Cholecystitis

A 1.7 cm gallstone in the gallbladder. Negative for CT evidence of acute cholecystitis. Unenhanced liver, spleen, and adrenal glands are unremarkable. Pancreatic duct is dilated in region of the head and neck of the pancreas measuring up to 7 mm, nonspecific. Negative for discreet mass on this noncontrast exam. Negative for parapancreatic inflammation. Unenhanced pancreatic parenchyma otherwise appears unremarkable. Distal ureters are somewhat difficult to trace. The radiopaque

Differential Diagnosis Depending on the location and duration of the abdominal pain one can decide differential diagnosis. Right upper quadrant pain (RUQ) could be from: acute cholecystitis and biliary colic, acute hepatitis, hepatic abscess, hepatomegaly due to congestive heart failure, perforated duodenal ulcer, and bilateral pain: acute pancreatitis, herpes zoster, and myocardial ischemia, pleural or pulmonary problems (pneumonia). While right lower quadrant pain (RLQ) could be from: appendicitis

the outside of the colon by thin strands of tissue (stalks). This condition causes sudden lower abdominal pain. EA is rare, and usually goes away on its own. It can resemble other abdominal conditions, such as appendicitis, a gallbladder attack (cholecystitis), or diverticulitis. What are the causes? This condition may be caused by: Blocked blood flow to the epipoloic appendages. Sometimes this happens when the appendages twist on their stalks (torsion). A condition that causes swelling and inflammation

is even greater when the international normalized ratio (INR) is greater than 4.5 (4). Coinciding with the increased utilization of anticoagulant medications is the increasing incidence of emergency general surgery (EGS) disease such as acute cholecystitis, peptic ulcer

Truncal neuralgia in diabetic patients: An ignored cause of abdominal pain Mohamed Lotfy, Hazem N. Ashri, Mostafa M. Khairy ABSTRACT Aims: We aim to study diabetic patients, with abdominal pain and neuralgia somewhere else in their bodies, and search for the relation between their abdominal pain and the lower intercostal neuralgia (T7–11 and subcostal). Methods: Between July 2016 and February 2017 this study was carried out in emergency department, Zagazig University Hospital. 23 patients were subjected

this principle was introduced by the defence to illustrate that they were not liable for the exacerbation of the plaintiff’s condition. The plaintiff attempted to claim for damages for the following conditions; diabetes type 2, progression of cholecystitis, fractured skull and traumatic brain injury. By applying contributory negligence to the case, the liability of the conditions was entirely placed on the plaintiff due to discharging himself from three hospitals within the district against medical

Kiviluoto et al. [34] randomized 63 patients with acute cholecystitis and similar demographic, physical and clinical characteristic to receive either LC or OC. They found a significantly lower postoperative complication rate, shorter postoperative hospital stay and lower mean length of stay in the LC group. El-Awadi

Cholelithiasis, are the reaming’s of the digestive fluid bile, which form within the gallbladder. They vary in size and shape from as small as a grain of sand to as large as a golf ball.[1] Cholelithiasis occur when there is an imbalance in the chemical constituents of bile that result in precipitation of one or more of the components. Gallstone disease is often thought to be a major affliction in modern society.[2] However, cholelithiasis must have been known to humans for many years, since they

John is a forty-five year old male who presented in the emergency room experiencing abdominal pain in the right lower quadrant of the ventral cavity. The pain is felt in the umbilical region, right iliac region, and right lumbar region. He is also experiencing pale skin and fatigue. John has a previous history of gastritis(inflammation of the stomach), gastroesophageal reflux disease(stomach acids coming into esophagus), and bradycardia (abnormally slow heart rate). After the laboratory drew blood

institution with abdominal pain and concern for preterm labor. The patient had undergone a laparoscopic cholecystectomy for chronic cholecystitis ten days prior at another institution, which is where she presented for a prenatal visit. In the transfer record, patient had multiple recent admissions due to emesis, attributed to hyperemesis gravidum and cholecystitis. On admission to our institution, she complained of uncomfortable cramping abdominal pain with concern for preterm labor. She was febrile