Suppose a drug blocks entry of calcium from the extracellular fluid. What if, anything, would happen to the force of skeletal muscle or smooth muscle contraction if you stimulated them to contract under these conditions (as compared to normal) skeletal: unable to contract; smooth: no change skeletal: decreased, but still contracts; smooth: decreased, but still contracts skeletal: decreased, but still contracts; smooth: unable to contract skeletal: no change in force; smooth: decreased, but still contracts skeletal: no change in force; smooth: unable to contract O O O O O

Suppose a drug blocks entry of calcium from the extracellular fluid. What if, anything, would happen to the force of skeletal muscle or smooth muscle contraction if you stimulated them to contract under these conditions (as compared to normal) skeletal: unable to contract; smooth: no change skeletal: decreased, but still contracts; smooth: decreased, but still contracts skeletal: decreased, but still contracts; smooth: unable to contract skeletal: no change in force; smooth: decreased, but still contracts skeletal: no change in force; smooth: unable to contract O O O O O

Anatomy & Physiology

1st Edition

ISBN:9781938168130

Author:Kelly A. Young, James A. Wise, Peter DeSaix, Dean H. Kruse, Brandon Poe, Eddie Johnson, Jody E. Johnson, Oksana Korol, J. Gordon Betts, Mark Womble

Publisher:Kelly A. Young, James A. Wise, Peter DeSaix, Dean H. Kruse, Brandon Poe, Eddie Johnson, Jody E. Johnson, Oksana Korol, J. Gordon Betts, Mark Womble

Chapter10: Muscle Tissue

Section: Chapter Questions

Problem 8RQ: Depolarization of the sarcolemma means ________. the inside of the membrane has become Less negative...

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Similar questions

Motor neurons innervate muscle fibers at specialized synapses called_______ . The release of acetylcholine at these synapses ultimately triggers an action potential in the muscle fiber, which invades the interior of the fiber through_____ and stimulates the release of calcium ions from the_______ .
hen an action potential arrives at the nerve terminal of a neuromuscular junction, which of the following statements best describes the events that occur? Depolarisation of the nerve terminal causes the release of ACh which activates nicotinic receptors on the skeletal muscle membrane to cause Ca2+ entry and muscle contraction. Depolarisation of the nerve terminal opens voltage-gated Ca2+ channels, Ca2+ entry brings vesicles containing ACh to the membrane which form fusion pores causing the release of ACh which activates voltage-gated Na+ channels at the end-plate. Depolarisation of the nerve terminal opens voltage-gated Ca2+ channels, Ca2+ entry brings vesicles containing ACh to the membrane which form fusion pores causing the release of ACh which binds to muscarinic receptors at the end plate. Depolarisation of the nerve terminal opens voltage-gated Ca2+ channels, Ca2+ entry brings vesicles containing ACh to the membrane which form fusion pores…
What property do all muscle tissues have in common?a. composed of collagen and elastin filamentsb. under conscious controlc. ability to form a communication networkd. ability to contracte. cells are joined by tight junctions
From which embryonic cell type does muscle tissuedevelop?a. ganglion cellsb. myotube cellsc. myoblast cellsd. satellite cells
Recently, it has been observed that several muscle diseases and disorders are linked to problems with triad structure and function.One such disorder is called malignant hyperthermia, a dire hypermetabolic response to certain inhaled anesthetics such as halothane. Muscles become very rigid due to excessive contraction (especially the masseter muscle), the body temperature spikes, andmuscles break down. Death is certain if not treated. Given the principal symptoms of malignant hyperthermia, what is its likely cause?Propose a theoretical mechanism for a treatment that would dampen the deleterious effects of this disorder.
When a(n) __________ moves along the sarcolemma, the membrane potential changes along the membrane due to the opening and closing of voltage-gated Na+ and K+ channels. action potential sarcoplasmic reticulum calcium ion sarcomere
Skeletal, smooth, and cardiac muscle are similar in that they all _____. A are divided into motor units B require calcium in order to contract C undergo mitosis frequently to replace dead or damaged cells D have small round cells connected by gap junctions
Calmodulin is found in smooth muscle cells and performs a similar function to troponin in striated muscle fibers. However, calmodulin not only gets activated by Ca2+ but also slows the release of Ca2+ from the sarcoplasmic reticulum. How might this difference relate to the different kinds of contractions smooth muscles do in comparison to striated muscles?
Which statement about striated skeletal muscle is true? A. The tension generated by a muscle is invariable. B. Mechanical summation of twitches in a muscle fiber leads to a graded increase in the tension that is above that generated by a single twitch. C. A single action potential arriving at the neuromuscular junction is not sufficient to cause a muscle fiber to twitch. D. Muscle twitches are able to mechanically sum when Ca2+ is quickly and completely removed from the sarcoplasm between action potentials. E. An action potential in the muscle cell activates contraction by releasing Na+ into the sarcoplasm.
Which statement or statements describe why depletion of ATP after death leads to rigor mortis? Select all that apply. ATP is required to move tropomyosin back over the active sites of thin filaments. ATP is required for myosin thick filaments to detach from actin thin filaments. ATP is required to pump calcium back into the sarcoplasmic reticulum. ATP is required for the action potential to spread across the sarcolemma.
muscle spidnles are _____ receptors and provide ____ information, whereas golgi tendon organs respond primarily to ___ and cause a ____ reaction 1. Tension 2. Sensory 3. relaxation 4. stretch
Which statement or statements describe the role of calcium in the stimulation of muscle fibers? Select all that apply. When the action potential reaches the axon terminals of a neuron, calcium channels open and calcium floods into the axon terminal, triggering the release of synaptic vesicles. When calcium ions flood out of the sarcoplasmic reticulum, they bind troponin molecules and troponin removes tropomyosin from the active sites on thin filaments. When myosin thick filaments are walking along actin thin filaments (the cross bridge cycle), calcium ions must be available to bind to myosin so that it can let go of the thin filaments and take another "step". When synaptic vesicles fuse with the plasma membrane of axon terminals, calcium is released into the synaptic cleft and binds to receptors in the motor end plate.