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A Study On Anterograde Amnesia

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Anterograde amnesia, as described earlier, is an inability to form lasting memories after some disturbance to the brain, such as a brain injury or degenerative brain disease. The revelation of the H.M. case was the identification of the medial temporal lobes as memory consolidation and storage centers. Within the medial temporal lobes, the hippocampus has been identified specifically as a brain area involved in learning new information (Gluck, 2014). To put it another way, people with damage to the hippocampus will have difficulty forming new memories because they won’t be able to learn new things. The hippocampus helps to integrate objects in a spatial and temporal context. Episodic and semantic memory forming is impaired, otherwise known as declarative memory, though the person can remember declarative memory from the past. The process of encoding, storage consolidation, and retrieval seems to go through the medial temporal lobe with the hippocampus playing a key role. In anterograde amnesia, the consolidation process is damaged. Thus, in the short term new information can be learned, but since it is not consolidated, it can’t be retrieved, so it is forgotten (Webbe slides). Additionally, damage to the basal forebrain can cause anterograde amnesia. This is because the basal forebrain sends neurotransmitters to the hippocampus to tell it whether and when to process and store information (Gluck, 2014). In this way, the hippocampus is the main brain structure involved so

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