Day 5
Complains of not being able to have cigarette when he wants. Chest x-ray reveal a mass growing in lungs, confirmed as cancer. Chemotherapy is started immediately.
1. It’s beneficial to quit because it can help to delay the progression of the cancer. McDonald and Penola (2015. p.695) state that smokers demonstrate a steady decrease in pulmonary function. It was also highlighted that smokers who quit at the age of 45 have a slower progress towards COPD compared to individuals who quit at 65. However, for an individual who didn’t stop smoking at all, had the fastest progression to death.
2. Chemotherapy may destroy cancer cells but, it also effects the healthy cells such as the inner lining of the digestive system, bone marrow and the reproductive system. Therefore, any cells are prone to damage. Anorexia is associated with chemo treatments and can result in taste alterations (Stephens, 2015. p. 1086). Chemotherapy can also lead to early satiety
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Does not have a pulmonary embolism. Severe dyspnoea, RR 28 breaths per minute, SaO2 of 91% and diagnosed with COPD. Is constantly tired.
1. The main cause of chronic obstructive pulmonary disease (COPD) is smoking. Smoking is the main promoter of the diseases chronic bronchitis and emphysema. Effects chronic bronchitis as inhaled irritant results in airway inflammation with the of invasion macrophages, neutrophils and lymphocytes into the bronchial wall which produce excess mucus. Mucus cannot be cleared due to impaired ciliary function. Defence mechanism of the pulmonary system is impaired and is more prone to pulmonary infection and injury.
Emphysema is due to the alveolar septa being damaged. Thus, the air sacs of the lungs are damaged and enlarged. Enlarged spaces within the lung tissue aren’t effective in gas exchange. Gas exchange between air and blood is lost due to the loss of alveolar tissue meaning loss of respiratory membrane (McDonald & Penola, 2015, p.
D.Z.is a 65-year-old man admitted to medical ward with an exacerbation of chronic obstructive pulmonary disease (COPD; emphysema). Past medical history (PMH) indicates hypertension (HTN), well managed with enalapril (Vasotec) past six years, diagnosis (Dx) of pneumonia yearly for the past three years. D.Z. appears cachectic with difficulty breathing at rest. Patient reports productive cough with thick yellow-green sputum. He seems anxious and irritable during subjective data collection. He states, he has been a 2-pack-a-day smoker for 38 years. He complains of (c/o) insomnia and
There are many different type of cancers, there are also many different types of treatments. One of the most known types of treatments would be chemotherapy. Chemotherapy can be used for a wide range of different types of cancers and diseases, and each of the different types of cancers or diseases require a different group, and sometimes order, of chemicals to properly treat the cancer or disease. These chemicals include: Alkylating agents, Antimetabolites, Anthracyclines, Topoisomerase inhibitors, mitotic inhibitors, corticosteroids, and more. Each of these drugs previously listed have its own cancer type(s) or disease(s) that it can assist in treating. Some of these cancers include: Leukemia, Lymphoma, Hodgkin disease, multiple
Have you ever known a person who smokes and has a hard time doing every day activities, due to difficulty of breath, or constantly coughing. He or she may have Chronic Obstructive Pulmonary Disease, or COPD. COPD is a progressive and treatable lung disease that causes shortness of breath due to obstruction of air way (COPD, 2013). Progressive means that is gradually gets worse over time. It is a combination of chronic bronchitis and emphysema (Causes,2014). Chronic bronchitis is inflammation of the bronchioles, which causes mucus build up (Davis,2016). Emphysema is when the air sacs get enlarged (Smoking, 2016). Since the disease does not have a cure yet it is important to know pathology (path of disease), epidemiology (who is effected in a population), ethology (who is effected genetically), manifestation (symptoms), treatment, and outcome.
Chronic obstructive pulmonary disease (COPD) is in the top five principal cause of death in the U.S. The disease is an abnormal inflammatory reaction in the lungs with limited airflow. COPD characteristically arises around the age 35. Smoking continues to be the main source of COPD, but is not the only known root cause. In many studies, smoking explanations for at least three fourths of COPD cases ("Chronic obstructive pulmonary disease | University of Maryland Medical Center," n.d.). Stopping smoking has been known to improve lung capabilities and help to prevent death from COPD. Genetic conditions and introductions to airborne toxins, irritants and gasses are correspondingly involved in the growth of the illness. A complete treatment plan could comprise of lifestyle changes, one or more medications, patient education, oxygen therapy respiratory rehabilitation, and surgery ("Chronic obstructive pulmonary disease | University of Maryland Medical Center," n.d.).
