
Human Anatomy & Physiology (11th Edition)
11th Edition
ISBN: 9780134580999
Author: Elaine N. Marieb, Katja N. Hoehn
Publisher: PEARSON
expand_more
expand_more
format_list_bulleted
Concept explainers
Question
Pigment of chicken feathers is regulated by two genes, the gene for feather pigment C, and a gene that inhibits pigment production, I. A mother chicken, who is homozygous dominant for gene C, and heterozygous for gene I, is crossed with a father chicken, who is heterozygous for the pigment gene, and heterozygous for gene I.
Inhibition is dominant to uninhibited, pigmented feathers are dominant to unpigmented feathers.
What is the probability of one of the offspring of these two chickens having white feathers? (percentage)
Expert Solution

Trending nowThis is a popular solution!
Step by stepSolved in 4 steps with 1 images

Knowledge Booster
Learn more about
Need a deep-dive on the concept behind this application? Look no further. Learn more about this topic, biology and related others by exploring similar questions and additional content below.Similar questions
Pigment of chicken feathers is regulated by two genes, the gene for feather pigment C, and a gene that inhibits pigment production, I. A mother chicken, who is homozygous dominant for gene C, and heterozygous for gene I, is crossed with a father chicken, who is heterozygous for the pigment gene, and heterozygous for gene I.Inhibition is dominant to uninhibited, pigmented feathers are dominant to unpigmented feathers.In a population of 150 chickens, produced from the same parents, how many chickens would you expect to have pigmented feathers?
arrow_forward
Pigment of chicken feathers is regulated by two genes, the gene for feather pigment C, and a gene that inhibits pigment production, I. A mother chicken, who is heterozygous for gene c, and heterozygous for gene I, is crossed with a father chicken, who is homozygous recessive for the pigment gene, and heterozygous for gene I.
Inhibition is dominant to uninhibited, pigmented feathers are dominant to unpigmented feathers.Â
The inhibitor gene (I) will prevent expression of pigmented chicken feathers.
Here is a DihybridCrossReview(8).pdf  Download DihybridCrossReview(8).pdfreviewing the FOIL method for determining gametes and performing a dihybrid cross if you need a refresher.
Use the letters C, c, I, and i for the genes. C is for color and I is for inhibitor
Enter your answer as a list of haplotypes separated by spaces such as AB ab Ab aB.
Do not write a sentence.Â
Do not list duplicate haplotypes.
Alphabatize your haplotypes, e.g. Ab, not bA.
A) What are the potential genotypes of…
arrow_forward
Can ypu answer ALL parts to this question
Pigment of chicken feathers is regulated by two genes, the gene for feather pigment C, and a gene that inhibits pigment production, I. A mother chicken, who is heterozygous for gene c, and heterozygous for gene I, is crossed with a father chicken, who is homozygous recessive for the pigment gene, and heterozygous for gene I.
Inhibition is dominant to uninhibited, pigmented feathers are dominant to unpigmented feathers.Â
The inhibitor gene (I) will prevent expression of pigmented chicken feathers.
Â
Use the letters C, c, I, and i for the genes. C is for color and I is for inhibitor
Enter your answer as a list of haplotypes separated by spaces such as AB ab Ab aB.
Do not write a sentence.Â
Do not list duplicate haplotypes.
Alphabatize your haplotypes, e.g. Ab, not bA.
A) What are the potential genotypes of the gametes produced by the mother for these two genes?  Â
B) What are the potential genotypes of the gametes produced by the father for…
arrow_forward
Coat color in mice is influenced by two genes, one for color (A) and one for the amount of pigment production (C). Mice with the wild type agouti coat color have a yellowish to brownish color. Mice also have a gene that determines the amount of pigment the hair produces.
Mouse Coat Phenotype and Genotype CorrelationÂ
Agouti coat
AA, Aa
Solid gray/black coat
aa
Pigment production
CC, Cc
Albinism
cc
Multiple crosses were made between male and female mice that were each heterozygous for both traits (AaCc). The data table shows the number of mice of each coat type. Calculate the average F1 generation coat color to answer the question.
Coat Color
Number of Mice Born in Each Trial Cross
Mean Number of Mice
1
2
3
4
5
6
7
8
9
10
Agouti coat
11
8
9
9
10
8
10
7
10
9
Â
Solid gray/black coat
5
2
1
3
4
3
4
3
3
2
Â
Albinism
3
3
6
4
4
3
3
7
4
3
Â
Which…
arrow_forward
In humans, free ear lobes (E) is dominant over attached ear lobes (e), and brown eyes (B) is dominant over blue eyes (b). A man with free ear lobes and brown eyes married a woman with attached ear lobes and brown eyes. Of four children, one has free ear lobes and brown eyes, one has free ear lobes and blue eyes, one has attached ear lobes and brown eyes, and one has attached ear lobes and blue eyes. Determine the genotypes of both parents.
arrow_forward
Kernel color in wheat
