Synthesis Of RTMS

Studying the brain has been an affective result in finding out the various neurochemicals that are involved with PTSD. Brain imaging systems nowadays focus on two brain structures, the amygdala and the hippocampus. The amygdala is involved with how we learn about our fear and hippocampus plays a role with our memory formation. Some research focuses on a hormonal system known as hypothalamic-pituitary

Post-Traumatic Stress Disorder can do a range of things to the brain. Post-Traumatic Stress Disorder makes the victim continuously remember the event. It was originally known as “shell shock” where vets were struggling going through daily life. Finally after the Vietnam War Post-Traumatic Stress Disorder was “identified and given its name.” When these discoveries were made, proper treatment was then given to the victims. Research shows that

When humans undergo traumatic events that threaten their safety and wellbeing, they may become vulnerable to nightmares, fear, excessive anxiety, depression, and trembling. Post Traumatic Stress Disorder (PTSD) is a psychological illness that results from the occurrence of a “terribly frightening, life-threatening, or otherwise unsafe experience” (Posttraumatic Stress Disorder (PTSD), 2012). This condition often leads to unbearable stress and anxiety. PTSD is significantly prevalent as indicated by data from the National Co-morbidity Survey which shows that at a particular time in their lives, 7.8% of 5, 877 adults in America suffered from PTSD (Andrew & Bisson, 2009). In the general population, the lifetime prevalence is estimated at 8%,

Spitalnick, Josh. Difede, JoAnn. Rizzo, Albert. O. Rothbaum, Barbara. “Emerging treatments for PTSD” Clinical Psychology Review, Volume 29, Issue 8, December 2009, Pages 715-726, ISSN 0272-7358, Web. 21 April 2016

It was not until the 1980’s that the diagnosis of PTSD as we know it today came to be. However, throughout history people have recognized that exposure to combat situations can have profound negative impact on the mind s and bodies of individuals in these situations. But there are other catastrophic events that can have such profound impact on people resulting in PTSD…

Those diagnosed with PTSD have shown a reduction in the volume of the hippocampus. The hippocampus helps humans remember new memories and then be able to recall them later, and also helps identify between past and present memories. The amygdala is another section of the brain affected by PTSD, and the amygdala is responsible for processing emotions and fear. The ventromedial prefrontal cortex is shown to have a decreased size in cases. The ventromedial prefrontal cortex regulates negative emotions like stress, anxiety, and fear. The strange behaviours of the patients diagnosed with PTSD can be explained by the damage to the brain. “Researchers believe that the brain changes caused by PTSD increase the tendency of a person developing other psychotic and mood disorders” (brainblogger.com). The brain is like a machine made up of small parts, and if one of these parts break, the machine does not function

Traumatic events also produce profound and lasting changes in physiological arousal, emotion, cognition, and memory. Moreover, traumatic events may result in the severance of these normally integrated functions from one another. Traumatized individuals may also suffer from the memories of the tragic or horrifying experiences they have undergone. Frequently, as a result of these many symptoms, it becomes inevitable for the individual to develop certain complications associated with trauma-related disorders, such as posttraumatic stress disorder.

At least 50% of all adults and children are exposed to a psychologically traumatic event (such as a life-threatening assault or accident, humanmade or natural disaster, or war). As many as 67% of trauma survivors experience lasting psychosocial impairment, including post-traumatic stress disorder (PTSD); panic, phobic, or generalized anxiety disorders; depression; or substance abuse.(Van der Kolk, et al, 1994) Symptoms of PTSD include persistent involuntary re-experiencing of traumatic distress, emotional numbing and detachment from other people, and hyperarousal (irritability, insomnia, fearfulness, nervous agitation). PTSD is linked to structural neurochemical changes in the central nervous system which may have a direct

