Cystic duct

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    Patients and methods The current study completed at the General Surgery department, Banha University Hospital in Egypt and King Saud Hospital in Saudi Arabia since January 2015 till May 2017. The present study includes 100 patients with acute calculous cholecystitis. After approval of the study protocol by the Ethical Committee and obtaining fully informed written patients' consent for the participation in the study. Patients hospitalized through the emergency unit to the General Surgery department

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    Cholelithiasis, are the reaming’s of the digestive fluid bile, which form within the gallbladder. They vary in size and shape from as small as a grain of sand to as large as a golf ball.[1] Cholelithiasis occur when there is an imbalance in the chemical constituents of bile that result in precipitation of one or more of the components. Gallstone disease is often thought to be a major affliction in modern society.[2] However, cholelithiasis must have been known to humans for many years, since they

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    The ultrasound will look for the presence of gallstones, inflammation of the liver, blockage of the bile duct, thickened gallbladder walls or an oversized bile duct tract. An ultrasound can detect gallstones that are two millimeters or larger in size. Treatment of Gallstones in Cats No Treatment Small gallstones that aren't causing symptoms or digestive problems may not need any treatment

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    Introduction: Intraductal papillary mucinous duct neoplasms of the pancreas (IPMN-p) are one of the subtypes of cystic neoplasms of the pancreas. They are neoplasms that arise in the pancreatic ducts and can be visualized as they are usually greater than 0.5 inch in length. They are comprised of tall mucin-producing columnar cells, that have the capability of transforming into pancreatic cancer if left untreated. Their ability to develop into pancreatic cancer is one of the main reasons behind their

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    cell membrane. Then a second ATP is bound NBD2 and hydrolyzed charging the closure of the channel (Gadsby & Nairn, 1999). Lastly, at the R-domain, the phosphatases protein returns the channel to its inactive state (Sheppard & Welsh, 1999). The Cystic Fibrosis Foundation [CFF] (2010) states the 1400 CFTR gene mutations are classified into 6 groups Class 1: The mutation occurring in around 10% of patients interferes with the protein synthesis, there is a premature stop codon so translation of mRNA

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    Cystic fibrosis, also known as mucoviscidosis, is a genetic disorder that affects mostly the lungs but also the pancreas, liver, kidneys, and intestines. Some symptoms are difficulty breathing and coughing up mucus as a result of frequent lung infections. Other signs and symptoms include sinus infections, poor growth, fatty stool, clubbing of the finger and toes, and infertility in males among others. Different people may have different stages of the symptoms. In the lungs the mucus stops the air

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    Cystic Fibrosis Cystic Fibrosis or CF is a life threatening genetic disorder that mainly affects the lungs and digestive system. Cystic Fibrosis is most common among the Caucasian population, particularly people of northwestern European descent and is less common in people of African ancestry and is very rare in people of Asian ancestry. According to the Cystic Fibrosis Foundation, “an estimated 30,000 children and adults in the United States (70,000 worldwide) have CF” (Cystic Fibrosis Foundation

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    Cystic fibrosis is an autosomal recessive disorder located on the seventh chromosome. It is a commonly inherited disorder and is seen in children. It is most commonly seen 1 in every 3419 live births to parents who are from white, European descent (VanMeter, Hubert, 2014). Cystic fibrosis is an obstructive lung disease. Thick, sticky mucous obstructs airflow in the bronchioles and small bronchi, causing air to be trapped leading to atelectasis and damage to the bronchiole walls (VanMeter, Hubert

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    Cystic fibrosis is a autosomal recessive inherited disease that affects many organ systems. Over time the outcome for patients with the disease has improved drastically. The life expectancy for patients has increased to 37 years old, compared to 31 years old (O’ Sullivan, 2009). Researchers have reviewed the etiology, pathogenesis and clinical manifestations for cystic fibrosis. The cause of Cystic Fibrosis is mutations in a single gene on chromosome 7 that encodes for the cystic fibrosis transmembrane

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    Cystic fibrosis is life threatening, progressive and genetic disorders that cause severe damage to the lungs and digestive systems. About 30,000 children’s and adults in the United States are affected by this chronic disease. Cystic fibrosis is an inherited condition which affects the body’s exocrine glands (mucus secretion glands). The secreted fluids such as mucus, sweat and digestive juices are normally thin and slippery. The secretions become thick and sticky in those people who are affected

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