Question 1 In a NMJ, what happens to the sarcolemma when acetylcholine is released into the synpatic cleft? allow influx of sodium ions and trigger action potential in muscle fiber n allow influx of calcium ions and trigger action potential in muscle fiber allow influx of calcium ions and trigger release of another neurotransmitter into the sarcoplasm allow influx of sodium ions and trigger release of another neurotransmitter into the sarcoplasm

Question 1 In a NMJ, what happens to the sarcolemma when acetylcholine is released into the synpatic cleft? allow influx of sodium ions and trigger action potential in muscle fiber n allow influx of calcium ions and trigger action potential in muscle fiber allow influx of calcium ions and trigger release of another neurotransmitter into the sarcoplasm allow influx of sodium ions and trigger release of another neurotransmitter into the sarcoplasm

Anatomy & Physiology

1st Edition

ISBN:9781938168130

Author:Kelly A. Young, James A. Wise, Peter DeSaix, Dean H. Kruse, Brandon Poe, Eddie Johnson, Jody E. Johnson, Oksana Korol, J. Gordon Betts, Mark Womble

Publisher:Kelly A. Young, James A. Wise, Peter DeSaix, Dean H. Kruse, Brandon Poe, Eddie Johnson, Jody E. Johnson, Oksana Korol, J. Gordon Betts, Mark Womble

Chapter10: Muscle Tissue

Section: Chapter Questions

Problem 12RQ: Muscle relaxation occurs when ________. calcium ions are actively transported out of the...

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Question 1. Describe an action potential in a muscle fiber in detail, know the changes in voltage starting from RMP. Question 2. What is the voltage that causes the sarcoplasmic reticulum to release calcium ions into the cell? Which integral membrane protein is responsible Question 3.What happens to a muscle fiber’s membrane (sarcolemma) once the motor neuron releases theneurotransmitter listed above? What is the change in voltage in the motor end plate? Question 4. Describe how is acetylcholine (neurotransmitter) is released from the synaptic knobs of motor neurons and how it activatesskeletal muscle. Please please please help with these questions, so so srry I know its a lot i am just really stuck! Thank youuuuuu.
Question 7 Between threshold and maximal stimuli, I observed: 1. that the force of contraction was most influenced by the placement of the electrode 2. that the force of contraction was unrelated to the magnitude of the stimulus 3. that the force of contraction decreased as the magnitude of the stimulus increased 4. that the force of contraction increased as the magnitude of the stimulus increased
Question 3 During muscle contraction, the agonist while the antagonist 1. Shortens; lengthens 2. Remains unchanged; lengthens 3. Shortens; remains unchanged 4 lengthens; shortens
Fatigue is not well understood. Some factors that have been proposed to explain the fall in force duringfatigue include: changes in the 'sense of effort', loss of 'central drive', failure of neuromuscular propagation, reduction in calcium release in excitation- contraction coupling, metabolic changes in the muscle, and reduction in muscle blood flow owing to compression of blood vessels. 1. Do your experiments help to decide which factors are important?
Human Anatomy & Physiology I (BIO168) Sequence of Events for Muscle Contraction Practice Worksheet See if you can place the events that occur during muscle contraction and relaxation in their proper order. Insert numbers in the paces provided to order the events in the proper sequence. _____ A. An electrical impulse travels over the sarcolemma and inward along the T-tubules, causing sacs in the sarcoplasmic reticulum to release calcium. ____ B. The release of ACh stops and acetylcholinesterase breaks down any remaining ACh. _____ C. Troponin and tropomyosin prevent the myosin heads from grasping the thin filament, and the muscle fiber relaxes. _____ D. An electrical impulse causes small vesicles at the end of a motor neuron to release the neurotransmitter acetylcholine (ACh). _____ E. Calcium binds with the troponin on the actin filament, exposing attachment points. _____ F. ACh diffuses across the synaptic cleft, where it stimulates receptors in the…
Please ASAP. Thanku. Question 15 If Na+ channels are blocked by lidocaine (sodium channel blocker), what happens to the equilibrium potential for Na+? Becomes more positive Becomes more negative Does not change
what is happening here in muscle contraction. Be sure to tell me what role Ca+, ATP, myosin, and actin playwhat is happening here in muscle contraction. Be sure to tell me what role Ca+, ATP, myosin, and actin play
10.In muscle function at the sarcomere level, the formation of cross-bridges is regulated by the binding of ___ ions to ____.(This is a multiple choice question choose the answer from the choices below) A.calcium; tropomyosin B.calcium; troponin C.sodium; tropomyosin D.sodium; troponin
Questions A. What happens to the axon potential that arrives at the sarcolemma? B. What happens once the action potential arrives at the terminal cisternae of sarcoplasmic recticulum? C. What happens once calcium ions are released into the sarcoplasm?
Question 10 What part of the brain acts as a relay station for incoming sensory information? Select one: a. Hypothalamus b. Thalamus c. Pituitary gland d. Endocrine system e. Amygdala
Please answer all of this following questions in your own words, thank you. Q1: Which muscles form the quadriceps? How do they function together? Q2: studied the steps of skeletal muscle contraction (sliding filament mechanism). Briefly outline the steps of the sliding filament model of muscle contraction. just mention what is happening with each of the "filaments." Q3: What would be the effect on the muscle if acetylcholinesterase was not present? (8)
Question- 2. How is your cardiac output (CO) maintained? In your explanation, give a detailed description of the ways that cardiac output is maintained. Based on your knowledge of cardiac output, how does your body regulate CO when you are lying down and then get up very quickly? Describe how the change in posture changes your CO and what effect that has and how your body corrects for this so that homeostasis is maintained.