The majority of Vietnam veterans experienced delayed or chronic PTSD which we know now, can remain dormant for years until symptoms are triggered. According to the Vietnam Veterans’ Readjustment Study, approximately 30.9% of men and 26.9% of women soldiers in Vietnam had PTSD at some point in their lives. (P8) Lifting these requirements from the DSM-III allowed many more veterans to step forward; and with this new massive population of test subjects- scientists were able to research the dynamics, variables, and patterns of PTSD, through the advancements of neuroimaging and neuroscience. (P9) “There were many more cases of PTSD among Vietnam veterans than any other war.” (P5) A number estimated to be near 500,000. (P11)

Although controversial when first introduced, the PTSD diagnosis has filled an important hole in psychiatric theory and practice when dealing with this plethora of symptoms. Throughout history the significant change brought upon by the theorization of the PTSD concept was the stipulation that the origination agent was outside the individual rather than an inborn weakness. The key to understanding the scientific theorem and clinical determination of PTSD is the concept of

It is the goal of this paper to demonstrate that the phenomenon of PTSD, despite the fact that its multiple emotional and psychosocial effects are constantly being debated, is soundly rooted in neurobiology, and that this aspect of PTSD lends support to the notion that brain = behavior.

Posttraumatic stress disorder (PTSD) is a widespread disorder that affects certain individuals psychologically, behaviorally, and emotionally following the experience of a traumatic event (Lee et al., 2005, p. 135). However, because of inconsistencies regarding the percentage of individuals who experience PTSD and the percentage of individuals who subsequently develop PTSD, researchers hypothesize that both biological and environmental factors contribute to the development of PTSD (Wolf et al. 2010, p. 328). In order gain a better understanding of this disorder and to discover contributing and predicative factors which contribute to the development of PTSD, this paper analyses the historical context and prevalence of PTSD, the

Post-traumatic stress disorder (PTSD) affects 7.7 million American adults and can also occur during childhood. PTSD is an anxiety disorder that stems from a recent emotional threat such as a natural, disaster, war, and car accidents. PTSD usually occurs from an injury or coming close death. A person who has experienced a past traumatic event has a heightened chance of being diagnosed with PTSD after a current trauma. PTSD can also be determined by looking at one’s genes, different emotions, and current or past family setting. Normally, when a person without PTSD goes through a traumatic event the body releases stress hormones, which in time returns back to normal; However, a person with PTSD releases stress hormones that do not return

The amygdala is known to learn from exposure to fear and store assessment of threat–related stimuli. The prefrontal cortex is involved in extinction and the retention of fear and is connected to the amygdala. Finally, the hippocampus encodes the context during fear learning process and sends it to the amygdala. People with PTSD have hyper-activity in the amygdala, while having hypo-activity in the prefrontal cortex and there is reduction of the hippocampus volume. This reduction may limit proper evaluation and categorization of the experience. A study on Vietnam soldiers revealed that lesions in the amygdala and prefrontal cortex resulted in the absence of PTSD. To get further into the molecular level studies been done on the hormonal system. “Stress is known to contribute to the pathogenesis of a variety of disorders, including the majority of psychiatric like major depression and posttraumatic stress disorder.” (http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3825244/pdf/DM30-02-343616.pdf). Research has revealed evidence that a hormonal system known as the hypothalamic-pituitary-adrenal (HPA) axis is the one that gets disrupted in people with PTSD. The hormonal system is involved in normal stress reactions, so the disruption of this system in people with PTSD creates this “false alarm”. It has been suggested by some scientist that the dysfunction of the HPA system results in hippocampal damage in people with PTSD. Damage in the hormones is caused by damage to

The second experiment run by Rau, Decola, & Fanselow addresses this interpretation and extinguishes the rats to Context A after they receive the pre-exposure treatment. The results show three important aspects of the enhancement of fear learning by past traumatic events. First, it shows that enhancement is prolonged and lasts at least 7 days after the trauma. The second is that it is obstinate to extinction treatment, even after being safely exposed to where the trauma occurred the effects of the trauma were not changed. Lastly, the results indicated that the enhancement in fear conditioning is not dependent on the current level of fear of the trauma. One of the potential issues with experiment 2 is that it is possible that the enhancement of fear observed in the second context is a summation of new fear-conditioned by the single shock and generalization of reinstated fear of the original traumatic context (Rau et al.,