Red kernel color in wheat results from the interaction between two dominant alleles. With only one dominant allele, the phenotype is brown while in the absence of any dominant allele, the phenotype is white. Suppose that plants of a variety that is true breeding for red kernels are crossed with plants true breeding for white kernels.
Â
What specific type of interaction can be observed?Â
A. duplicate dominant genes
B. duplicate genes with cumulative effects
C. complementary genes
D. dominant epistasis (case 2)
arrow_forward
In wheat, aleurone cells form a thin layer of the seed coat that is critical to early gene expression in plant development. The color of this layer of cells is controlled by two alleles of a gene [colored aleurone (R) is dominant to colorless (r)]. A second gene is known to control the color of leaf tips [green leaf tip (G) is dominant to yellow (g)]. Two plants, each heterozygous for both characteristics, are test crossed to homozygous recessives, and their progeny are combined to produce the following totals:
colored green 102
colored yellow 98
colorless green 103
colorless yellow 97
a) Use chi-square analysis to test these data for an independent assortment of the two characteristics (table provided). Please show work, how your expected values are calculated, and explain what your results indicate about the data.Â
b) You decide to be cautious in your analysis, and decide to analyze the progeny from each of the crosses individually (instead of adding them together as shown above).…
arrow_forward
Two different genes control the expression of kernel colour in Mexican black corn: a black pigment gene (B) and a dotted pigment gene (D). Gene B influences the expression of gene D. The dotted phenotype appears only when gene B is in the homozygous recessive state. A colourless variation occurs when both genes are homozygous recessive.
After pure-breeding black-pigmented plants were crossed with colourless plants, all offspring were black-pigmented.
Plants of the F 1generation are suspected of being heterozygous for both genes. A test cross of colourless plants with the heterozygote plants should produce a genotypic ratio in the offspring of
A. 1:0
B. 3:1
C. 2:1:1
D. 1:1:1:1
arrow_forward
In onions, presence of gene C causes production of an enzyme which catalyzes theproduction of bulb-color pigment. Gene K prevents this enzyme from functioning, thuspreventing enzyme production. The two genes are inherited independently. A true breedingcolored variety is crossed with a white one of genotype ccKK. (a) what are the expectedphenotypes and ratios in the F1 and F2. (b) Another pair of alleles R and r, independent ofgenes C and K controls the specific color for bulbs, R giving red and r, yellow bulbs. If thetrue-breeding colored variety are homozygous for R and the white ones homozygous for r,what would be the phenotypic ratios in the F1 and F2 of the above cross?
arrow_forward
Two different genes control the expression of kernel colour in Mexican black corn: black pigment gene B and dotted pigment gene D. Gene B influences the expression of gene D. The dotted phenotype appears only when gene B is in the homozygous recessive state. A colourless variation occurs when both genes are homozygous recessive.
After pure-breeding black-pigmented plants were crossed with colourless plants, all of the offspring were black-pigmented.
What is the probability of dotted offspring being produced from the test cross described above?
(Record your answer as a percentage - do not include a decimal).
arrow_forward
In onions, presence of gene C causes production of an enzyme which catalyzes theproduction of bulb-color pigment. Gene K prevents this enzyme from functioning, thuspreventing enzyme production. The two genes are inherited independently. A true breedingcolored variety is crossed with a white one of genotype ccKK. (b) Another pair of alleles R and r, independent of genes C and K controls the specific color for bulbs, R giving red and r, yellow bulbs. If the true-breeding colored variety are homozygous for R and the white ones homozygous for r, what would be the phenotypic ratios in the F1 and F2 of the above cross?
arrow_forward
In silkmoths (Bombyx mori), red eyes (re) and white-banded wings (wb) are encoded by two mutant alleles that are recessive to those that produce wild-type traits (re+ and wb+); these two genes are on the same chromosome. A moth homozygous for red eyes and white-banded wings is crossed with a moth homozygous for the wild-type traits. The F1 have wild-type eyes and wild-type wings. The F1 are crossed with moths that have red eyes and white-banded wings in a testcross. The progeny of this testcross are wild-type eyes, wild-type wings red eyes, wild-type wings wild-type eyes, white-banded wings red eyes, white-banded wings a. What phenotypic proportions would be expected if the genes for red eyes and for white-banded wings were located on different chromosomes? b. What is the rate of recombination between the gene for red eyes and the gene for white-banded wings?
arrow_forward
arrow_back_ios
SEE MORE QUESTIONS
arrow_forward_ios
Recommended textbooks for you

