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- The product of the human papillomavirous oncogene E7promotes cancer by emhancing expression of telomere inhibiting DNA damage repair pathways binding to Rb protein and preventing itd function ubiquitinating the tumor suppressor p53Is the following true or false AND EXPLAIN: Cells with one functional copy of a proto-oncogene will usually proliferate faster than normal cells.Which of the following describes the role typical proto-oncogenes have when they are expressed in cells that are not cancerous
- Mutations in proto-oncogenes that turn them into oncogenes tend to be dominant, while cancer-causing mutations in tumor suppressor genes tend to be recessive. Please explain why.Which of the following could be classified as an oncogene? None are possible oncogenes A mutant of MAP kinase that was active with or without being phosphorylated All are possible oncogenes A mutant of MEK with significantly reduced enzyme activity A mutant cAMP phosphodiesterase that made it super-activeThe palladin gene, which plays a role in pancreatic cancer (see theintroduction to this chapter), is said to be an oncogene. Which of itscharacteristics suggest that it is an oncogene rather than a tumorsuppressorgene?
- Mutations in the ras gene family induce normal cells to proceed into the replication cycle. This converts the ras gene from a ________ gene to a ________ gene. a. proto-oncogene; oncogene b. oncogene; proto-oncogene c. mutant; oncogene d. tumor suppressor; proto-oncogenedescribe the relationship between a proto-oncogene and an oncogene, and explain how one arises from the other. Explain how a mutation in the EGF receptor, or in a GTP- binding protein, can lead to unregulated cell division.A normally functioning gene that regulates the cell cycle by stopping cell division is most likely to be a(n) __________. Group of answer choices carcino-gene tumor suppressor proto-oncogene muta-gene oncogene
- Explain in general what is meant by a proto-oncogene and how they are involved in the formation of a tumorPlease answer all questions if possible. -B-Raf is not overexpressed in cancer though shows altered structure, why? -What is the specific structural change in B-Raf that causes oncogenic activation of this proto oncogene.BRCA1 and BRCA2 are genes that encode proteins involved in DNA repair. If DNA can not be repaired, BRCA1 and BRCA2 activate a cell cycle checkpoint. Are BRCA1 and BRCA2 proto-oncogenes or tumor suppressor genes. Please explain why