COPD is almost always caused by smoking, or second hand smoke. The tobacco smoke irritates the airways and destroys the fibers in the lungs. Breathing in chemical fumes, dust, or air pollution over a long period of time may also cause it. It usually takes a long time for the lung damage to start causing symptoms, so COPD is most come in people who are older than 60.
It causes a decrease in lung functions, and very often, shows signs of breathlessness. It is indeed a destructive disease of the lung in which the alveoli (small sacs) that promote oxygen exchange between the air and the bloodstream are destroyed. Emphysema is a progressive, degenerative kind of disease that destroys many alveolar walls. As a result, clusters of small air sacs merge into larger chambers, which decrease the total surface area of the alveolar walls. At the same time, the alveolar walls lose their elasticity and the capillary networks associated with the alveoli diminish (Shier et al; 2010). It is in this sense that a person with emphysema finds it very difficult and increasingly hard to breath, has to force air out of the lungs because the tissue elasticity of the lungs and for that matter, the alveolar sacs have reduced or completely destroyed. Furthermore, abnormal muscular efforts are required to compensate for the lack of elastic recoil that normally contributes to
His lung sounds are diminished bilaterally. Pulse oximetry reading is 90%. He is receiving oxygen at 2 liters per nasal cannula. He complains of shortness of breath with exertion.
One of the biggest causes of COPD is a cigarettes smoking. Habitual smoking can inflame the linings of the airways in the lungs and can make the
Emphysema and Asthma are progressive diseases where the lung tissue is gradually destroyed and narrowing of the air passages obstructs oxygen uptake, causing shortness of breath. The lung damage caused by Emphysema typically occurs as a result of long-term inhalation of irritant gases and particles, such as tobacco smoke.
The World Health Organization (WHO) (2006A) defines COPD as a disease state characterized by airflow limitation that is not wholly reversible. The airflow limitation is usually both progressive and associated with abnormal inflammatory response of the lungs to noxious particles or gases. John's chronic bronchitis is defined, clinically, as the presence of a chronic productive cough for 3 months in each of 2 successive years, provided other causes of chronic cough have been ruled out. (Mannino, 2003). The British lung Foundation (BLF) (2005) announces that chronic bronchitis is the inflammation and eventual scarring of the lining of the bronchial tubes which is the explanation for John's dyspnea. The BLF (2005) believe that when the bronchi become inflamed less air is able to flow to and from the lungs and once the bronchial tubes have been irritated over a long period of time, excessive mucus is produced. This increased sputum results from an increase in the size and number of goblet cells (Jeffery, 2001) resulting in John's excessive mucus production. The lining of the bronchial tubes becomes thickened and an irritating cough develops, (Waugh & Grant 2004) which is an additional symptoms that john is experiencing.
Due to the inability of the lungs to exchange gases and air, those suffering from emphysema experience a chronic cough, difficulty breathing, and an increased risk of lung infections.
As air sacs are destroyed, the lungs are able to transfer less and less oxygen to the bloodstream, causing shortness of breath. The lungs also lose their elasticity, which is important to keep airways open. The patient experiences great difficulty exhaling. Emphysema doesn't develop suddenly, it comes on very gradually.
As mentioned above in the introduction, smoking is one of the biggest causes of death and illness, the figures almost speak for themselves. Every year around 100,000 people die from smoking, with many more deaths caused by smoking-related illnesses (NHS, 2014). Smoking increases your risk of developing more than 50 serious health conditions, 14 of those being cancers (Cancer Research UK, 2014). Some may be fatal and others can cause irreversible long-term damage to your health. These include diseases such as lung cancer, respiratory problems, COPD and if you have asthma it can trigger an attack or make it worse (NHS, 2014). You can become ill if you smoke yourself or through other people’s smoke, for example passive smoking or second hand smoke. For the purpose of this assignment however I shall be looking at COPD.
The effect smoking has on the respiratory system are irreversible. Smoking irritates and damages the respiratory tract. In the lungs an accumulation of tar from smoking cause irritation and damage. This causes a variety of symptoms, including wheezing, productive cough, sputum production and respiratory infections, such as bronchitis and pneumonia. These effects can be reduced but not entirely reversed by quitting. Smoking is the primary risk factor of developing COPD, i.e. chronic bronchitis, emphysema (American Thoracic
The good news is that stopping smoking has a very important bearing on reducing the risk of getting lung cancer. For instance, a man who has smoked throughout his life carries a 15.9% risk of dying from lung cancer by the time he is 75. For a man who stops smoking by the age of 50, this risk drops to 6% and is only 3% for someone who stops by the time they are 40.