• Set A₁A₁ (HbAA) • Set A₁A₂ (HbAS) • Set A₂A₂ (Hbss) Using your simulations as examples, explain why the HbS allele is more common in some parts of the world than others. Include information about the genotypes in your response.
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- . A gene called Q has two alleles, QFand QG, that encode alternative forms of a red blood cell protein thatallows blood group typing. A different, independentlysegregating gene called R has two alleles, RC and RD,permitting a different kind of blood group typing. Arandom, representative population of football fanswas examined, and on the basis of their blood typing,the following distribution of genotypes was inferred(all genotypes were distributed equally betweenmales and females):QFQFRCRC 202QFQG RCRC 101QGQG RCRC 101QFQFRCRD 372QFQG RCRD 186QGQG RCRD 186QFQFRDRD 166QFQG RDRD 83QGQG RDRD 83This sample contains 1480 fans in total.a. Is the population at Hardy-Weinberg equilibriumwith respect to either or both of the Q andR genes?b. After one generation of random mating within thisgroup, what fraction of the next generation of football fans will be QFQF(independent of their Rgenotype)?c. After one generation of random mating, what fraction of the next generation of football fans will…Explain in your own words the predictions of the “standard model of population genetics” as depicted in Figure 1.I'm confused about how I would figure out these two questions as I don't really understand the difference or where to start. What would genotypic frequencies be for Wildrose at pt-8 if it were in equilibrium? and How many individuals (to 2 decimal places) of each genotype would there be from Wildrose at pt-8 if it were in equilibrium?
- e. Why can’t we learn everything about a single trait with a genome-wide association study?According to Amar J. S. Klar, is there a such thing as “left handedness”? What is the preferred term? Describe how the genetics works, according to his hypothesis. What are the alleles, and which combinations of alleles make for which type of hand use?Using the HardyWeinberg Law in Human Genetics Suppose you are monitoring the allelic and genotypic frequencies of the MN blood group locus (see Question 2 for a description of the MN blood group) in a small human population. You find that for 1-year-old children, the genotypic frequencies are MM = 0.25, MN = 0.5, and NN = 0.25, whereas the genotypic frequencies for adults are MM = 0.3, MN = 0.4, and NN = 0.3. a. Compute the M and N allele frequencies for 1-year-olds and adults. b. Are the allele frequencies in equilibrium in this population? c. Are the genotypic frequencies in equilibrium?
- While studying the frequency of sickle-cell disease ("sickle cell anemia") in a population living in sub-Saharan Africa, you obtain the following data from a sample of n= 100 people (note that I chose a simpler system for identifying the alleles rather than using "Hb S" for sickle cell allele), which is the actual name and what you used in lab). What is the frequency of the sickle cell allele (b) in the sample below from a human population? Sample Data BB-60 individuals (No sickle cell disease) Bb-30 individuals (No sickle cell disease) bb-10 individuals (Sickle cell disease) 1. 0.25 2. 0.10 3. 0.35 4. 0.60 5. 0.20Below is a histogram showing the results from a previous class’s dataset. Some trials resulted (ended) with the white allele fixing (reaching 100%), some resulted with the white allele going extinct (0%), and some fell somewhere in between. What phenomenon most likely drove this pattern?Imagine that genetic technology advances to the point where the genetic profile of an individual can be easily obtained, and that we have a full understanding of how genes interact to influence our health. Based on this genetic information alone, how reliably do you think we would be able to predict a person's chance of getting a specific disease? Where do you think this type of genetic profiling would lead with respect to selection of offspring or genetic superiority?
- Could you help fill in tables UV-13 to find the allele and genotype frequencies using the Hardy- Weinberg equations Thank youIn garden pea plants, the yellow seed colour (Y) is dominant over the green seed colour (y).In a population of 10 pea plants, 5 pea plants are homozygous dominant, 3 are heterozygous dominant, and 2 are recessive.Find the frequency of thea. YY genotype Answerb. Yy genotype Answerc. yy genotype AnswerRecord your response using three significant digits.****Note for question 1 - you have been given the frequency of the two alleles (dominant and recessive = p + q=1) and you are using those allele frequencies to calculate the genotype frequencies. p2 + 2pq + p2