for wildtype hemoglobin (HbA) and one allele for Sickle Cell (HbS), making him HgA/HbS. More importantly, he has two alleles for Marfan Syndrome, a pleiotropic dominant disorder that can affect up to 30 different traits (M+/M+). No one in Debare's family has ever had PKU and no one in Adenike’s family has ever had Sickle Cell or Marfan Syndrome. They consult a genetics counselor over concern of the health of their baby. The genetics counselor collects the medical histories of three generations; their grandparents, their parents, and them. In her calculations, she produces a phenotypic key that indicates M = Marfan, Hb = sickle cell, and pah-…Adenike and her partner, Debare, are expecting their first child. Adenike is healthy as are her two sisters but her brother has PKU, a recessive disorder of the PAH gene. Genotypically he must be pah-/pah-. Neither of Adenike's parents has PKU but both of her grandmothers did. Debare is a carrier of Sickle Cell trait, which means he has one allele for wildtype hemoglobin (HbA) and one allele for Sickle Cell (HbS), making him HgA/HbS. More importantly, he has two alleles for Marfan Syndrome, a pleiotropic dominant disorder that can affect up to 30 different traits (M+/M+). No one in Debare's family has ever had PKU and no one in Adenike’s family has ever had Sickle Cell or Marfan Syndrome. They consult a genetics counselor over concern of the health of their baby. The genetics counselor collects the medical histories of three generations; their grandparents, their parents, and them. In her calculations, she produces a phenotypic key that indicates M = Marfan, Hb = sickle cell, and pah-…Sickle cell anemia is a disease that is caused by a mutation in the gene that produces hemoglobin. Hemoglobin carries oxygen in red blood cells. The HbA allele produces normal hemoglobin and the HbS allele produces hemoglobin that sticks together and causes red blood cells to sickle. Heterozygous individuals (HbAHbS) produce both normal and "sickle" hemoglobin so the HbA and HbS alleles are codominant. Heterozygotes do not develop sickle cell anemia and are described as having the sickle cell trait. Individuals that are homozygous for the sickle allele (HbSHbS) only produce "sickle" hemoglobin and develop sickle cell disease.A man who is homozygous for the normal allele married a woman with the sickle cell trait. What is the expected probability of this couple having a boy with the sickle cell trait? Record your answer as a value between 0 and 1 rounded to two decimal places.Answer
- Sickle cell anemia is a disease that is caused by a mutation in the gene that produces hemoglobin. Hemoglobin carries oxygen in red blood cells. The HbA allele produces normal hemoglobin and the HbS allele produces hemoglobin that sticks together and causes red blood cells to sickle. Heterozygous individuals (HbAHbS) produce both normal and "sickle" hemoglobin so the HbA and HbS alleles are codominant. Heterozygotes do not develop sickle cell anemia and are described as having the sickle cell trait. Individuals that are homozygous for the sickle allele (HbSHbS) only produce "sickle" hemoglobin and develop sickle cell disease. A man with the sickle cell trait married a woman with the sickle cell trait. Determine the probability that they will have children with the sickle cell trait or sickle cell disease.Record your answer as a value between 0 and 1 rounded to two decimal places. AnswerFor practice, let's work with blood groups. Three alleles exist for human blood types. They were not numbered, but instead marked with the letters for A, B and O. The labeling convention is : IA, IB, and Ii. IA and IB are codominant alleles, whereas Ii is the recessive allele for type O. Humans are diploid; every individual carries only two alleles. For the following question list all 6 possible Blood group genotypes. The first has been done for you below:Sickle cell anemia is a genetic disease most common in those of African ancestry that results from the mutation of a single basepair. This mutation results in a change to the hemoglobin protein. In this experiment, “S” will represent the normal allele and “s” thesickle allele. They are codominant alleles. Therefore, SS is normal, Ss is not fatal but mildly symptomatic, and ss is debilitating,painful, and often fatal. After hundreds or even thousands of generations, both alleles are still common in those of African ancestry. How would you explain this?