Essay On Asd Children

The DSM-5 notes frequent co-occurrence of other neurodevelopmental disorders. However, if a child’s symptoms are better and more completely explained by ASD, intellectual developmental disorder, global developmental delay or another mental disorder, a diagnosis of SCD cannot be made. The notable absence of restricted and repetitive behaviors is the important in appropriately diagnosing a patient with SCD versus ASD.

The symptoms of Asperger’s are some of the following. They usually are socially and emotionally inappropriate. They lack the ability of being able to interact with other individuals successfully. They have a peculiar use of language and speech. They sometimes speak in

In 2013 DSM-5 was published and the information within this publication has changed greatly in relation to possible ASD diagnoses. DSM-5 has removed the indicators of cognitive delay and language delay from diagnoses, and that they are in fact markers of intellectual impairment, which 40% of children with an ASD

A.1.Motivation of the proposed research. NDDs, including ID and ASDs, are the most devastating brain disorders affecting children (13). The prevalence of ASDs is on the rise, and yet, no effective treatments exist. The US Centers for Disease Control and Prevention (CDC) reported that 1 in 68 children at 8 years of age had ASDs in 2014, up 220% since 2002. ID, another severe NDD affecting 3% population characterized with IQ less than 70 (14). Over 3.5 million Americans live with ASDs suffering from life-long disabilities. An ASD family spends $60,000 a year on average for care. The clinical presentation of ASDs is complex. The impaired social-communication functions and restricted, repetitive pattern of behaviors can be detected in children

While there are many difficulties imposed by ASD, it does not appear that ASD has the power to take away one’s ability to learn. During the aforementioned 2003 study of symptom severity in autism, a group of adolescents (age 10-21, mean age = 15.61) was compared to a cohort of adults (age 22 and older, mean age = 31.57), it was observed that symptoms seem to improve overtime. The study attempted to compare childhood symptoms to current symptoms. The results indicated that there was differential improvement; there was a pattern of symptoms suggesting and improved use of language, ability to communicate nonverbally, and reduced stereotyped, repetitive, or idiosyncratic speech over time

Researchers today have conducted new studies to revise how ASD can be diagnosed and treated. As I said before, the disorder is commonly

The prevalence of ASD ranges between 3.3 and 10.6 for every 1000 children with a general mean prevalence of 6.6 per 1000 children. There is significant higher prevalence among white non-Hispanic kids compared with black non-Hispanic kids (Centers for Disease

As well as a broad age group, the researchers sought out high functioning ASD patients as well as a homogenous sample which they concluded to be males. Extensive surveys were given to children and adults alike to test for comorbidity, since a participant with an undiagnosed disorder or unusual symptoms could inadvertently and extensively alter results.

ASD affects a child’s ability to interact and communicate with others. No child is the same

One obvious difference between the years 2002 and 2012 is the medical understanding of ASD and the change in diagnosing criteria. In the fourth edition of the Diagnostic and Statistical Manual

ASD can be detected as early stage of infancy. The primary signs of a child with ASD may include

In the medical community, the prevailing belief is that the rare genetic changes—mutations, essentially—associated with ASD may be sufficient to cause the disorder. This idea of “rare genetic changes” is vague but it is the strongest foothold on which to rest scientific inquiry. Doctors feel sure that genetics play a part in ASD but it is unclear if genetics alone are culpable. Most research indicates that environmental factors contribute impact ASD. It is fair to say, then, that ASD has roots in both genes and in the environment. What share each cause has

ASD is identified by three distinguishing primary impairments in language, social skills and behavioural flexibility, which are referred to as the Triad of Impairments (Happe’ & Ronald, 2009). Additionally, the primary characteristics that define ASD are often accompanied by various other features such as; learning disabilities, self-injury, aggression, repetition, ritualistic eating, drinking or sleeping behaviours and a compelling need for routine (Happe’ & Ronald, 2009). Diagnosis is often carried out using the Autism Spectrum Quotient or AQ (Baron-Cohen, 2006) and it consists of a fifty item

Currently, there is no dietary approach that has shown to improve the condition of patients with ASD and adapted in the management guideline. However, the evidence of the elimination diet is arising as one of the alternative dietary approach for ASD patient. This intervention was first articulated in 1991, and has been emerging in the recent decades with more studies. 4

Lastly, the impairments cannot be attributed to the individual’s developmental or sociocultural context, and they cannot be caused by drug use. Due to the extensive nature of these DSM criteria, however, I used only three aspects of the diagnostic list to assess Holmes. The complete list of diagnostic criteria for ASP can be found in the Appendix (American Psychiatric Association, 2013).