Athletes With Abnormal Av Conduction Characterized By An Hv Interval

Supraventricular tachycardia or SVT is a heart condition where the sufferer’s heart beats very fast for reasons such as exercise, high fever, caffeine or stress. For the majority of people who have supraventricular tachycardia, the heart still works as normal where it pumps blood across the body. During an episode of supraventricular tachycardia, the heart's electrical system doesn't work properly, causing the heart to beat very fast. The heart will beat at least 100 beats per minute and may reach up to 300 beats per minute. After medical treatment or treatment of its own, the heart will usually return to a normal rate of around 60 to 100 beats per minute. Supraventricular Tachycardia may start suddenly and then end suddenly, and you may not

Should student get screen for heart disease before athletics? Sadly In today’s society, student athletes are dying of heart attacks, at an early age. Which is why student athletes should be required to get screened for heart disease. When the individual gets a screening, they should take both popular diagnostic tests, such as the electrogram (EKG) and the echocardiography (ECHOS). Sudden cardiac arrest (SCA) is the leading cause of death in young athletes (Drezner at al., 2007). SCA in young athletes is not only a concern for the medical community, but also for the community’s at large. SCA occurs when electrical impulses in the heart become rapid or chaotic, which causes the heart to stop beating. Approximately 1 in 220,000 youthful student competitors experience sudden cardiac death (SCD) every year (baggish et al., 2010). Athletes are known to be some of the healthiest people in society, however SCD while being active in sports is odd, its manifestation is universally recorded in the media, caused by the age and health conditions of the athlete. The latest events in many parts of the world show that congestive heart failure of student athletes is still a reality and it keeps challenging experts in cardiology that take care of student athletes (Ferreira et al., 2010). It has come to mind that some easy pre-participation screening, adding a physical, electrocardiograms (ECG/EKG) additionally gathering

Ablation. During this procedure the heart tissue causing the problem is destroyed. This procedure may be done if atrial flutter lasts a long time or happens often.

SCA in athletes under the age of 35 years predominantly occurs when there is a malfunction of the normal electrical conduction in the

Fackelmann, Kathleen. "Flaws of the heart; sudden death in athletes is often caused by cardiac defects." Science News 3 Aug. 1996: 76+. Academic OneFile. Web. 27 Oct. 2015.

The patient has been diagnosed with atrial defibrillation and congestive heart failure. There is no visible jugular vein distention or pulsations on either side of the neck. The patient was not comfortable with removing her sweater, therefore, pulsations, lifts, or heaves were not seen if present. Palpated and auscultated the carotid arteries for vascular sounds, no bruits heard. Heart sounds were auscultated with the bell and diaphragm of the stethoscope. S1 82/min, even and regular. S2 85/min, even and regular. S3 82/min, even and regular. S4 83/even and regular. No murmurs were heard. The apical pulse rate was 92 bmp, regular and was accessed with patient in sitting position and between the 4th and 5th intercostal space. Patient stated

A website for the American Heart Association (AHA) and the American College of Cardiology Foundation showed guidelines as to how to treat all classifications of atrial fibrillation. This article mentioned recommendations for which procedure/treatment was appropriate for each classification of AF and if the benefit was greater than the risk. Another search was done and was geared towards certain medical websites such as the Journal of the American Medical Association (JAMA), and the New England Journal of Medicine (NEJM), which prompted a search for the topic that led to the discovery of a few articles that are presented in this paper. The last tool that was used was Long Island University’s library online database that is available to all students to help locate articles when working on a research paper. The databases that were primarily used were Medline: PubMed and Medline: Ebsco created by the National Library of Medicine and it was found to be quite helpful in finding copious articles relevant to the topic. In the search bar, keywords such as: “atrial fibrillation”, “catheter ablation”, and “paroxysmal”, were used which resulted in many articles. The filters were set to human subjects, the English language, full text articles, and articles published since 2005. Results that did not include atrial

My patient was in accelerated junctional rhythm or AV disassociation. My patient was not symptomatic so we did not have any concerns about her circulation. If her rhythm would to change we would follow protocols especially if she was symptomatic. We could administer 02 if she became symptomatic and we would notify the care service

Susan Arvin is a woman the suffers from tachycardia. Tachycardia is a heartbeat that's too fast, a heart rate of more than 100 beats per minute (BPM). Susan states that her heart will race for hours at a time. Disrupting cells cause her to have a rapid heartbeat. Having her heart beat faster than usual, it became a problem at home and at work.

