Long term use of high-dosage tetracycline causes cellular damage that mimics mitochondrial disease because Choose one :A. mitochondrial proteins are easily denatured by drugs like tetracycline. B. mitochondrial ribosomes are susceptible to inhibition by tetracycline. C. mitochondrial lipid bilayers become leaky due to drugs like tetracycline. D. the mitochondrial inner membrane is solubilized by tetracycline. E. mitochondrial DNA is alkylated by drugs like tetracycline..

Long term use of high-dosage tetracycline causes cellular damage that mimics mitochondrial disease because Choose one :A. mitochondrial proteins are easily denatured by drugs like tetracycline. B. mitochondrial ribosomes are susceptible to inhibition by tetracycline. C. mitochondrial lipid bilayers become leaky due to drugs like tetracycline. D. the mitochondrial inner membrane is solubilized by tetracycline. E. mitochondrial DNA is alkylated by drugs like tetracycline..

Name: Long term use of high-dosage tetracycline causes cellular damage that
Uploaded: 2024-03-20T06:42:55.000Z
Channel: Bartleby
Description: Long term use of high-dosage tetracycline causes cellular damage that mimics mitochondrial disease becauseChoose one:A. mitochondrial proteins are easily denatured by drugs like tetracycline.B. mitochondrial ribosomes are susceptible to inhibition by

Biology: The Dynamic Science (MindTap Course List)

4th Edition

ISBN:9781305389892

Author:Peter J. Russell, Paul E. Hertz, Beverly McMillan

Publisher:Peter J. Russell, Paul E. Hertz, Beverly McMillan

Chapter4: Cells

Section: Chapter Questions

Problem 15TYK

See similar textbooks

Similar questions

Some human diseases are caused by mutations in mitochondrialgenes. Which of the following statements is false?a. Human mitochondrial diseases follow a maternal inheritance pattern.b. Mutations associated with mitochondrial diseases oftenaffect cells with a high demand for ATP.c. The symptoms associated with mitochondrial diseases tendto improve with age.d. Heteroplasmy plays a key role in the severity of mitochondrial disease symptoms
Consider a gene in the mitochondrial genome that codes for a channel protein in the inner membrane that allows magnesium ions to travel freely across. If a mutation in that gene resulted in this protein allowing H+ to pass through it rather than magnesium, what would be the consequence for cellular respiration?
Describe what would happen to the precursor of a mitochondrial matrix protein in the following types of mitochondrial mutants: (a) a mutation in the Tom22 signal receptor; (b) a mutation in the Tom70 signal receptor; (c) a mutation in the matrix Hsp70; and (d) a mutation in the matrix signal peptidase.
In the Electron Transport Chain:(a). Upon the transfers, what is the final recipient of these electrons?(b). What is the compound obtained upon the completion of electron transportation?(c). What causes the changes in pH between the inner mitochondrial membrane during the electron transfer?
Which of the following statements is NOT true about the electron transport chain (ETC) and oxidative phosphorylation? A. Oxidative phosphorylation requires the enzyme complexes to be soluble in the mitochondrial matrix. B. If the proton gradient is too high, electrons will not move through the ETC. C. The movement of electrons down the ETC only happens if protons are pumped out of the mitochondrial matrix. D. Oxidative phosphorylation requires the mitochondrial intermembrane space to be more positively charged than the matrix. E. The free energy of the proton gradient can be used to create high energy bonds.
Which of the following statements is NOT true about the electron transport chain (ETC) and oxidative phosphorylation? (Only one answer applies) A. Oxidative phosphorylation requires the enzyme complexes to be soluble in the mitochondrial matrix. B. If the proton gradient is too high, electrons will not move through the ETC. C. The movement of electrons down the ETC only happens if protons are pumped out of the mitochondrial matrix. D. Oxidative phosphorylation requires the mitochondrial intermembrane space to be more positively charged than the matrix. E. The free energy of the proton gradient can be used to create high energy bonds.
Which of the following statements is MOST ACCURATE? A. The oxidation of NADH in the mitochondrial matrix results in the transfer of 1 electron to the electron transport chain. B. The reduction of NADH in the mitochondrial matrix results in the transfer of 1 electron to the electron transport chain. C. The reduction of NADH in the mitochondrial matrix results in the transfer of 2 electrons to the electron transport chain. D. The oxidation of NADH in the cytoplasm results in the transfer of 1 electron to the electron transport chain. E. The oxidation of NADH in the mitochondrial matrix results in the transfer of 2 electrons to the electron transport chain.
What are two advantages of the components of the electron transport chain being embedded in the inner mitochondrial membrane?
Proteins in the inner mitochondrial membrane are involved in which of the following processes? a. nucleotide synthesis b. OXPHOS c. autophagy d. NADH synthesis
In the 1930’s DNP was given to people to lose weight. DNP makes the mitochondrial membrane leaky to hydrogen ions. Why might this cause one to lose weight? Why would this cause death? How is this similar or different to cyanide which binds to complex IV in the mitochondria?
You are given a suspension of purified mitochondria that are known to have adequate reserves of NAD+, FAD, and ADP/inorganic phosphate. Assume that the mitochondrial preparation you are working with is free of cytosolic components (enzymes and metabolites) and assume that all the compounds listed below can cross both mitochondrial membranes, When glucose is added to this suspension: a. What is the effect on oxygen consumption by the purified mitochondria? b. Explain your answer above. When citrate is added to this suspension: c. What is the effect on oxygen consumption by the purified mitochondria? d. Explain your answer above.
Which of the following statements regarding CoQ is false a. It resides in the inner mitochondrial membrane b. It helps to pump protons after getting oxidized by complex 2 c. It is an electron carrier common to both nadh and fadh2 electron transport chains d. It is a hydrophobic protein