Deaths from myocardial infarction

disease is a global health problem causing nearly 1 in 3 deaths every year. Many of these deaths are due to coronary heart disease. Myocardial infarction (MI) is a major manifestation of coronary heart disease. In 1959, the World Health Organization defined MI as a combination of 2 of 3 characteristics: typical symptoms, enzyme rise, and a typical electrocardiographic (Patil, 2011). In this study, the age of the studied patients ranged from 15-55 years with female predominance. All cases were accidentally

occlusion of blood flow. When the plague crack or tear itself, blood platelets stick to this and form blood clots which can result in complete blockage of the blood flow. Other factors such as coronary artery spasm, platelet aggregation and emboli from mural thrombi (thrombi lining the walls of the cardiac chambers) can also lead to MI (Riedl, 2016). Risk Factors: The risk factors include age, male sex, family history and ethnic background, smoking, abnormal lipids, hypertension, diabetes, lack

Angina pectoris is a term used to describe the syndrome of chest pain resulting from myocardial ischemia (Griffin et al., 2008). Normal non-ischemic myocardial tissues differ from the cardiac tissue of an individual with myocardial ischemia because the normal tissue has adequate blood supply whereas the tissue in angina has inadequate blood supply from blocked coronary arteries. Unstable angina is diagnosed when ischemia is neither severe nor prolongs for more than 20 minutes and regularly occurs

(2012) states that an acute myocardial infarction (AMI) is when blood flow to a portion of cardiac muscle is obstructed, which causes prolonged tissue ischaemia and irreversible hypoxaemic cell necrosis and tissue damage. It is the necrosis of myocardial cells caused by a coronary occlusion which is usually due to the rupture of a complicated atherosclerotic lesion (LeMone, Burke, & Bostick, 2012). Farrell & Dempsey (2014) suggests that the main cause of myocardial infarction (MI) is the underlying coronary

A Myocardial Infarction also known as a “heart attack” or acute coronary syndrome is a medical emergency that requires immediate intervention to prevent extenuating damage or death to the muscle that pumps the human body with oxygen therefore, life. Many of those who’ve suffered damage from a heart attack and survived are able to go back to enjoying a normal life if damage is limited with proper treatment and preventive lifestyle changes. This paper will encompass the pathophysiology that is a Myocardial

Cardiovascular Disease with Vitamin D Deficiency Introduction Vitamin D is a fat-soluble vitamin. the primarily source of vitamin d is exposure to sunlight, and we can find the vitamin D naturally . Vitamin D2 is generally produced in humans from 7- dehydrocholecalciferol upon exposure to ultraviolet light. in addition vitamin D2 and D3 can metabolized by human body (Zhang & Naughton, 2008). However, highly prevalent of deficiency of vitamin D in the orarld spicially in United State. Vitamin

therapy has proven to be beneficial in reducing the risk of recurrent cardiovascular events in patients who have suffered a previous myocardial infarction (MI).1,5 However, it is unclear whether one statin is more effective versus another in reducing future incidence of acute MI and at what intensity is it beneficial.1,2 Myocardial infarction or acute myocardial infarction (AMI), also known as a heart attack, is due to a blood clot that occludes coronary arteries, which brings oxygen-rich blood to the

Primary percutaneous coronary intervention (PCI) and PCI with fibrinolysis are current therapy options used for patients who have had an acute ST elevation myocardial infarction (STEMI). These six article discuss multiple elements involved in the discussion comparing the many factors that affect which forms of therapy is preferred to which patients. Concerns regarding the safety and effectiveness of primary PCI have risen. Factors include the optimal time for therapy, the important of hospital staff

Introduction A systematized and precise assessment is a vital tool for a clinician to identify not only patients who are in unpredictable life threatening situations but also to recognize the initial signs of patient’s deterioration. (Tough, 2004) In order to accomplish a systematic and detailed assessment, a sound clinical judgment and strong clinical decision should be made (Croskerry, 2009) This will aid in formulating a pertinent diagnosis, which is the key in devising a safe and effective

constriction of blood vessels and prevent adequate blood flow. Overweightness and sedentary lifestyle contributes to dyslipidaemia and altered fat metabolism which decrease HDL and increase BP (Heather & Buckley, 2015, P.598). Emotional and physical stress from the passing of his friend would increase the production of cortisol and promote dyslipidaemia. Excessive amount of LDL promotes atherosclerosis as the deposit of lipid forms plaque. (Heather & Buckley, 2015, p. 597). 3. 1. John has excessive LDL and