Thrombus

Cardiovascular diseases are diseases which involve the heart or blood vessels, they are in fact the leading causes of death and disability in the world. Cardiovascular diseases include coronary artery disease, or ischaemic heart disease (heart attack), cerebrovascular disease (stroke) and diseases of the aorta and arteries including hypertension and peripheral vascular disease (Mendis, Puska, & Norrving, 2011). They are all due to a disease known as atherosclerosis which affects arteries (George

our assessment is focused on a potential deep vein thrombosis (DVT). A DVT is a essentially a ball of blood cells that are clumped together impeding blood flow which can cause irritation to the vein and potentially more dangerous problems. If the thrombus moves, it can travel through the body and into the lungs and impeded breathing which is not good. Therefore, it is essential to catch DVT’s right away and treat them to prevent them from moving. The first thing that needs to be assessed when a patient

Hemostasis is the stoppage of blood flow. In order to do this our body’s rely on an “adequate amount of platelets, normal levels of coagulation factors, and absence of defects in vessels walls” (Huether and McCance 540). In platelet disorders both quantitative and qualitative factors can interrupt normal blood coagulation which prevent hemostasis (Huether and McCance 541). Quantitative abnormalities in a platelet disorder would be an increase or decrease in the number of platelets disorders that

Thrombus formation is generated by vessel damage and the subsequent thrombogenic stimuli exposure and consists of activated platelets and fibrin protein. 3 This barrier that is created limits the blood flow through coronary vessels causing myocardial ischemia

Introduction The previous reviews have highlighted the treatment interventions for structural heart disease. Noninvasive cardiac imaging has played a critical part in the evaluation, monitoring and follow-up of these procedures. This review will highlight the role of cardiac CT, fluoroscopy and fusion imaging to guide the percutaneous strategies used to treat valvular and structural heart disease described in the previous reviews. TAVR Role of CT Individuals deemed inoperable, high, or intermediate

acute vasoconstriction or thrombus. In normal development, the individual does not develop angina because the lumen of the coronary arteries are patent without occlusion from atherosclerotic plaques, vasoconstriction or thrombus. The onset of symptoms is sudden when thrombus formation is the direct cause of unstable angina. Following the onset of symptoms, unstable angina progresses rapidly and ends in severe symptoms at rest. The rupture of a vulnerable plague with thrombus formation influences the

be sucked down towards the cannula resulting in obstruction of inlet flow. We will see this first from the parasternal long axis view. When applying Doppler to the inflow cannula, the characteristic of the flow pattern can represent obstruction or thrombus within the cannula. Flow pattern showing slow, turbulent and/or lack of flow indicates some type of obstruction. With an LVAD, the aortic valve should remain shut or only open once every few beats. Increase your beats on the PLAX view and

mainly from an embolism. An embolism is the breakage of an atherosclerosis formed in any coronary arteries. This floats freely in the blood stream, which eventually may plug a major artery in the brain (stroke), heart (MI) or lungs (lung failure). A thrombus occurs when an already

Complications of hemodialysis Although used as life sustaining therapy, hemodialysis has its own set of complications and it is indeed a challenge for nephrologists to deal with the complex relationships between the patients’ comorbidities, uremic syndrome and the potential complications of dialysis. The complications of hemodialysis have been grouped in several categories. 1) Vascular access complications a) Arteriovenous fistula complications An arteriovenous fistula is a connection of an artery

Haemostasis = stable fibrin-platelet plug. • Haemostasis divided into two part based on function: -Blood vessels , Platelets and Coagulation factor : the function of this three components is make thrombus formation. - Coagulation inhibitors and Fibrinolysis : to Localize thrombus on injury and Prevent new thrombus formation. • How this process is done? ( Through the following explanation we will understand the function of each component of Haemostasis)  In Primary Haemostasis:( Adhesion,degranulation