The reason a heroin addict feels very ill immediately after going "cold turkey" from heroin is likely a result of upregulation of opiate receptors in the postsynaptic cells. upregulation of endogenous opiates. downregulation of opiate receptors in the postsynatic cells. increased postsynaptic receptor sensitivity. downregulation of opiate reuptake transporters.

The reason a heroin addict feels very ill immediately after going "cold turkey" from heroin is likely a result of upregulation of opiate receptors in the postsynaptic cells. upregulation of endogenous opiates. downregulation of opiate receptors in the postsynatic cells. increased postsynaptic receptor sensitivity. downregulation of opiate reuptake transporters.

An Illustrated Guide To Vet Med Term

4th Edition

ISBN:9781305465763

Author:ROMICH

Publisher:ROMICH

Chapter13: Nerves Of Steel

Section: Chapter Questions

Problem 5CTE

See similar textbooks

Similar questions

A patient takes a stimulant that produces euphoria, as well as an increase in heart rate and blood pressure and it treats narcolepsy. To exert its effect, the drug must be taken up into nerve terminals, where it stimulates the release of norepinephrine and dopamine from their vascular storage sites.
the degree of whole body effect of a neurohormone in the body is larger than the effect of a paracrine agent in the body true or false
The discovery that the brain contains specific receptors for manufactured opiate drugs such as morphine implies that the body must make an _______ substance to interact with the same receptors. inhibitory excitatory endogenous exogenous
Reserpine is a drug that can control high blood pressure by reducing the number of catecholamine neurotransmitters present in the synapse. Epinephrine, norepinephrine, and dopamine are examples of catecholamine neurotransmitters. One of the known side effects of reserpine is to cause the symptoms of Parkinson’s disease. Parkinson's disease is associated with dopamine. Parkinson's disease occurs when the nerve cells in the part of the brain that controls muscle movement are gradually destroyed and the neurons can no longer produce dopamine to coordinate muscle movements. Reserpine causes symptoms by a. inhibiting the release of dopamine from the presynaptic neuron b. blocking the dopamine receptor in the postsynaptic neuron c. breaking down the neurotransmitter acetylcholine in the synapse d. breaking down cholinesterase enzyme in the synapse
Hormones (and/or medicines) which bind to the same receptor and have the same effects are called antagonistic
Describe how the process of opioid withdrawal leads to a reduction in the release of dopamine? Explain in a few sentences
Which of the following is an effect of cocaine and amphetamine? Group of answer choices Blocking dopamine reuptake Activating acetylcholine receptors on VTA neurons Directly activating dopamine receptors Inhibiting GABA release from interneurons All of the above
Which of the following is NOT a functional or structural change that occurs in the mesocorticolimbic dopamine system as a result of exposure to addictive drugs? A) Decreased baseline levels of dopamine activity, causing a decreased response to normal rewarding stimuli. B) Neural plasticity in response to drugs and drug-related stimuli decreases. C) Potentiation of synapses between glumate-releasing neurons and dopamine-releasing neurons in the VTA. D) Increased dendrite length and greater synaptic complexity in the nucleus accumbens and prefrontal lobes.
The effector is the organ that response to the release of hormones in the body. Based on diagram what organ would be the effector?
If incoming serotonin axons were destroyed, LSD would still have its full effects. However, if incoming dopamine axons were destroyed, amphetamine and cocaine would lose their effects. Explain the difference.
Which of the following hormone is responsible for the activation of phospholipase C?a) Serotoninb) Cortisolc) Vasopressind) Adrenaline
Imagine that a new type of psychoactive drug has been developed in a laboratory. It works by slowing the reuptake of dopamine in some brain circuits, increasing the amount of dopamine in the synapse. It also blocks serotonin binding in other brain circuits. Based on only this information, you can conclude that this new psychoactive drug is a: dopamine and serotonin antagonist. dopamine antagonist and serotonin agonist. dopamine and serotonin agonist. dopamine agonist and serotonin antagonist.