Cirrhosis

Papaya Seeds as a Treatment for Liver Cirrhosis Liver cirrhosis is a disease, usually caused by excessive alcohol consumption over many years, wherein the liver shrinks and becomes hardened. In this state it is ineffective at removing toxins from the body, leading to a variety of serious health problems. Papaya seeds are often reported as an effective treatment for liver cirrhosis. One method is to grind up around five dried seeds in a pepper grinder, or crush up fresh ones in a mortar and pestle

Cirrhosis: An Analysis of Evidence-Based Practice Characterized by progressive tissue injury and scarring, cirrhosis is among the leading causes of chronic liver failure in the United States. Recent research conducted by the National Institutes of Health indicated that the prevalence of cirrhosis, especially in clients diagnosed with Hepatitis C, has increased steadily throughout the past decade and will likely continue to increase in the future (Kanwal et al., 2011). Caused by recurrent tissue

Portal vein thrombosis in patients with liver cirrhosis: insights to risk factors, clinical presentation and outcome Afifi F. Afifi 1, Osama M. Basha 1 , Fady M. Wadea 1, Abdelaziz E. Samack 2, Raghda Abd-elatif Hafez 3 1- GIT and hepatology unit, Internal Medicine department, Faculty of Medicine, Zagazig University. 2- Radiology department, Faculty of Medicine, Zagazig University. 3- Microbiology&Immunology department, Faculty of Medicine, Zagazig University. EGYPT Manuscript Info Abstract

Stages of Alcoholism Leading to Cirrhosis of the Liver Mikal Norvell Fortis Institute, Dr. Bassant Abstract This paper describes, briefly, the stages of the alcoholism as it slowly damages the liver, and finally results in permanent, irreversible damage called cirrhosis. Excessive alcohol consumption, or alcoholism is the number one cause of cirrhosis in the U.S. Though tolerance levels are different for each individual, daily consumption of more than ten alcoholic drinks over ten or more years

Thrombosis, risk factors, cirrhosis. *Corresponding Author Pawan Kumar Thakur Background and objectives: Portal vein thrombosis (PVT) is an increasingly recognized complication of liver cirrhosis. It is associated with worsening liver function, ascites and the occurrence of gastroesophageal variceal bleeding. The aim of this work was to clarify the risk factors, clinical presentation and complications of portal vein thrombosis in Egyptian patients with liver cirrhosis and to study the outcome with

Cirrhosis is a condition that results in development of fibrosis in liver, resulting in decrease in cellular mass of liver, and thus a decrease in function of liver. Various causes of cirrhosis are alcohol, chronic viral hepatitis (hepatitis B and hepatitis C), autoimmune hepatitis, NASH, biliary cirrhosis, and various inherited metabolic liver disorders. Cirrhotic liver has reduced ability to store and release glycogen. Glycogen is used to provide energy when we need it. So, in the absence of glycogen

another acid. Cirrhosis involves a complication in the liver which cause the loss of liver cells and irreversible scarring of the liver. Some of the common causes of the Cirrhosis is the abuse of alcohol fatty liver disease, viral hepatitis B and C. Other possible causes that cause Cirrhosis are Iron buildup in the body, Cystic fibrosis, Copper accumulated in the liver, poorly formed bile ducts, inherited disorders of sugar metabolism and Infection such as Schistosomiasis. Cirrhosis does not show

to increase with age 9, but as the mean age of the patients was 45.38±12.1 years in this study, this was not considered to significantly influence the results. Alcoholism was the commonest cause of cirrhosis 51 (72.85%), with incidence of chronic hepatitis B being 5(7.14%). 13 patients had cirrhosis of unknown etiology. This finding was different from that of Maric et al 10 where 43% had chronic hepatitis B infection

for cryptogenic cirrhosis or NASH-cirrhosis ranges from 25%-100%, while NASH development is observed in 10%-37.5% of these cases without leading to early allograft failure. The ongoing discussions of the limits of alcohol intake in “nonalcoholic” fatty liver revolve around the complex interactions of host the genetic and metabolic factors. The type of alcohol utilized, patterns of drinking, and the potentially beneficial interactions of modest alcohol

Egyptian patients with chronic hepatitis C mono infection (67%, males and 33% females); their mean age (± SD) was 40.29 years (± 10.98). Significant fibrosis (≥ F2) was present in 62 patients, advanced fibrosis (≥ F3) was present in 24 patients and cirrhosis (F4) was present in 11 patients, other general characteristics of the study population are summarized in table 1. The mean values ± SD of Hepa-Index parameters were: Platelets count (201.58 ± 59.02 109/L), α2 MG (2.57 ± 0.54 g/L), total bilirubin