The wide receiver catches the football and immediately drops to the ground, unconscious. This is exactly how unexpected sudden cardiac arrest is in adolescents. The reported incidence of pediatric sudden cardiac death ranges from 0.8 to 6.2 cases per 100,000 children in the United States every year (Berger, Kugler, Thomas, and Friedberg 1201). Approximately 20-25% of the deaths occur during sports (Gajewski and Saul 107). “Sudden cardiac arrest (SCA) is a condition in which the heart suddenly and unexpectedly stops beating. If this happens, blood stops flowing to the brain and other vital organs (“What is Sudden Cardiac Arsrest?”).” Sudden cardiac death, or SCD, usually occurs “within one hour of onset symptoms

They also must withdraw blood to check for any condition. Inappropriate sinus tachycardia is a condition that is presented with similar symptoms of Postural Orthostatic Tachycardia Syndrome (Busmer 19). It may be difficult for medical professionals to differentiate these two conditions. Some techniques have been prone to minimize the risk of falls which consist of avoiding standing for long periods of times and if possible to keep moving. ”Postural Orthostatic Tachycardia Syndrome is a life changing condition, often affecting healthy, young, fit and active people. It affects every aspect of their lives and consequently their physiological wellbeing” (Busmer 20). Some things that could happen because of Postural Orthostatic Tachycardia Syndrome are deconditioning because of poor tolerance of exercise. People who are diagnosed with Postural Orthostatic Tachycardia Syndrome need to go through a support of any kind such as practically and emotional. Some things are made to raise awareness through healthcare professionals. Research is also needed to understand Postural Orthostatic Tachycardia Syndrome and how to approach and manage or cure this

For many year’s patients with atrial fibrillation have been treated with anticoagulants such as Warfarin to prevent strokes and embolisms. Unfortunately, Warfarin must be closely monitored and that is an irritant for some patients. In October 2010, the FDA approved a new generational anticoagulant drug called Dabigatran (Pradaxa). This alternate medication gives patients the benefit of no dietary restrictions since dabigatran is not affected by certain foods. Another benefit of taking dabigatran is a monthly blood test is not required to measure its effectiveness, so for this particular reason many patients switch from taking other anticoagulants to dabigatran (Talati & White, 2011). Since this medication does not require close monitoring, some wonder if is it truly a better option or can more harm than good come from taking it. While the benefits of using dabigatran have shown significant improvement over warfarin, there are still risks associated with using dabigatran.

It is important to recognize ECG tracings, even if described and not visually seen. Much can be deciphered from this vignette to rule out the other answers. P waves are discernable (Choice A), the rate is > 120 beats/pmin (Choice B), and the QRS waves are normal—-meaning no evident delta waves (Choice E), and QRS waves are not wide (Choice D). For Step 2 purposes, knowing the diagnosis based on application of one’s basic knowledge to the clinical scenario would be enough. The next best step in this case would be synchronized cardioversion since he is tachycardic and hemodynamically unstable. If the QRS was > 0.12/second and the patient is still hemodynamically unstable, then one would consider an anti-arrhythmic antiarrhythmic drug; the choice depending on the actual morphology of the QRS (monomorphic vs. polymorphic). Based on the vignette’s ECG description: the tracing has distinguishing P waves, normal appearing QRS length and shape, and a heart rate greater than 120

In this particular (case or scenario) the cardiologist diagnosed T.G. with vasovagal syncope based on his history and physical. The pediatrician diagnosed a murmur and provided a list of differential diagnoses. The Cardiologist chose not to perform an EKG or do a long QT measurement. When reviewing syncopal algorithms one would have to question the cardiologists thought process of why these diagnostic components were missed when ruling out diffentials for T.G. Literature reviews suggest fainting is due to a sudden drop in heart rate and blood pressure.

The pacemaker is a complex device that sends electrical impulses to the heart to keep it in a normal rhythm. Most of the time doctors implant pacemakers to treat arrhythmias. “Arrhythmias are problems with the rate or rhythm of the heartbeat. During an arrhythmia, the heart can beat too fast, too slow, or with an irregular rhythm” (MedicineNet). Arrhythmia has two different stages and they're called: bradycardia and tachycardia. Tachycardia is the condition where the heart beats too fast and bradycardia is the condition where the heart beats too slow. The pacemaker then changes the defective rhythm