Prostate cancer

Abstract: Prostate cancer is the leading mortality cancer amongst males. This cancer is due to the signaling increase of androgen and androgen receptors, which is a gene that associates with the risk of prostate cancer. Over expression of androgen receptors occurs in androgen independent cancer. Prostate tumors advance to androgen-independent state where they progress in the absence of circulating testosterone. Typically, treatments for prostate cancer are surgery and radiation. Most effective

Prostate cancer and hormone treatment to address this disease is one approach some have taken. The idea with this type of treatment is to decrease the production of testosterone that could stimulate the cancerous prostatic cells. These cancerous prostatic cells cause the disease to worsen. And with its reaction to the male androgenic hormone testosterone the spread of these cancer cells will be hastened. Prostate cancer and hormone treatment for this disease, looks at this angle of testosterone production

Prostate Cancer The prostate gland is an egg-sized organ that rings the male urethra. The secretions of the prostate give nutrients to the semen. Prostate cancer is the most commonly diagnosed cancer and the second most common cause of cancer death in men. (Ernstoff,Heaney,Peschel,1998,pviii) Like all cancers, prostate cancer is an uncontrolled cell production in a particular organ or area of the body. In the case of prostate cancer, these cells begin to split impulsively in the prostate and

One of the most common cancers to be diagnosed in men, prostate cancer has a good prognosis when diagnosed early. Approximately 2.8 million men in the United States have prostate cancer. Raising awareness for this type of cancer is key in being able to detect this cancer early and saving more lives. Definition Prostate cancer begins in the prostate, which is a walnut-shaped gland that is responsible for producing seminal fluid. Some types of prostate cancer grow slowly and may need very little

PSYCHOLOGICAL AND PSYCHOSEXUAL ADJUSTMENT OF PROSTATE CANCER Sexual dysfunction represents a prevalent long-term complication among prostate cancer survivors with a wide spectrum of manifestations and a huge impact on quality of life [4]. Resnick et al (2013) reported long-term effects of sexual function after radical prostate cancer treatment reported by prostate cancer survivors. As shown in a 15-yr follow-up study consisted of 1655 men who had localized prostate cancer the decline in erectile function following

A Literature Review of the Journal Club Article Entitled “Facilitating Shared Decision Making About Prostate Cancer Screening Among African American Men” According to the ACS Statistics of cancer incidence rates among US males for 2003-2007, prostate cancer occurs more commonly in African American men than in white males (ACS, 2011-2012). Therefore, it is not only essential, but also vital to increase knowledge on the subject matter, raise general awareness, and promote shared decision making about

Prostate cancer is the one of the most lethal disease in the United Sates. The pattern of disease recurrence being the major cause of morbidity and mortality. In spite of recent advances in our understanding of the molecular mechanisms responsible for the development of prostate cancer, the survival rate of men with this disease has remained relatively unchanged in over the decades. Since the Nobel prize winning discovery by Dr. Huggins and Dr. Hodges (Huggins C., 1941) androgen deprivation therapy

Prostate cancer is now the most common cancer in men in the UK and the second highest cause of male cancer death. Yet despite its high prevalence it is also highly survivable, with figures from Cancer Research UK showing that 84 per cent of men diagnosed with the disease survive ten years or more following treatment. With such a high survival rate would be easy to assume that the needs of this patient group are being adequately met. But this isn’t the case, says specialist prostate cancer physiotherapist

Interest Prostate cancer being a huge cause of mortality and medical expense in men age forty and above has only recently become a topic of general conversation to men in America (Plowden, 2009). African-American men are affected by prostate cancer at a disproportional level than all other men. They are diagnosed up to 65% more frequently and the mortality rate is twice that of Caucasian counterparts (Emerson, 2009). The African-American male is also less likely to take advantage of free prostate cancer

They found that the solution lied in the saturation, where the maximal growth of the prostate cancer was achieved at a low level of testosterone. This model was produced by Fowler and Whitmore, who concluded “normal endogenous testosterone levels may be sufficient to cause near maximal stimulation of prostatic tumors.” There final conclusion was that “there is not today—nor has there ever been—a scientific basis for the contention that a higher T concentration causes pCA growth, acutely or long-term