Air Entering The Vascular System

Mr. S was driving when he experience a stabbing chest and back pain for the first time. The pain was so severe he immediately went to his local ER. Pulmonary ventilation and perfusion (VQ) scan and Computed tomography angiography (CTA) was done at his local ER. VQ scan was negative for pulmonary embolism (PE). CTA of the chest revealed

Cerebral air embolism is a very rare complication but lethal. We report a case of patient of cerebral air embolism as a complication of placement of pigtail pleural catheter placement while a patient on (PPV).

The patient did have black soot around his nose and mouth. Thats when first responders started manual ventilation's via BVM and 02 at 15 LPM. At this time Medic 1 assumed patient care. Medic 1 assigned first responders to obtaining vitals signs that are stated in the vital section of the report. It was at this time that Medic 1 applied a OPA after first measuring on what size to use. First responders also applied fast patches to the patients right upper chest and left midaxillary line At this time Medic 1 assigned first responders to start chest compressions a 15:2 ratio. Medic 1 at this time started a IO in the patients plateau region of the right leg. The Plateau region is inferior and lateral to the knee cap. At this time Normal Saline bolus was started with a 60 drop per ML set. Medic 1 found the patient to be in a sinus rhythm At this time miscommunication with Medic 1 and first responders happen with chest comparisons started. We then secured the patient on the cot via 4 straps and transported a code red patient to the nearest hospital. While enroute to hospital radio report was given with chief compliant and treatments listed in the appropriate category of the report. Vitals was continued to be taken every 5

A.W., a 52-year-old woman disabled from severe emphysema, was walking at a mall when she suddenly grabbed her right side and gasped, “Oh, something just popped.” A.W. whispered to her walking companion, “I can’t get any air.” Her companion yelled for someone to call 911 and helped her to the nearest bench. By the time the rescue unit arrived, A.W. was stuporous and in severe respiratory distress. She was intubated, an IV of lactated Ringer’s (LR) to KVO (keep vein open) was started, and she was transported to the nearest emergency department (ED).

This scenario helped me understand the pathophysiologic process of pneumonia and decompensated shock and how they could possibly manifest in children. Since in our first simulation of the semester we learned different methods of assisting a patient in improving his/her breathing status I was better able to intervene and know what to do to improve our patient’s breathing status. However, I have never been exposed to a patient undergoing decompensated shock. Therefore, this time I was able to learn what to do in case a situation as such arises in the future on a real patient. Shock can be due to several reasons such as bleeding or severe dehydration. However, it was apparent in this case that the patient was not externally bleeding, but she was

Acute chest syndrome has been studied extensively in the past due to two reasons. First, it is the leading cause of death in patients with sickle cell disease. Secondly, there is not optimal treatment for acute chest syndrome. This is because there are so many factors that can cause it. Pulmonary fat embolism can cause acute chest syndrome, but so can viral infections, bacterial infections, and mixed infections (Vichinsky, 2000). While there are so many causes of acute chest syndrome, the most noticeable was pulmonary fat embolism, which account for 8.8% of all acute chest syndrome occurrences (Vichinsky, 2000). On table 4, you can see each disease that caused the onset of acute chest syndrome, and how many people it affected.

The cardiovascular system is a network of the heart, blood vessels and the blood. It circulates blood throughout the blood vessels in our body in order to provide individual cells with oxygen and nutrients, and help to dispose of metabolic waste. Capillaries, which are the smallest blood vessels, have extremely thin walls. This is so diffusion can happen more efficiently. Every cell has capillaries passing through very close so there is a shorter distance for diffusion to occur. During my experiment, it showed that the larger the agar cube, the more of remaining agar which was left to be diffused. It also showed that the smaller the agar cube, the less remaining agar was left to be diffused. The findings from my experiment prove that the smaller

We present a case of 51-year-old male who presented to emergency room with complaints of typical chest pain.

One day we will all face the inevitable guest of death. But nevertheless, there are many options when a terminal illness strikes you or someone you love. For example, you may choose to fight and attempt to move forward with a sense of hope or allow the prognosis to paralyze you, and eventually give up on life. Paul Kalanithi, the author and main character of the autobiography, When Breath Becomes Air, was faced with this choice after being diagnosed with lung cancer at the age of 36. Paul did not have a clear vision of the future, but he deemed himself worthy to go on.

Venous thromboembolism refers to the formation of a blood clot in a blood vessel. While clots can form in an artery or a vein, this article focuses only on clots that occur in a vein ("," 2015). Critically ill patients are at an increased risk of a venous thromboembolism (VTE) due to VTE can manifest as a deep venous thrombosis (DVT) or a pulmonary embolism (PE). Risk factors include venous stasis, vascular injury, and hypercoagulable disorders. A majority of ICU patients carry at least one risk factor for VTE; additional risk factors are considered to have a cumulative effect…it is impossible to predict which patients will experience a

Pt also has a history of deep vein thrombosis (DVT) with an inferior vena cava filter to capture any lose blood cells. The chest pain he is experiencing is no cardiac secondary to chest wall pain which is symptoms of his small cell carcinoma of the lung. Patient is required to use oxygen as needed for his SOB secondary to his lung cancer. No evidence of a compression fracture was found in the spine, patient has prior laminectomy postoperative changes in the lower lumbar secondary to previous falls.

In 1974 Ward reported an incidence of 3% of symptomatic pneumothorax after ISB by paraesthesia blind technique. The pneumothorax was almost certainly produced by the prior attempt to carry out a supraclavicular block, as it is difficult to imagine the apex of the lung reaching as high as C6, the level of an

Compare and contrasts the anatomy and physiology of blood vessels: arteries, veins and capillaries. Why are these differences significant in the overall function of the circulatory system? Please be specific.

The high risk patients are those who are I danger of severe complications such as death due to the severity of symptoms in which they are hemodynamically unstable. Low risk patients usually have different treatment regimens from high risk patients as they are not in any immediate danger. Van der Hulle, T. (2015, August 19). When it comes to high risk patients they are usually given thrombolytic therapy to resolve the blood clot obstruction in the pulmonary artery. Because thrombolytic can thin the blood so easily they are only ever used in life threatening situations because it can cause major bleeding. There are alternatives to thrombolytic therapy if a patient isn’t able to use that treatment. There is surgical embolectomy in which the embolus is surgically removed. Low risk patients are patients who are hemodynamically stable. The treatments for low risk patients are usually blood thinners such as Coumadin (Warfarin) to help thin the blood so that the clot can be dissolved. Although it runs the risk problems such as death from blood loss due to the inhibited ability for the blood to clot. It is recommended that patients adhere to the plan of care and take the drugs as prescribed to prevent the reoccurrence of another embolism which could lead to even worse complications such as organ damage or even death. Low risk patients are

veins to the tissues of the body. These walls do not let out blood but