Blood Test Persuasive Speech

PHYSICAL EXAMINATION: Vital Signs. TEMPERATURE: 101.0, Blood Pressure- 127/179, Heart Rate-129, Respirations- 185, Weight-215. Situations 96% on room air. Pain Scale- 8/10. HEENT-Normal cephalic, atrumatic pupils equally round and reactive to light. Extra ocular motions intact. ORAL: Shows oral pharynx clear but slightly dry mucosal membranes. TMS: Clear. NECK: Supple, No thrangegally or JVD. No cervical, subclavicular, axilarry or lingual lymphinalpathy.

PLAN: I have reviewed the chest x-rays available here and agree with the finding of bleb formation in the right and left upper lobes. Despite the fact that the patient has had a high INR, because of his history of tuberculosis and hemoptysis I believe obtaining sputum for TB is very, very important. We should rule out any other endobronchial lesions as the cause for his bleeding. I have discussed this matter with the patient and his wife. I have told them that there is the possibility of observing the condition by x-rays and repeated tests of his sputum. They understand that this is an option; however, they decided that because of concern regarding his repeated hemoptysis, they would consent to bronchoscopy. We will arrange for the patient to have a bronchoscopy done. He is off Coumadin.

Physical Examination: General: The patient is an alert, oriented male appearing his stated age. He appears to be in moderate distress. Vital signs: blood pressure 132/78 and pulse 68 and regular. Temperature is 38.56 oC (101.4 oF). HEENT:Normocephalic, atraumatic. Pupils were equal, round, and reactive to light. Ears are clear. Throat is normal. Neck: The neck is supple with no carotid bruits. Lungs: The lungs are clear to auscultation and percussion. Heart: Regular rate and rhythm. Abdomen:Bowel sounds are normal. There is rebound tenderness with maximal discomfort on palpation in the right lower quadrant. Extremities: No clubbing, cyanosis, or edema.

The presence of fluid in the alveolar space could potentially cause the lung capacity to be effected as well.

The secondary pathology is the pathology caused by the primary. With the cavitation of the lung, swelling of tissues, and lymph nodes surrounding the chest cavity, this can put pressure on the heart, veins, and arteries. Bob displays symptoms of the upper limb, headache, congestion in the nasal passages and hoarseness. These are symptoms as a result of damaging blood flow to the upper body.

At this time we don’t have a status on his medical condition. We will continue to follow this story as it develops.

Lungs: Clear to auscultation bilaterally. No wheezes, rales, or rhonchi. Symmetric chest expansion. Breathing nonlabored.

Pleural line abnormalities – “The pleura only become visible when there is an abnormality present…Some diseases of the pleura cause pleural thickening, and others lead to fluid or air gathering in the pleural spaces.” (https://www.radiologymasterclass.co.uk/tutorials/chest/chest_pathology/chest_pathology_page4)

I agree with you, in the U.S. we spend too much time on preparation for testing. I did not expect to find such a high numbers, over one hundred mandated standardized test required between K-12. You mentioned in your writing about hands on testing I'm a strong supporter of that approach and have seen the benefits not only in educational institutions but also during workplace training sessions.

A: Janie is a 60 year old Female with PMH of A-Fib, COPD, Hypothyroidism, HTN, Lung Cancer and recently diagnosed Pulmonary Embolism. Janie presents to ER for evaluation on SOB, cough with greenish sputum, sore thoart, hoarseness and generalized weakness. Janie lives at home with her husband, use to smoke ½ pack per week, but quit many years ago, denies alcohol or drugs. Family history is non-contributory. Allergies: NKDA. Differential diagnosis includes worsening Lung Ca, PE, COPD and CHF. Janie uses home O2 at 4 L/NC. V/S: T=98.7, HR=89, R=16, B/P=132/56, O2 sats=100% on 4L/NC, Pain=6/10. Labs: WBC=7.6, H&H=8.5/27, Na=141, Troponin=0.08/0.06, BNP=495, INR=4.2, UA=3+ protein, 1+ blood and 6-10 RBC. CXR: Impression:1). COPD with nonspecific coarsening of the basilar interstitium. 2). Mild cardiomegaly with borderline cardiac compensation. 3). Right

On January 4th, 2017, you issued an emergency call for blood and platelet donation because the severe winter weather was eating up your blood supply causing a shortage and once again you issued a statement on July 5th, 2017, saying that, “The decline in summer donations is causing a significant draw-down of our overall blood supply, and we urgently need people to give now to restock hospital shelves and help save lives,” (Mandal). So, if I am understanding this correctly, the nation is still experiencing a blood shortage? Ok, here’s the part that I find funny, there’s an entire population that is able to donate but yet are deferred by your system. Can you guess what that population is? No? Well, it’s gay and bisexual men.

Pleural effusion is defined by Liz Allibone as “an abnormal collection of fluid in the pleural space caused by a variety of mechanisms.” The pleural space is a cavity between the chest wall and the lung that consists of two layers, the parietal pleura and the visceral pleura. These two layers normally have pleural fluid within them to reduce friction during respiration. Allibone quotes about normal pleural fluid levels that “…it is suggested that there is around 0.2-0.4 ml/kg in a healthy adult.”

Pulmonary Enema can be identified in a PA and lateral chest radiograph, using a horizontal beam which is critical for the projection of air-fluid levels. It appears an increasingly diffuse in radiodensity in the hilar regions, interstitial spaces and the interlobar septa. Clinical indications or signs of the condition (pulmonary edema) include Signs of the condition include a rapid respiratory rate, heaving of the chest and abdomen, intercostal muscle retractions, and cyanosis. To improve the movement of air into and out of the chest, the patient will often sit upright to breathe and resist laying down. (Venes, D, 2005 p. 666 )

Johns’s low blood pressure may cause due to the restriction of blood flow to the heart. John was initially diagnosed with spontaneous pneumothrax caused by several ruptured bleds. In cases of pneumothorax, it is common to experience tension pneumothorax. In tension pneumothorax, air gets into the pleural space and gets trapped there (Luh, 2010). As the pressure builds the lung collapeses and pushes major structures in the center of the chest. The shift in major structures causes the other lung to become compressed and affects the flow of blood returning to the heart (Luh. 2010). The lack of blood returning to the heart is why we see that John had such a low blood

Just imagine if one of your brothers or sisters was born with a heart defect and had to have daily transfusions of blood in order to have a chance of survival. Unfortunately, things happen like this everyday and some of you may have experienced them already. In this essay I am going to persuade you to become a blood donor. I try to donate blood every two months or so because it makes me feel like I am doing something good and saving people’s lives. Do you know that 95% of all Americans will need a blood transfusion sometime in our lives (Red Cross web site)? That statistic means that there is a possibility that you, and your family and friends could need support from volunteer blood donors. In this essay I will discuss the need for blood donations, solutions that can increase blood donations, and actions that need to be taken to increase blood donations.