Human Anatomy & Physiology (11th Edition)
Biology
ISBN:9780134580999
Author:Elaine N. Marieb, Katja N. Hoehn
Publisher:PEARSON

Biology 2e
Biology
ISBN:9781947172517
Author:Matthew Douglas, Jung Choi, Mary Ann Clark
Publisher:OpenStax

Anatomy & Physiology
Biology
ISBN:9781259398629
Author:McKinley, Michael P., O'loughlin, Valerie Dean, Bidle, Theresa Stouter
Publisher:Mcgraw Hill Education,

Molecular Biology of the Cell (Sixth Edition)
Biology
ISBN:9780815344322
Author:Bruce Alberts, Alexander D. Johnson, Julian Lewis, David Morgan, Martin Raff, Keith Roberts, Peter Walter
Publisher:W. W. Norton & Company

Laboratory Manual For Human Anatomy & Physiology
Biology
ISBN:9781260159363
Author:Martin, Terry R., Prentice-craver, Cynthia
Publisher:McGraw-Hill Publishing Co.

Inquiry Into Life (16th Edition)
Biology
ISBN:9781260231700
Author:Sylvia S. Mader, Michael Windelspecht
Publisher:McGraw Hill Education

Human Anatomy & Physiology (11th Edition)
Biology
ISBN:9780134580999
Author:Elaine N. Marieb, Katja N. Hoehn
Publisher:PEARSON

Biology 2e
Biology
ISBN:9781947172517
Author:Matthew Douglas, Jung Choi, Mary Ann Clark
Publisher:OpenStax

Anatomy & Physiology
Biology
ISBN:9781259398629
Author:McKinley, Michael P., O'loughlin, Valerie Dean, Bidle, Theresa Stouter
Publisher:Mcgraw Hill Education,

Molecular Biology of the Cell (Sixth Edition)
Biology
ISBN:9780815344322
Author:Bruce Alberts, Alexander D. Johnson, Julian Lewis, David Morgan, Martin Raff, Keith Roberts, Peter Walter
Publisher:W. W. Norton & Company

Laboratory Manual For Human Anatomy & Physiology
Biology
ISBN:9781260159363
Author:Martin, Terry R., Prentice-craver, Cynthia
Publisher:McGraw-Hill Publishing Co.

Inquiry Into Life (16th Edition)
Biology
ISBN:9781260231700
Author:Sylvia S. Mader, Michael Windelspecht
Publisher:McGraw Hill Education