Left ventricular hypertrophy

Left ventricular hypertrophy is defined as the increase in muscle mass of the left ventricle; It can be hypertrophy of the muscle leading to a systolic HVG or a dilation of the muscle leading to diastolic HVG. The diagnosis is suspected on the ECG and confirmed by ultrasound. Among the causes of left ventricular hypertrophy are age, intensive practice of one or more sports, obesity, prolonged hypertension. The seriousness of hypertension arises from its impact on a number of organs (target organs)

Left atrial volume index is the strongest predictor of development of persistent atrial fibrillation in obese non-hypertensives. Introduction: The global nature of the obesity epidemic was formally recognized by a World Health Organization consultation in 1997(1). Prior epidemiologic studies have yielded conflicting results regarding whether obesity is a risk factor for Atrial fibrillation (AF), but these studies were potentially limited by short-term follow up and lack of echocardiography data(2)

using commercially available ELISA kit. Associatin between the NT-proBNP concentration and various features of LVH including left ventricular wall thickness will be also be evaluated. We hypothesized that rhesus macaques with LVH show higher concentrations of NT-proBNP attributed to the hypertrophied ventricular walls with diastolic dysfunction, and the severity of left ventricular wall thickness is associated with the plasma NT-proBNP concentration. Material and Methods Subject and Housing All study

changes can include left ventricular hypertrophy with repolarisation changes with T wave inversion and deep Q waves. In family members carrying HCM gene mutations, the electrocardiogram may demonstrate only minor abnormalities. The presence of non-sustained ventricular tachycardia, a risk factor for sudden death, should be tested for by means of Holter monitoring (Maron et al., 2003). At present, the diagnosis of HCM relies on echocardiography revealing symmetric or asymmetric hypertrophy. Secondary causes

They can be differentiated into groups with different characteristics: dilated, hypertrophic, restrictive and arrythmyogenic right ventricular hypertrophies (report of the 1995 WHO). Hypertrophic cardiomyopathy (HCM), a familial condition affecting the heart, is characterised by unexplained and variable hypertrophy of the left ventricular wall and the inter-ventricular septum in the absence of any alternate causes such as aortic stenosis or hypertension (Oxford Concise Medical Dictionary, Kumar and

Cardiac Hypertrophy/ “Athlete’s Heart” The body’s ability to adapt to changing conditions is a constant endeavor. Maintaining during intense change causes the body to adjust rapidly. The cardiovascular system’s ability to adapt during exercise has numerous adjustments to counteract the body’s newly acquired needs. The heart is capable of altering many functions such as stroke volume, heart rate, and cardiac output. In addition the heart can also undergo hypertrophy and increase in size. Cardiac

known as a silent killer and if left untreated can cause serious damage to the heart. What causes high blood can differ from person to person. What causes this for some may be different with another. Genetics, diet, family history, weight, and use of salt are just some of the causes of high blood pressure. Some of the ways that high blood pressure damages the heart is through narrowing of the arteries in the heart, called arteriosclerosis, left ventricular hypertrophy, and restricted blood flow that

blood from aorta back to the left ventricle. HEMODYNAMICS: The backward leak of blood from the aorta to left ventricle during diastole increases left ventricular volume. The left ventricle accommodates extra volume of blood by increasing ventricular size. This regurgitation leads to impaired forward systemic blood flow reducing cardiac output. Left ventricle increases ejection during early part of systole to compensate this. In increased regurgitation, left ventricular pressure increases, which may

Left ventricular mass In humans there is a relation between left ventricular mass and cardiovascular mortality and morbidity independent of the blood pressure.5,6,7 In normal blood pressure subjects left ventricu-lar mass and diastolic filling have been found to be positively correlated with urinary sodium excretion8,9 and in two other normal BP groups followed up for 3 to 8 years the initial left ventricular mass and wall thickness were significantly related to the subsequent development of hypertension

defined as the heart’s inability to pump an ample supply of blood ultimately disrupting major bodily function. When there is heart failure occurring on the left side both the preceding and following regions of the heart will be affected. The left side of the heart is responsible for pumping blood systemically via the left ventricle. When the left ventricle fails this result in in inadequate amount of blood pumped systemically; in addition, since cardiac output has decreased, the blood will back up