Myelin

mechanism after spinal cord injury. There was no considerable disparity in the 10-Gy and the 20-Gy group from this experiment. In our results myelin showed first a decrease, and then an increase following spinal cord injury in rats. This outcome indicated that the spinal cord injury and repair coexisted, by being injured first and then repairing it. In addition, the myelin count significantly decreased in the irradiation group at week 14 after spinal cord injury, which showed that ionizing radiation also

Multiple Sclerosis (MS) is a lifelong disorder of the central nervous system: that consist of the brain and spinal cord. MS is known as an autoimmune disease in which the immune system mistakenly attacks myelin sheath. The myelin sheath is the cover that protects the nerve cells. When the myelin is damaged, messages between the brain and the whole body get disrupt. MS is more probable to affect people between the ages of 20 to 40 years. The effects of the disease vary for everybody who suffers from

Pathophysiology Multiple sclerosis is a disease where your immune system attacks the protective barrier around your nerves, called myelin sheaths (Mayo Clinic Staff, 2014). Your immune system falsely recognizes these sheaths as being intruders in the body causing it to attack them leading to nerve damage (this is called an autoimmune disease). With this nerve damage, there is disruption of the signals being sent from your brain and travelling to the rest of the body impairing movements, causing pain

targeting has not yet been determined. Multiple Sclerosis mainly affects the brain and spinal cord, or the Central Nervous System. Within the Central Nervous System, or CNS, the structures mostly affected by this disease would be the myelin and the nerves it protects. If the myelin is damaged, then scar tissue will form.

animal demyelination model of this kind is the experimental allergic encephalomyelitis (EAE) model. EAE can be induced in a number of species, including mice, rats, pigs, rabbits and primates. Commonly used antigens include purified myelin protein such as MBP, PLP, and myelin/oligodendrocyte glycoprotein (MOG), or peptides of these proteins and spinal cord homogenate (SCH), all of them can serve as distinct models with different disease characteristics of immunological response and pathology (Robinson

the muscles to elicit movement. There are more than 100 types of peripheral neuropathy and each is classified based on the type of damage on the nerve. Some peripheral neuropathies are due to damage to the myelin sheath which is the fatty protein that coats and insulate the axon. This myelin sheath is important for increasing the speed at which impulses propagate down a myelinated nerve fiber.

Multiple Sclerosis called by MS is a disease that which the fatty myelin sheaths around the axons of the brain and spinal cord which make up the Central Nervous System (CNS) are damaged, leading to demyelination and scarring as well as a broad spectrum of signs and symptoms and it is also referred as Insular Sclerosis or Disseminated Sclerosis. The name "multiple sclerosis" is derived as multiple (many) since it occurs in many locations within the nervous system and sclerosis (scars) which means

from the cell body and send the messages to other neurons, muscles, or glands. Myers explains it simply, “dendrites listen, axons speak (2014, pp. 38). Axons are insulated by a layer of fatty tissue called a myelin sheath. Myelin sheaths help speed up neural impulses. Degeneration of myelin sheaths results in deceleration of muscle communication and eventual loss of muscle control; a disease called multiple sclerosis. In addition to neural cells, message transmission is assisted by glial cells. Glial

typically is diagnosed in the second or third decade of life. Normally, nerves are enclosed in myelin sheaths that help facilitate transmission of nerve impulses within the CNS and the peripheral nervous system throughout the body. In patients with MS, the myelin sheath is damaged and eventually degenerates, causing patches of scar tissue called plaques or lesions to occur anywhere randomly on the myelin sheath (Ruto, 2013). This results in impaired nerve conductivity, which interferes with message

this process the brain develops myelin which wraps itself around the axons. This increases the signal speed which lends to what we know as automaticity, “The state or quality of being spontaneous, involuntary, or self-regulating” (Farlax, n.p.) Over time one’s skill becomes a spontaneous act. This skill is what separates mediocrity from genius. Myelin is the trigger point. This is good news. This means that such talent is accessible to all. As Coyle notes, “We are myelin beings” (p. 72) Targeted practice