Which of the following possibilities could explain how PcGcomplexesare able to silence genes?a. The compaction of nucleosomesb. The attachment of ubiquitin to histone proteinsc. The direct inhibition of transcription factors, such as TFIIDd. All of the above

Which of the following possibilities could explain how PcGcomplexesare able to silence genes?a. The compaction of nucleosomesb. The attachment of ubiquitin to histone proteinsc. The direct inhibition of transcription factors, such as TFIIDd. All of the above

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Description: Which of the following possibilities could explain how PcGcomplexesare able to silence genes?a. The compaction of nucleosomesb. The attachment of ubiquitin to histone proteinsc. The direct inhibition of transcription factors, such as TFIIDd. All of t

Biology (MindTap Course List)

11th Edition

ISBN:9781337392938

Author:Eldra Solomon, Charles Martin, Diana W. Martin, Linda R. Berg

Publisher:Eldra Solomon, Charles Martin, Diana W. Martin, Linda R. Berg

Chapter14: Gene Regulation

Section: Chapter Questions

Problem 6TYU

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Which of the following mechanisms may be involved when PRC1complexes silence gene expression?a. The compaction of nucleosomesb. The attachment of ubiquitin to histone proteinsc. The direct inhibition of transcription factors, such as TFIIDd. Any of the above may be involved in silencing of gene expressionby PRC1 complexes.
Which of the following is true about chromodomains? a) bind methylated histone tails b) are associated with repression c) both a and b d) none of the above
Which of the following is a function of the piRISC?a. Inhibits transcription of TEsb. Causes the degradation of TE RNAc. Causes chromosome breakaged. Both a and b
Which of the following is true? a. Enhancers are proteins that bind to activator DNA sequences. b. Activators are proteins that bind to insulator sequences c. Repressors are proteins that bind to enhancer DNA sequences. d. Repressors are proteins which bind to silencer DNA sequencers.
Which of the following is a function of the piRISC?a. Inhibits transcription of TEsb. Causes the degradation of TE RNAc. Causes chromosome breakaged. Both a and b are functions of the piRISC.
Which of the following mutations could be appropriately describedas a position effect?A. A point mutation at the –10 position in the promoter regionprevents transcription. B. A translocation places the coding sequence for a muscle-specificgene next to an enhancer that is turned on in nerve cells.C. An inversion flips a gene from the long arm of chromosome 17(which is euchromatic) to the short arm (which isheterochromatic).
Which of the following best represents the central dogma of geneexpression?a. During transcription, DNA codes for polypeptides.b. During transcription, DNA codes for mRNA, which codes forpolypeptides during translation.c. During translation, DNA codes for mRNA, which codes forpolypeptides during transcription.d. none of the above
Which of the following are examples of molecular changes thatcan have an epigenetic effect on gene expression?a. Chromatin remodelingb. Covalent histone modificationc. Localization of histone variantsd. DNA methylatione. Feedback loopsf. All of the above
Which of the following decrease gene activity? a. acetylation of histone proteins b. methylation of histone proteins c. methylation of cytosines in DNA d. all the above decrease gene activity
Explain how a gene knockdown reagent, such as RNAi or a morpholino, could interfere with expression of a gene without affecting the rate of transcription of the target gene.
If a mutation occurred in a control element sequence associated with a particular gene, which of the following would be a likely consequence of this? A. This mutation is not likely to affect the expression of the associated gene, because RNA polymerase does not bind to control elements. B. This will likely affect the expression of the associated gene because it will loosen the coiling of chromatin at this site. C. The expression of the associated gene will be affected because this change will result in a nonsense mutation. D. This mutation is not likely to affect the expression of the associated gene, since control elements are noncoding sequences. E. The associated gene's expression would likely be affected, since this mutation would change the ability of at least one transcription factor to bind.
You manipulate the cro and cI/lambda repressor proteins so that they end up with swapped DNA binding domains (cI now has cro’s DNA binding domain, and cro now has cIs). This mutant bacteriophage then infects a happy, healthy population of bacterial cells. How will this modified cro protein interact with the three OR sites, and how would cro expression be affected? Include a drawing if possible.