Cerebellar lesions

without scars or lesions noted, umbilicus is midline and inverted 2. Bowel Sounds Active x 4 quadrants without bruits noted 3. Vascular Sounds not ausculated. 4. Light and deep palpation without tenderness, mass or guarding evidenced. 5. Palpation of liver – edge not palpated, percussion approximated at 8 cm in Right mid clavicular line. Spleen non palpable. Upper Extremities: 1. Upper Extremities are symmetrical in appearance without scaring, lesions or tenderness evident

Oculomotor (III), trochlear (IV), Abducens (VI) cranial nerves Although each of these nerves control separate extraocular muscles, they are normally examined together due to their close functional interrelationships. • Look Similar to other cranial nerve examination, start with inspection of the eyes. Look at - The position of the head position: If diplopia is present, the head turned or tilted to minimize double vision. - Inspect for ptosis and eye position. - Ask the child to look at an

can use soap, perfume, etc. if patient able to recognized the smell that indicates it’s normal. But if patient not able to be recognized the smell of the stimulant that indicates its damage to all factory nerve. If unilateral loss It could be brain lesion affecting the olfactory bulb or blocked nasal passage. bilateral loss can occur with rhinitis. XII Hypoglossal Nerve: This nerve is a motor to the tongue. For this test, you can use a tongue depressor. In this test, physician observes the tongue atrophy

CHAPTER 13: VERTIGO INTRODUCTION Vertigo is a symptom of illusory movement, commonly spinning, swaying or tilting. Some perceive themselves to be in motion while others perceive motion of the environment around them. Vertigo arises because of asymmetry in the vestibular system due to damage to or dysfunction of the labyrinth, vestibular nerve, or central vestibular structures in the brainstem. The patient who presents to an office visit with dizziness, vertigo or other neuro-otologic symptoms is

Due to this, it has been deemed difficult to determine which deficit is the consequence of which part of a lesion. To overcome this problem, other methods are being used to aid in the visualisation of memory processes in the healthy parts of the brain. These come in the form of functional neuroimaging studies using Positron Emission Tomography (PET) and Functional

This study uses secondary research in order to make connections between ideas and concepts that can illuminate the topic. Through search of databases and online book resources, the development of a rich foundation of resources can help to explore the subject matter. Using keywords to define the search, the literature can be used in order to determine how connections can be made between PTSD and TBI. Once the literature was accumulated and reviewed, the information from those works was put into context

This literature review will assess the value of magnetic resonance (MR) and computed tomography (CT) in detecting pathologies associated with vertigo. The common two types of vertigo peripheral and central vertigo are defined. Previous studies suggest that both MR and CT are good options for detecting semicircular dehiscence syndrome related peripheral vertigo. Although the accuracy rates of MR and CT are comparable in detecting acute intracranial hemorrhage, MR has an overall higher diagnostic yield

Physical Exam: General Appearance: Alert and oriented x3. Calm and cooperative. Mood and affect normal. Pleasant, well-dressed. Thin stature. Leaning forward in a sitting position. Labored breathing. No acute distress noted. Vital signs: heart rate 100 beat/minutes; respiratory rate, 38 breaths/ minutes and labored; Oral Temperature 99. 2 F; B/P 140/90mm Hg; O2 sat 86; Weight 120 lbs.; Height 5’ 11’’; BMI 16.7 HEAD: NCAT HEENT: Head is normocephalic, no scalp, or facial tenderness. Eyes: Conjunctivae

Multiple System Atrophy (MSA) is an adult onset progressive neurodegenerative disease, characterized by various degrees of Parkinsonism, cerebellar ataxia, and failure of the autonomic nervous system. MSA is classified as a α-synucleinopathy, a subset of neurodegenerative diseases characterized by the accumulation of misfolded α-synuclein (α-Syn) in the CNS (Fellner et al., 2011; MartÌ et al., 2003). In MSA, α-syn aggregates appear primarily as glial cytoplasmic inclusions (GCIs) in oligodendrocytes

Chief Complaint Abdominal pain. History Patient is a 63-year-old right-hand white male who states that since April when he was in rehab for his left total knee replacement he has been having pain along his abdomen. He describes this as a band of numbness and dysesthesia. He outlines a strip involving T9-T11. He states this is mostly on the right. It does extend past the midline a little to the left, but is not significantly disturbing on the left. He gets spontaneous stabbing pain, tingling