Ventricular hypertrophy

Sort By:
Page 8 of 36 - About 358 essays
  • Good Essays

    (HHR) and a normal heart rat (NHR). The HHR model showed no cardiac hypertrophy at maturity. Tissue from both HHR and NHR had been extracted. The cardiac weight index (CWI) was collected. The cardiomyocyte were then obtained via enzymatic procedure from both neonatal and adult NHR na HHR. Nuclei are then stained. Polymerase chain reaction (PCR) were used to search mRNA expression levels of ANP, BNP,MHC,p53, bax-1 and bcl-2 in ventricular tissues of HHR and NHR. Pl3K assay and western blot analysis were

    • 1078 Words
    • 5 Pages
    Good Essays
  • Decent Essays

    There no direct cause as why to atrial fibrillation occur but, there is many other disease and disorders that Atrial Fibrillation is associated with. For example Atrial Fibrillation is closely associated with heart attack due to the fact that a during a heart attack the coronary artery is unable to supply oxygen to the heart which then causes the muscles in the heart to starve for oxygen. This can then lead to the death of some muscle tissue in the heart which then can lead to AFib. Hypertension

    • 407 Words
    • 2 Pages
    Decent Essays
  • Decent Essays

    Due to the changes in resistance, the right ventricular afterload is diminished, and the disproportion of the heart chambers resolves.2 Sonographic Findings Detection of ductus arteriosus constriction is achieved through 2-dimensional imaging and the interrogation of Doppler velocities through the ductus

    • 755 Words
    • 4 Pages
    Decent Essays
  • Good Essays

    Diabetic Cardiomyopathy

    • 997 Words
    • 4 Pages

    but the second stage of DCM is characterized by diastolic dysfunction (including: ventricular hypertrophy and stiffness, high end-diastolic pressure and volume) (Connelly et al. 2008;van et al. 2008) and cardiac inter-cellular matrix remodeling (including: an accumulation of insoluble type VI collagen and a deposition of fibrous tissue) (Khaidar et al. 1994;Spiro et al. 1993). Several mechanisms contribute to ventricular stiffness in diabetic heart mainly due to sustained hyperglycemia that promotes formation

    • 997 Words
    • 4 Pages
    Good Essays
  • Decent Essays

    to the upturning of the cardiac apex because the right ventricle hypertrophy and the concavity of the main pulmonary artery. There are four components of a TET heart defect. Pulmonary artery stenosis is a narrowing that happens in the pulmonary artery. This can occur in the main pulmonary artery and in the left or right pulmonary artery branches. This can make it hard for the blood to get to the lungs to pick up oxygen. Ventricular septal defect is a hole in the heart. The hole is in the lower

    • 300 Words
    • 2 Pages
    Decent Essays
  • Decent Essays

    Ventricular septal defect (VSD) Definition A ventricular septal defect (VSD) is one of the most common congenital heart defects. During fetus development, a gap occurs in the heart’s lower wall between two ventricular chambers and allows blood to pass through from the left to the right side of the heart. The blood rich with oxygen, pumped back to the lungs instead of out to the body and cause the heart to work harder than usual. Pathophysiology Ventricular septal defect cause the blood flow move

    • 969 Words
    • 4 Pages
    Decent Essays
  • Decent Essays

    Doppler Echocardiography

    • 519 Words
    • 3 Pages

    lateral chest x ray shows filling of the retrosternal air space due to right ventricular enlargement.Prominent pulmonary arteries also may be here.9 In 95% of patients with COPD and pulmonary hypertention the diameter of the descending branch of right pulmonary artery is greater than 20mm in width.Oligaemic lung fields are caused by peripheral vessels attenuation. Electrocardiogram(ECG) Right ventricular hypertrophy,right axis

    • 519 Words
    • 3 Pages
    Decent Essays
  • Decent Essays

    Long-term hypertension can result in a variety of consequences. These consequences are the result of the heart having to adapt and work harder, i.e. against an increased afterload due to the increased systolic pressure. The heart adapts via hypertrophy of the smooth muscle. Chronic hypertension can also lead to a disruption of the endothelium, thus increasing the

    • 1437 Words
    • 6 Pages
    Decent Essays
  • Decent Essays

    Bipa Case Study Essay

    • 418 Words
    • 2 Pages

    of the ascending aorta and aortic arch. Aortography confirmed the diagnosis of severe calcified juxtaductal CoA. There was a 70 mmHg peak to peak gradient across the coarctation, 90 mmHg gradient across the aortic valve and the mean left ventricular pressure was 290/26 mmHg. Coronary evaluation demonstrated a significant proximal LAD lesion with a fractional flow reserve (FFR) value of 0.78. A significant dilatation of the left internal mammary artery was also noted. The presence of high

    • 418 Words
    • 2 Pages
    Decent Essays
  • Decent Essays

    Introduction: Heart failure is a progressive clinical syndrome characterized by impairment of filling pressures or blood ejection, neurohormonal system activation and molecular derangements. Typical signs, symptoms and other investigative studies like echocardiography, cardiac MRI, exercise testing and biomarkers diagnose heart failure in children (Rossano & Shaddy 2014; Hsu & Pearson 2009b). Essential part of the evaluation includes exhausting effort to determine the etiology of the heart failure

    • 781 Words
    • 4 Pages
    Decent Essays