There are many reasons why being overweight is bad for your health. It can, for example, cause or aggravate type 2 diabetes. Obesity is also a risk factor for heart disease and other cardiovascular problems. So what do you have to do to lose weight? Eat less and move more is the trite answer usually received by someone who is overweight. Of course you can lose weight by reducing the food you eat (energy intake) or increasing the amount of exercise you get (energy output). But the problem of effective weight-loss is much more complex than simply changing the balance between the calories you consume and the calories you expend in your daily activities. The search for an effective weight-loss formula requires answers to these four …show more content…
These findings explain why people with the obesity-risk variant of the FTO gene eat more and prefer higher calorie foods... even before they become overweight... compared with those with the low-risk version of the gene. The FTO gene is not the only genetic cause of obesity, which is likely to be due to the sum of several genes working together. If you have these 'bad ' genes, however, you are not necessarily destined to become overweight... but you are more likely to end up obese if you over-eat. Having these genes also means that you will need to exercise greater discipline over your diet throughout out your life, especially when you have managed to shred a few pounds and want to keep them off. How many calories should you cut to lose weight? The big question for dieters has always been... how many calories do I need to cut out of my diet in order to reduce my weight by a set amount, eg one pound or kilogram? Once upon a time there was a clear-cut answer to this question. In 1958 Max Wishnofsky, a New York doctor, wrote a paper that summed up everything known at that time about how calories are stored in our bodies. He concluded that, if your weight is being held steady, it would take a deficit of 3,500 calories to lose one pound (454 grams) in weight. You could create the calorie deficit either by eating less or exercising more (to use up more calories). For example, if your weight is holding steady on a diet of 2,000
Genes control almost every aspect of human life, and when it comes to weight there is no exception. Little is known, however, how much of an individual’s genes actually control the weight of certain individuals. Body fat can vary from person to person, yet some people have always carried more weight than others. Often times, when one person is overweight in the family, most of the other individuals in the same family are overweight as well. However, more than just genetics can go into being overweight, and a person’s genes are not the end all be all of obesity. Many times the environment surrounding people who are obese contributes to the overall weight of the individual, and when obesity promoting genes are mixed
A person’s genetic make up has a significant influence on whether the person will become obese or not. If both parents are obese, the likelihood that their children may end up being obese too is higher compared to a situation where neither of the parents is obese or where only one of the parents is obese. This is particularly so because genetics influence the way the body stores energy and how energy is used. This can be seen in the differences that have recorded in the basal metabolic rates (BMR) among groups of people who differ by age, gender and the make up of their bodies. People who have a low metabolic rate have a higher risk of becoming overweight. The genetic similarities shared by members of one family can explain why people who come form certain families end up being overweight (DeBruyne, Pinna
Obesity has been linked to genetics. It has been researched and proven that children with parents who are obese, would have between 5-25% chance of likely inheriting genes that would eventually make them obese. However, genes is not an excuse for obesity because it does not work alone but must have the right factors present to manifest.
genetic variations effect obesity will make it much easier to prevent and treat the condition of
Genetics can also cause you to become obese; the reason this can cause you to become obese
Polygenic obesity also known as “common obesity” is mutations in many genes, or the development of many environmental factors. Polygenic obesity occurs when both the genes and the environment interact with each other. An example of this would be a person being surrounded by a bad environment, promoting unhealthy habits, such as lack of exercise and an excess consumption of foods, plus with the genetic makeup of the person adding on to it. “In fact, if a biologic mother is heavy as an adult, there is a 75% chance that her children will be heavy.”(wvdhhr.org). Because this type of obesity is prone to many complex interactions, it gives way to various different outcomes. Polygenic obesity is studied in the DNA mainly through single nucleotide polymorphisms, meaning there is a single variation in a sequence of pairs in the DNA. Polygenic obesity can be controlled by the environment a person is in, by choosing healthy foods and exercising regularly obesity can be prevented. Also, a parent can prevent obesity in a child by giving them healthy food options in order to learn it at a young age a use it later on in their adult
Furthermore Science has shown that genetics contribute to a person's weight. A disorder like Prader-Willi causes a child to gain weight. Therefore leading to obstacles such as type two diabetes, high cholesterol levels, and high blood pressure. Likewise Bardet-Biedl syndrome increases one's chances of obesity. Although Bardet-Biedl syndrome results from mutations in 14 different genes, while Prader-Willi
We need enough exercise to digestion the extra fat we’ve been intake. if you want to keep the weight off, you have to combine a calorie-controlled diet with regular exercise. The most important method is management the diet. Make the right diet choice is much better than, good diet has effect of pills unable to do (baidu.com/question/49065635). We need to Consider eating reasonable allocate, to ensure that human body needs many kinds of nutriment absorb balance and enough and not overeating.
