Cardiomyopathy

It is a normal day, and you decide to go bike riding with your son. You hear wheezing all of the sudden, and the next thing you know, your son is crashing onto the cement unconscious. What happened to him? Is he okay? These are questions that you have to answer because you happen to have a blind spot in your knowledge when it comes to your family’s heart health. Unfortunately, you are not the only one who has heart disease in your family and isn’t knowledgeable on the subject. Education about one’s

In cardiomyopathy the heart muscle becomes enlarged, thick or rigid, and in rare cases the muscle tissue can be replaced with scar tissue. As this disease worsens, the heart becomes weaker and unable to pump blood normally through the body and maintain a normal electrical rhythm. This can lead to heart failure or irregular heartbeats. The weakening of the heart also can lead to other problems such as heart valve problems. Cardiomyopathy can be acquired or inherited. It

Introduction Sudden cardiac death (SCD) is the leading cause of death in exercising young athletes. 1 Current estimates of the incidence of SCD in athletes range from almost one in a million to 1:23,000 athletes per year, while some subpopulations of athletes are reported at even higher risk with an incidence of 1 in 3,000. 2 Such deaths often receive more attention because of the youth of the victims and the perception that physically trained athletes are healthy. 3 SCD is often a result of structural

Crystal is a 20yo, G2 P1001, who was seen for a follow-up ultrasound assessment. The patient’s primary issue is the development of a postpartum cardiomyopathy after her 1st delivery in 2015. She also reportedly had an anti-M antibody but all of her antibody screens have been negative including one 3 weeks ago. She recovered from the cardiomyopathy and overall is asymptomatic. Her BP was normal today at 127/76 and her heart rate was 98. She states that she actually came to triage 3 weeks ago and

Chapter 1 THE PROBLEM AND ITS BACKGROUND   Introduction Having a heart disease is one of the most life-threatening experiences a particular person could ever had. Half of his or her life is in danger; not knowing when will he/she die. That's why people should take good care of themselves, especially their hearts; for heart is one of the main body parts in human anatomy. Heart disease is very different from heartbreaks; that after you get hurt, you'll just move on and    "…the heart is the most

Coronary artery bypasses surgery, percutaneous angioplasty-stent, aspirin use, cholesterol-lowering therapies, and increased awareness of coronary artery disease prevention are the main strategies for the preventive treatment of SCD [166]. As the main etiology behind coronary artery ischemia is the plaque rupture, methods to prevent plaque rupture, plaque stabilization, decreasing plaque formation and searching risk markers for sudden plaque rupture should be discovered. The role of cholesterol in

Dr. Volk had several different heart issues that resulted in his eventual heart transplant. One of the issues was called dilated cardiomyopathy, or the enlargement of chambers of the heart that typically starts with the enlargement of the left ventricle (American Heart Association, 2016). When a heart is enlarged too much the cardiac muscles can’t work as efficiently resulting in weaker contractions. Since his left ventricle was enlarged he was unable to pump a sufficient amount of blood to his body

Ghanshyam Patel, EMPL ID: 23591518 CHSS Research Paper Instructor: Dr. Christian Grov SUDDEN CARDIAC DEATH Author: Ghanshyam Patel MBBS, MPH EMPL Id: 23591518, Ghanshyam.patel18@sphmail.cuny.edu FROM, CUNY Graduate School of Public Health and Health Policy, 55 West 125th Street, New York, NY 10027, USA Abstract: Sudden cardiac death (SCD) is major public health problem. SCD continues to be one of the leading causes of mortality worldwide, with an annual incidence estimated at 250000 to

Introduction: Cardiac amyloidosis typically presents as a restrictive cardiomyopathy (CM) with slowly progressive heart failure (HF). Symptoms result from pulmonary edema, pleural effusions, and often right HF with peripheral edema. Echocardiographic findings include enlarged atria, diastolic dysfunction, and wall thickening due to amyloid deposition that typically results in low voltage on the ECG. Transthyretin cardiac amyloidosis (ATTR) and Light Chain (AL) cardiac amyloidosis are the most common

Introduction N-terminal prohormone of brain natriuretic peptide (NT-proBNP) is an inactive byproduct of brain natriuretic peptide (BNP). They are released from myocytes after the prohormone BNP (ProBNP) is cleaved into the NT-proBNP and the BNP. The synthesis of ProBNP and the release of NT-proBNP and BNP are caused by cardiac wall stress including stretching, ischemic and/or hypoxic stimuli of myocytes. Although NT-proBNP is not the active polypeptide product, it is less labile and possesses a