Obesity has reached epidemic proportions in the United States and developing countries. Although the trend of decreased physical activity and increased caloric intake is probably responsible for the recent rise in obesity, it is important to understand that these trends are playing out on a background of genetic variation in the population. Each individual's genetic background remains an important determinant of susceptibility to obesity. Discovery of the genes involved in the development of common forms of obesity, thereby identifying pathways that are causal in patients, will guide clinicians and scientists in designing more effective therapies and in identifying high-risk individuals for early intervention.
We need to be able to fully understand how genetics influences obesity. For example, in 2007, there was a gene identified as FTO, it was labeled the first “obesity gene”. After studying the gene for six years there was no conclusive evidence of how this gene was related to obesity, all they knew was those people with the FTO were at higher risk for obesity (Sjogren, K, 2013, section 2. Para 3).
The hormones leptin and insulin, along with sex and growth hormones influence our appetite, metabolism, and distribution of body fat. Obese people have hormone levels that encourage an abnormal metabolism which leads to the accumulation of body fat. This occurs because the human body’s endocrine system secretes these hormones into our bloodstream. If there’s an excess or deficits of hormones it leads to obesity. For example, the lower leptin levels increase a person’s appetite and slow down their metabolism, causing them to gain weight. This also plays into the role of behavioral
This genetic aspect of obesity has been shown through polygenetic obesity, that can be linked to the FTO gene, found in the hypothalamus in the brain that regulates hunger and satiety (2). The variation in the FTO gene causes people to overeat due to the lack of satisfaction and fullness that most people feel after eating (2). Studies have shown that individuals carrying this gene variation in FTO also have higher BMIs (2). By understanding how genetics play a role in obesity, it will help people understand it is not solely just the environment having an impact. This will help fix the stigma of people who live with obesity as just being lazy people. Furthermore, through Canadian Medical Association's recognition of obesity being a disease will help Canadians who are of low socioeconomic status become educated on the topic of obesity and obtain services to help improve their life style (3). According to the National Longitude Survey of Children Youth, children in lower socio-economic communities have a higher prevalence of obesity (3). Through bringing attention to obesity by declaring it a disease, it will bring more attentiveness to school around Canada, this will encourage schools to educate the students about obesity more
The calories calculators on the Internet will make you believe that your bodywork is to add and subtract calories. If you keep on eating high calorie food items your body will increase the weight. You eat more food without any exercise, you will gain weight. What they do not tell you is that your body perform other actions also. The most important factor to lose weight is your metabolic rate. Your metabolic may be able in different manner. The kind of physical activity you do is also responsible for your metabolic rate. There are several strategies, which you can apply to enhance it. You can get detailed information from ClenCycle.com.
Factors associated to obesity include an interplay between genetic, behavioral, and envi-ronmental factors (Rogers, 2010). In fact, the combination of genetic and environmental factors increases the
Everybody knows the child that can eat any type of food all day and never seem to gain weight. This occurrence has led researchers to investigate the role that genetics plays in childhood obesity. Not all children who are inactive or who eat poorly are obese, much in the same way that some obese children eat fairly healthy, and exercise moderately. Heredity has recently been shown to influence body fat percentage, regional fat storage, and the body?s response to overeating (Rush, 1). Children who have obese parents are 80% more likely to be obese than their lean parented counterparts (Buffington, 16). This familial correlation is contributed to genetics as well as the parents eating habits. Children with obese parents typically aren?t taught the correct way to choose when and what food to eat, leading to poor eating habits and eventually obesity. Many genetic defects can have a significant effect on obesity such as variable thyroid activity and pituitary defects. Abnormalities in any one of these regulators could be responsible for appetite abnormalities and weight gain. Furthermore, obesity leads to defects in appetite regulation, hormone production, and metabolic events (Oklahoma Cooperative, 4) that are responsible for further weight gain,